Skip to content
  • (03) 5756 2444
  • reception@alpinevet.com.au
  • Alpine Animal Doctors, 7047 Great Alpine Rd, Porepunkah, VIC 3740
  • Home
  • About Us
  • Services
  • Hospital Facilities
  • After Hours Vet
  • Animal Health Advice
  • News
  • Contact Us
  • Home
  • About Us
  • Services
  • Hospital Facilities
  • After Hours Vet
  • Animal Health Advice
  • News
  • Contact Us

Animal Health
Advice

Compassionate care for every creature, great and small.

Contact Us Emergency
  • Atopy
  • Arthritis
  • Cat flu
  • Chronic Renal Failure
  • Conjunctivitis
  • Constipation
  • Dental disease
  • Ear infections
  • Fatty Liver disease
  • Feline Infectious Anaemia (FIA)
  • Feline Infectious Peritonitis (FIP)
  • Feline Immunodeficiency Virus (FIV)
  • Feline Leukaemia Virus (FeLV)
  • FLUTD and Cystitis
  • Flea bite hypersensitivity (FAD)
  • Heart disease
  • Hyperthyroidism
  • Obesity
  • Ringworm
  • Solar dermatitis

 

Atopy

Atopy is an allergic condition that can affect both cats and dogs. It is thought to be inherited, and is usually first seen between 6 months and 4 years of age, although it’s possible from 3 months to 7 years.  Susceptible animals become allergic to pollens — grasses, trees, weeds — dust mites, mould and fungal spores, wool, flea saliva antigens, and become intensely itchy when exposed to their allergen(s). Allergy is usually due to a range of allergens.

Clinical signs:

The signs of atopy are generalised, all over itching. Dogs are particularly itchy around the ears, under front legs, groin and inside the hind legs, feet, around the eyes and on the belly. Mild cases have only one or two affected areas, more severe cases involve them all. Cats are usually most itchy around the head.

Initially the itch may wax and wane with seasonal changes in pollen numbers and types. Over time this seasonal pattern often becomes permanent. The intense itchiness causes the animal to chew, scratch and bite resulting in inflammation/redness of the skin, hair loss, brown saliva staining of the fur from licking (especially seen on front feet), scaling, crustiness, darkening and thickening of the skin, ulceration and secondary skin infections.

Inflammation and redness of the eyes may develop from animals rubbing their irritated eyes, and recurrent ear infections are also very common as the ears are lined in skin, which is affected.

How is atopy diagnosed?

The diagnosis of atopy can be made after we have seen a pet more than once with itchiness, or in severe cases involving most of the usual affected areas of skin. Other possible causes of the itching are food hypersensitivity, flea allergic dermatitis, mange mites, yeast infection of skin, or contact irritation.

We will examine your pet and usually take skin scrapings, ‘sticky tape’ preparations, and/or ear smears to examine under the microscope, or test in our pathology lab. In some cases we ask you undertake a food allergy trial to rule out this as possible or contributing cause of the itchiness.

Treatment protocols:

Atopy, and other allergies, are generally not curable, but they can be managed to keep your pet more comfortable. The one exception to this is hyposensitisation (see, referrals, below).

Treatment is based on the severity of clinical signs. Mild cases can often be managed with oral antihistamine tablets. We may have to work through a few different types of these before we find one that works well for your pet, trialing each for around 10 days. Advantages of using antihistamines include them being non-prescription, so once an effective medication is determined they can be purchased over the counter from us without a consultation. They are administered as needed not as a course, and have few side effects apart from drowsiness — which may be seen initially but most patients develop a tolerance to this after a short time — and increased appetite.

More severe or chronic cases with inflamed irritated skin are usually treated with corticosteriod injections and tablets. These settle the inflammation and itch, allowing healing. Short term use of these prescribed medications is fine, however high doses and long term use can result in side effects of  immune suppression with an increased susceptibility to infections, increased hunger, thirst, and urination, muscle weakness, thin skin and hair loss.

If secondary skin infections are present (which are common), antibiotics and specialised shampoos may also be prescribed.

Most atopic pets are also extremely sensitive to flea bites, so a regular good quality flea treatment like monthly Frontline or Advantage is important. We recommend all atopic patients and their housemate pets be treated. Although other pets in the house may not themselves be affected by flea bites they carry fleas which will bite the affected pet. Many patients have their overall itch level reduced by good flea control, including oral fatty acid supplements in their diet, or by using special anti itch shampoos and conditioners such as Epi-soothe, Resi-soothe or Aloveen.

Ear infections are frequently present and need to be treated with ear drops or oral medication.

Referral:

Atopic patients have the option of referral to specialist veterinary dermatologists. We sometimes recommend this for severe cases, especially those with a year round pattern, and those that are difficult to control. Dermatological specialists may perform a skin patch test to identify the allergens involved, and try to reduce your pets response to these through hyposensitisation. Hyposensitisation involves a course of injections of the allergens at gradually increasing doses over a long period, to ‘train’ the pets immune system to respond normally rather than excessively when exposed to these naturally in the future.

Arthritis

Osteoarthritis (also called degenerative joint disease  or DJD in veterinary medicine) is a progressive deterioration within the joints, where degeneration of the cartilage lining the ends of bones, changes of the underlying bone at the joints and changes of the joint capsule that secretes the joint fluid leads to constant and often severe pain. Osteoarthritis affects both dogs and cats and arthritic changes are seen in most older patients at some stage in their lives, but the disease can also affect young animals if associated with inherited or growth disorders such as hip dysplasia or patella luxation, or sometimes as a result of trauma such as a fracture, dislocation or ligament injury.

In older patients long term exercise and simple ageing are usually the cause. Working dogs and overweight pets are more likely to suffer osteoarthritis at a younger age. Other possible causes are immune-mediated arthritis — rheumatoid arthritis — infectious arthritis or a tumour.

Clinical signs:

Intermittent lameness that slowly becomes more frequent and severe is the usual sign. Stiff movement, difficulty rising and difficulty going up and down stairs are also common signs. Exercise can result in increased lameness, as can cold weather and lying down for long periods. As these signs develop very gradually it is often easy to accept them as signs of old age, rather than as signs of our pet suffering treatable pain associated with osteoarthritis.

Because arthritis usually develops so gradually, many pet parents find it hard to remember how their pet was before arthritis set in. Your pet is definitely in pain if they are showing the signs of arthritis: limping, sore after exercise, getting up or down gingerly (slowly), or can’t or won’t climb steps. The pain is constant and our pets very stoic, so these signs are the only way to assess how painful the arthritis is, If seen the pain should be treated. Response to medication in most cases is excellent, and can give an older pet a new lease on life.

How is osteoarthritis diagnosed?

X-rays are often taken and, in some cases, analysis of fluid from within a joint or biopsy of tissues within the joint may be needed. For the most part though, a diagnosis can be made from a simple examination and our knowledge of the history of your pet.

Treatment protocols:

The degeneration within the joint is permanent and will continue so treatment of osteoarthritis is primarily palliative i.e., designed to relieve the clinical signs with painkillers, and in some cases slow the progression of osteoarthritis.

Two types of medical treatment exist. Straight pain relief is available in oral tablets or drop form. This type of medication gives very good results but has the disadvantage of needing daily or twice daily dosing.

A second type of medication frequently used is a course of 4 injections of Cartrophen, given at 7 day intervals. Improvement with this treatment is usually gradual over the 4 weeks, so additional pain relief may also be given for the first 1-2 weeks. These injections treat the pain of osteoarthritis, but also slow the ongoing degeneration by protecting the cartilage, and improving the quality and amount of joint fluid in all the joints of the body. An excellent response in usually seen with this medication. Duration of effect is variable, often related to the initial severity of signs. Further treatment is tailored to the individual; some patients may be extremely comfortable for 6-12 months before requiring a repeat course of injections, others may deteriorate after a couple of months and require a single injection to be given every 1-2 months. Some animals need ongoing pain relief as well as the injections.

Sometimes when the diagnosis of osteoarthritis is made an underlying cause is also diagnosed, such as hip dysplasia, cruciate ligament rupture or patella instability, which may need surgical treatment.

Home care:

Diet plays a large part in ongoing treatment. There are a number of ‘joint support’ diets available, all of which contain elements which can help reduce the effects of the disease. These can be the addition of green-lipped mussels, traditionally used in treatment of arthritis for the past 25 years and/or glycosaminoglycans, a vital joint nutrient. These complete and balanced formulations not only promote joint health, they deliver a nutritionally balanced diet for overall patient well-being. One of the most important things you can do for a pet suffering from arthritis is to not overfeed — keep them lean.

Being overweight puts added pressure on joints, increasing pain and degeneration. It’s very easy for an arthritic patient to become overweight as their activity levels fall due to the pain. Overweight pets need to be on strict diets — please talk to us for advice about this.

Activity should be restricted to a level that doesn’t aggravate the condition. If your pet is sore after a walk try shorter walks. If chasing the ball makes things worse, play for less time, or not at all. Swimming is an excellent form of exercise as it is non-weight bearing, and puts less stress on joints, but cold aggravates osteoarthritis so a warm pool is needed.

Cold should be avoided as much as possible. Arthritis will always be worse in winter. If your pet is allowed to sleep inside this will help. Try to minimise exposure to cold by providing thick warm bedding, ideally not on concrete), a warm sheltered area and consider a warm dog coat for winter.

Cat flu

Cat flu, or Feline Respiratory Disease, is primarily caused by any one of many strains of two viruses — a herpes virus and a calicivirus. The infection is spread by contact with another infected cat, or through contact with infected objects, which can be brushes, food and water bowls, bedding, or anything an infected animal has been in contact with, including the owner’s hands. Inhaling infected air can also transmit the disease.

Symptoms appear anything from 1 to 10 days after exposure. The disease usually persists for 1 to 3 weeks, or longer in some cases.

Clinical signs:

Infected cats may exhibit some or all of the following signs:

  • decreased appetite
  • discharge from eyes and/or nose
  • sneezing and/or coughing
  • snuffly nose
  • lethargy
  • ulcers on tongue

Treatment protocols:

We will prescribe medications for your sick cat. These are effective but nursing at home is just as important in treating cat flu.Try to make your cat more comfortable by…

Keep it inside in a warm place, and isolated from other cats in the house

Regularly wipe away any eye or nasal discharge with damp cotton wool

An unwell cat will often not groom itself, which makes it uncomfortable. Frequent grooming on your part, and plenty of TLC, will help it feel more comfortable.

Getting your cat to eat is critical, but it will be reluctant as a cat that can’t smell often won’t eat. Feed strong smelling foods such as tinned fish, or dabs vegemite on food to encourage eating.

Fluid intake is equally vital. Try anything to get your cat to keep drinking —  water, water and glucose, milk, vegemite dissolved in water, broth.

To help clear a blocked nose try using a steam vapouriser in the room with your cat, or keep it in the bathroom with you while you shower. The steam will help loosen the mucous. Small dabs of Vicks Vapour Rub under the chin may also help.

Chronic Renal Failure

Chronic renal failure, sometimes referred to as kidney disease, is a serious disease that progresses over time, and is one of the most common conditions affecting cats. Chronic renal failure can occur in cats of any age but is most commonly seen in middle-aged to senior cats. Around one in five cats over 15 years of age has renal failure. In general, chronic renal failure is seen about three times more frequently in cats than it is in dogs.

Although the disease is irreversible once advanced, appropriate support and treatment can both increase the quality of life of affected cats and prolong life by slowing down the progression of the disease.

Chronic renal failure occurs when long-standing, irreversible damage to the kidneys impairs their ability to filter and remove waste products from the blood. In most cases, the exact cause of the disease remains unknown. Biopsies taken from affected kidneys will often show large amounts of fibrous tissue has replaced the normal kidney tissue, often with some inflammation — ‘chronic interstitial nephritis.’ However, these fibrous changes are also present in a number of diseases. There are, however, some well-recognised causes of chronic renal failure,including:

Polycystic kidney disease, an inherited condition seen mainly in Persian and Exotic cats where normal kidney tissue is gradually replaced by multiple fluid filled cysts that develop within the kidneys.

Kidney tumours such as lymphoma, a solid tumour of white blood cells, can affect the kidneys and cause renal failure.

Bacterial infections of the kidneys — ‘pyelonephritis’ — may occur as an extension of bladder infections, and sufficiently damage the kidneys to cause renal failure.

Damage to the kidneys by toxins, defects in the development of the kidneys at birth, persistent inflammation such as glomerulonephritis are also known causes.

In most cases a specific cause cannot be found for renal failure, but where an underlying cause can be diagnosed, and is treatable, there may be some potential to halt progression of the disease.

Clinical signs:

The onset of chronic renal failure is slow and insidious, although occasionally signs may appear to develop quite suddenly. Many of the clinical signs are quite vague and non-specific.

The most common signs seen in affected cats are a poor appetite, weight loss, dehydration, lethargy and depression. An increased thirst often goes along with increased volumes of urine being produced due to an inability to concentrate the urine in many affected cats.

Other signs may include a poor coat, vomiting, bad-smelling breath, mouth ulceration, and weakness. These signs tend to get worse over time.

Given the diverse functions of the kidneys, a wide variety of complications can arise in affected cats. These might include electrolyte abnormalities, high phosphate concentrations, acidosis (the retention of too much acid by the body), the development of hypertension and anaemia.

How is renal failure diagnosed?

Our diagnosis of chronic renal failure is made by analysis of blood and urine samples. Two substances in the blood — urea and creatinine, products of metabolism that are normally excreted by the kidneys — are commonly analysed. A urine sample is usually analysed at the same time. Typically with chronic renal failure, as well as appropriate clinical signs and increased urea and creatinine concentrations we will also see poorly concentrated urine, so urine ‘specific gravity’ is measured to assess its concentration.

Treatment protocols:

If a specific cause for the renal failure, such as bacterial infection of the kidneys, can be identified then treatment for the cause may be possible. In most cases however, a cause cannot be identified and treatment is aimed at managing the renal failure.

Some cats may require initial intravenous fluids to correct dehydration, and perhaps electrolyte abnormalities, but once stable, treatment is aimed at supporting renal function and minimising the complications.

Optimal management of renal failure usually requires repeat investigations at regular intervals to identify treatable complications as they arise, e.g., anaemia, low potassium, high phosphate, urinary infections, and hypertension. Chronic renal failure is irreversible and, in most cases, will progress over time despite appropriate therapy.

Good Dietary management is important for cats with chronic renal failure, for three reasons.

Water intake: Cats with chronic renal failure are more likely to become dehydrated because of the reduced ability of the kidneys to conserve water by concentrating urine. Maintaining a good fluid intake is therefore very important. As cats generally gain much of their water from their food, whenever possible, cats with chronic renal failure should be fed canned (or sachet) foods rather than dry foods.

Protein content: A cat with renal failure should have a low protein diet. Many of the toxic products that accumulate in the blood in renal failure are due to protein breakdown; feeding a low protein diet will minimise this. However, too little protein in the diet can lead to excessive and detrimental weight loss. It’s best to use specially designed commercial products.

Low phosphate content: Restricting the phosphate intake appears to be quite beneficial in protecting the kidneys from further damage. Commercial diets designed to help manage renal failure are restricted in both protein and phosphate. If a cat is not eating a low phosphate/low protein diet, or if blood phosphate concentrations are high despite a low phosphate diet,  ‘phosphate binders’ can be added to the diet (under veterinary supervision) to reduce the amount of phosphate absorbed.

Commercial diets for cats with renal failure also often have additional fibre and polyunsaturated fatty acids, which may offer some additional benefits in the management of the condition.

Cats with chronic renal failure tend to become dehydrated, with significant adverse effects on kidney function. A good supply of water should always available, and cats should be encouraged to drink by offering water from different bowls, offering flavoured waters (chicken or tuna, for example), etc. In some patients, usually those with advanced renal failure, there may be some benefit from intermittent administration of fluids by intravenous drip, during visits to the hospital.

Other treatments are generally aimed at specific complications that can arise as a result of renal failure, including:

Potassium supplementation to help combat low blood potassium levels.

Cats with chronic renal failure that also develop hypertension are at risk of developing further problems (such as bleeding into their eyes and retinal detachment leading to blindness, or bleeding into the brain causing neurological signs) in addition to the high blood pressure potentially further damaging the kidneys. It is important to treat hypertension when it is diagnosed, usually with medication.

In advanced renal failure, anaemia is common and can contribute to lethargy and weakness. Depending on the cause and severity treatment options include anabolic steroids, iron supplementation, management of any gastrointestinal ulcers and, in some advanced cases supplementation, with erythropoietin — the hormone that stimulates red cell production.

 

Conjunctivitis

The conjunctiva is the thin, pink membranes surrounding the eye, lining the globe, inner eyelids and third eyelid and forming a seal with the inner surface of the eyelids. Conjunctivitis is an inflammation of this membrane which then becomes swollen and reddened, often making it more visible. Conjunctivitis can affect one or both eyes in cats and dogs.

In dogs, most forms of conjunctivitis are allergy based or bacterial infections. In cats, viral (herpes), bacterial (Chlamydia) or mycoplasma (similar to bacteria) infections are more prevalent. Another cause is ‘dry eye,’ where tear production is decreased or absent. There are many other less common causes.

Long haired cats may be more susceptible to conjunctivitis as their eyes are more prominent and less protected by the eye socket, or there may be abnormalities in tear drainage causing an overflow of tears.

Clinical signs:

  • Red eyes
  • Swollen conjunctivae
  • Mucky discharge
  • Rubbing or scratching at eyes
  • in dry eye, dogs show a thick, ropy discharge and the eyes look dry

How is conjunctivitis diagnosed?

The condition is diagnosed by physical examination.

Treatment protocols:

In most cases, antibiotic drops or ointments applied directly into the eye are the usual treatments. For allergic conjunctivitis, anti-inflammatory drops are used, but occasionally oral antibiotics/anti-inflammatories are also required. Some conditions require a more intense action, such as swabs of the cornea to diagnose the cause.

An important part of treatment is cleaning of the eyes. Often thick discharges become stuck to your pet’s fur and are difficult to remove and uncomfortable to the patient. This can be done at home. The best way to bathe the eyes is as follows:

1. Wet a wad of cotton wool in warm saline water (warm enough that you can still touch it).

2. Squeeze out some of the water and place over the animal’s closed lids. Most animals don’t object; they find this very soothing.

3. Hold the pad there for 10 seconds. This dissolves and loosens the discharge.

4. Wipe away the discharge, from inside towards outside, in one movement.

5. Repeat as necessary until all discharge is cleaned away.

Bathing the eye in this way is soothing to your pet’s irritated eyes, removes discharge and remnants of the last application of ointment, and improves the blood supply to the area and aids healing.

Regular treatment with the eye medication is essential. Most ointments need to be administered at least twice a day. Drops need to be given more frequently and so are less practical. Unfortunately, some treatments cause transient discomfort after administration, meaning two people are required, one to hold the cat and the other to give the treatment.

Liquid preparations can be applied directly onto the surface of the eye, 1 or 2 drops per eye is usually sufficient. Ointment is applied across the surface of the eye, about half an inch per eye. The eyelids are then closed to spread the ointment over the entire surface of the eye. Generally ointments need to be given less frequently than drops as they persist for longer but some people find drops easier to administer. If you have any doubts as to how to give the medication prescribed then don’t be afraid to ask us for a demonstration.

Allergic conjunctivitis usually resolves quickly but, like all allergies, may return at certain times of year or on exposure to the allergen, especially if the allergen is pollens in the air.

Infectious conjunctivitis has a good prognosis, however dogs and cats often transfer the infection from one eye to the other, or to other dog or cat members of the household.

Dry eye requires lifelong treatment, but the eyes can be made much more comfortable.

Constipation

Constipation is a condition most commonly seen in middle-aged and senior cats, and in entire male dogs with prostatic enlargement. It’s an uncomfortable and frequently very painful condition resulting from an abnormal accumulation of faeces leading to difficulty in passing bowel motions. This may result in reduced frequency or absence of defecation, and/or excessive straining to defecate.

The faeces are retained in the large intestine, the colon. One of the functions of this part of the bowel is water absorption. In a constipated cat, the retained faeces become hard and dry, which makes passage even more difficult and painful. Paradoxically, some constipated animals may also pass small amounts of liquid faeces.

Causative factors associated with constipation in cats include:

  • Excessive hair in the stool, especially in long-haired cats, and cats with a flea allergy
  • Ingestion of foreign bodies.
  • Obstruction caused by tumours, strictures or masses compressing the large intestine.
  • Pelvic injuries, most commonly after a road accident, resulting in a narrowed pelvic canal.
  • Damage of the nerves which cause the colon to contract. This may arise following traumatic damage such as a road accident, or may be seen as part of more generalised neurological disease.

In some cases, there is no obvious cause but in all cases of extended or severe constipation the affected cat will require treatment at the hospital.

Frequently associated with constipation is megacolon, the term for a dilated and poorly motile colon. When the colon becomes distended with faecal material over a prolonged period  its ability to contract may be reduced or lost, resulting in megacolon. Megacolon may be seen as a primary disease, or it may arise following long-term constipation.

Clinical signs:

Affected pets usually strain, unsuccessfully, to defecate and may cry in pain while trying. Any faeces passed are hard and dry. The animal may also show signs of lethargy, reluctance to eat and vomiting.

How are constipation and megacolon diagnosed?

In most cases, a diagnosis of constipation can be made on the basis of the clinical signs your pet is showing. Further tests may be needed to establish the cause of the constipation. These may include x-rays of the abdomen and pelvis in order to look for pelvic injuries, colonic strictures or tumours. X-rays are also required for the diagnosis of megacolon.

Treatment protocols:

Initial treatment of a pet with constipation may involve administration of enemas and manual extraction of faeces at the hospital. The latter may necessitate an anaesthetic or sedative. Treatment of dehydration with intravenous fluids may also be needed in severely affected animals.

If the constipation recurs or becomes a long-term problem continuous therapy may be needed to prevent recurrence. A wide variety of treatments are available and the aim of these is to enable regular defecation of soft faeces. Altering the diet so that this contains more fibre (e.g., Metamucil) may be helpful, and lubricating laxatives or stool softeners may also be used in mildly affected animals.

More severely affected cats may need drugs that stimulate contraction of the colon. The doses of all of these drugs may need to be altered to produce the desired effect. Ideally, animals should defecate at least once every other day (most normal pets defecate once or twice a day depending on their diet).  Over a period of time, resistance to the treatment may be encountered, necessitating an increase in the drug dosage or a change to another therapy. No changes to the treatment protocol should be made without consulting your veterinary surgeon.

In long-haired cats regular grooming may reduce hair ingestion and reduce the likelihood of hairballs causing constipation.  Likewise treating any concurrent allergic skin disease, for example by flea control, is also vital. It is important to ensure there is always access to a clean litter tray so that frequent defecation is encouraged.

If megacolon develops, or if the constipation is severe and medical treatment is unsuccessful at controlling this, then surgery may be advisable. Surgical treatment involves removal of most of the colon which initially will result in diarrhoea since the colon is responsible for water removal from faeces. Over time this usually improves. The surgery should not result in loss of bowel control.

The long-term outlook varies tremendously according to the cause of the constipation, however, most patients can be adequately managed without having to resort to surgery, especially if medical therapy is implemented early.

Dental disease

Dental disease is far and away the biggest health issue facing cats. The extent of poor oral health in cats is huge, of epidemic proportions. We don’t really know how extensive the problem is in cats but it’s certainly worse than in in dogs — and, by the time they reach the age of three, 80% of all dogs are showing some signs of dental disease. For cats, it’s thought that as many as 98% may be suffering dental disease.

The good news is that, first, most dental health issues are preventable. You can keep your cat out of the hands of the pet dentist with some simple home care, and through regular annual veterinary dental checks. If your pet already has some degree of oral health problems, caught early enough, they are most likely reversible.

Dental health problems in pets begin with plaque, which is formed by food particles and bacteria collecting along the gumline. Plaque begins to mineralise 3-5 days after it forms. If it is not removed by regular brushing and cleaning, minerals in the animal’s saliva combine with the plaque to form tartar (or calculus) which sticks strongly to the teeth. Tartar irritates the gums and causes gingivitis, an inflammation that can be seen as a reddening of the gums adjacent to the teeth. Gingivitis is what causes your pet to have bad breath. Left untreated, gingivitis will progress to periodontal disease, which is irreversible.

If tartar is not removed it builds up under the gums, separating the gums from the teeth to form ‘pockets.’ These pockets provide an ideal environment for further bacterial growth and more damage to the teeth and gums. Once infection gets into the tooth roots, where it can damage ligaments and bones, your pet has full blown, irreversible periodontal disease (periodontitis) and faces long term health problems. Periodontitis can lead to loose teeth, abscesses, and bone loss or infection and, if bacteria enter the bloodstream, to serious, life threatening heart and kidney disease. Periodontal disease is also always extremely painful for your pet.

Pet carers can prevent the development of serious periodontal disease with simple, regular oral health care at home:

  • Check your pet’s teeth regularly, including the back teeth
  • Ensure your cat’s diet is suitable
  • Brush and clean your pet’s teeth at least twice a week
  • Have their teeth scaled and polished once a year at the hospital’s dental surgery

Clinical signs:

The first signs of tooth and gum disease are usually:

  • Yellow or brown stains on the teeth, indicating a build up of tartar
  • Persistent bad breath
  • Drooling and dribbling
  • Redness of the gums, particularly at the point where the gum meets the tooth, indicates gingivitis
  • The signs of more serious levels of gum disease include:
  • Signs of pus (purulent exudate) around the tooth
  • Gums that bleed easily
  • Sensitivity around the mouth
  • Pawing at the mouth
  • Inflamed or receding gums
  • Loose or missing teeth
  • Loss of appetite
  • Stomach or intestinal upsets
  • Difficulty chewing or eating
  • Irritability or depression

Numerous factors play a role in the formation of plaque, tartar, and the development of periodontal disease. These include:

  • Age and general health status
  • Diet and chewing behavior
  • Breed, genetics, and tooth alignment
  • Grooming habits
  • Home care
  • Mouth environment
  • Tooth anatomy

Age and Health Status: Periodontal disease more commonly affects older animals.

Diet and Chewing Behaviour: Studies show that hard kibbles are slightly better than canned foods at keeping plaque from accumulating on the teeth. Cats that chew on various toys or edible dental chews may remove some of the plaque build-up.

Breed, Genetics, and Tooth Alignment: Some breeds of cats are at greater risk of periodontal disease because their teeth are often crowded together. This results in an increased accumulation of plaque because the normal cleansing mechanisms are hindered.

Grooming Habits: Hair accumulation and impaction around the tooth can increase the development of tartar.

Home Care: Regular brushing of your cat’s teeth can greatly reduce the accumulation of plaque and development of tartar, thus reducing the risk of periodontal disease.

In general, the more acid the saliva, the more rapid the build-up of plaque. The number and type of bacteria in the mouth influence the progression of periodontal disease.

How is periodontal disease diagnosed?

We use a number of criteria to assess the oral health of a cat, including the level and distribution of plaque and tartar, the health of the gums (gingiva), radiologic (x-ray) appearance, and the depth of the gingival sulcus (the ‘pocket’ formed between the tooth and the gum). After assessing these parameters, we can establish the extent and severity of disease and make a prognosis.

Tooth and gum disease are graded according to severity:

Grade I: An animal showing a mild degree of plaque and only minor redness of the gums with no radiologic change is classed as having Early Gingivitis. The prognosis is good, with appropriate treatment able to fully reverse the disease with no lasting effects.

Grade II: Advanced Gingivitis. At this stage, animals will show subgingival (below the gumline) plaque, with edema and redness of the gums as well as as some radiologic changes. With treatment, the prognosis for full reversal of symptoms remains good.

Grade III: At this stage, the animal will be showing signs of Early Peridontitis. Subgingival calculus will be present, the gums will show redness and edema, some degree of recession (receding gumline) or hyperplasia and will bleed when gently probed. Radiology will show a 10 to 30% loss of bone support. Changes are irreversible and the prognosis is for palliative treatment.

Grade IV: Established Periodontitis will show larger amounts of subgingival calculus, severe inflammation and recession of the gums, pus, loose and/or missing teeth, deep pockets and gums that bleed easily. X-rays will reveal bone losses of greater than 30%. Prognosis is for palliative treatment.

Treatment protocols:

Treatment depends upon the severity (Grade) of the disease but in every case your pet will undergo a pre-surgical examination. This may include some pre-anesthetic blood testing to assess the health of your pet. In most cases we will also prescribe antibiotics several days before any scheduled dental procedures. Because animals are somewhat reluctant to ‘open wide’ and allow us to poke around in their mouths, your cat must be anaesthetised and monitored during any dental procedure.

Treatment for Grade I or Grade II involves a routine professional prophylaxis (dental cleaning and polishing). Plaque and tartar will be removed from the teeth, above and below the gumline, with handheld and ultrasonic scalers. Each tooth and the entire oral cavity is checked for disease. We will then polish the teeth to remove any microscopic scratches that would otherwise assist plaque formation and tartar build-up.

Following Grade I and II dental procedures we will suggest a regular home care program to control plaque. An individual patient plan might include the removal of plaque through brushing with specially formulated toothpastes, gels and rinses, and a nutrition plan.

Grade III and Grade IV periodontal disease requires more extensive treatment that may include root planing and subgingival curettage, periodontal debridement, gingivectomy, periodontal surgery, special therapeutics, and tooth extraction.

Root planing is a complex and difficult procedure that involves removing residual calculus and diseased cementum or dentin, and smoothing the root surface. Subgingival curettage removes diseased epithelium and connective tissue. This is also a difficult procedure.

An alternative to root planing and gingival curettage is periodontal debridement. In this procedure we use special ultrasonic scalers to remove irritants from the tooth and root surface such as bacteria and endotoxins produced by the bacteria.

In a gingivectomy hyperplastic or excess gingiva is removed. The area between this excess tissue and the tooth is a perfect habitat for bacteria. Periodontal surgery involve opening a flap of the gingiva over the root of the tooth to be able to reach the deeper structures. In some cases, a tooth cannot be saved and extraction is the only alternative.

Home aftercare for pets with Grade III or IV periodontal disease is critical. Pain relief and anti-inflammatory medication will need to be administered post-operatively and for several weeks following the procedures. Antibiotic therapy, usually for one or two weeks after surgery, will also be prescribed. You may also need to apply products containing zinc ascorbate, stannous fluoride, chlorhexidine or plaque preventives to the teeth on a regular basis.

Pets with periodontal disease will need frequent check-ups to assess their continuing oral health. Routine examinations at least twice a year will be needed.

Preventing tooth and gum disease:

There are a number of things you can do at home to help prevent your cat from ever developing serious oral health issues.

Brush your pet’s teeth regularly, two or three times a week. Yes, it can be difficult if your cat is uncooperative, but they can be trained and regular brushing is the most effective way to reduce plaque and prevent the build-up of tartar. You can gradually condition your pet to accept brushing by just using your finger, with a little bit of liver paste or something on the tip, to gently rub the outside of the teeth. Gradually replace the paste on your finger with a bit of pet toothpaste and slowly work up to using a brush. We also have finger brushes available, like a thimble with bristles. Many pet carers find these easier to control. It may take a few weeks but eventually your pet will come to accept having their teeth brushed as just part of the routine.

You’ll need a product designed for animals; human toothpaste should not be used because of their foaming action and high levels of fluoride. The taste is also unpalatable to most pets. There are a number of pet pastes and gels available, often flavoured to be acceptable. Only the outside of each tooth needs to be brushed. That rough tongue does a good job of cleaning the inside of the teeth.

Diet is a crucial part of dental care. Some researchers and vets believe that the reduced amount of bones pets get in these days of convenient packaged pet foods may account for the dramatically increased incidence of tooth and gum disease we are seeing in dogs and cats today. Whether that’s true or not, there is no doubt that feeding your cat raw bones a couple of times a week is very beneficial for the health of their teeth and gums. Chewing through bone and cartilage, and tearing the meat off the bone, exercises and cleans the entire tooth, right up to the gum line. Bones must be raw, not cooked.

Bones need to be part of a balanced diet that may include special dental diets. Contrary to popular belief, feeding your cat exclusively on dry food (or kibble) is no better for their teeth than any good commercial packaged food, wet or dry. The exception are the ‘dental’ diet dry foods. The kibble used in these is designed so that your pet has to chew each piece. The structure of the kibble remains intact longer than other dry foods, providing a degree of ‘scrubbing’ against the teeth that helps reduce plaque buildup. Dental foods are also often mildly medicated with chemical plaque cleansers.

The regular use of good quality artificial chews such as ‘Greenies’, which are designed to stimulate natural cleaning of teeth and gums, can also be helpful.

The single most important thing a cat parent can do to keep their pet’s teeth and gums healthy is to schedule an annual dental checkup, and a scale and polish every year. Periodontal disease is painful, irreversible and can be very expensive to treat. There is no need for you or your dog to have to go through it if you get your pet on a good oral health care program now. We are happy to provide free dental checks and to advise you on the best home care plan for your particular pet.

Ear infections

Sore ears are common in both dogs and cats. The design their ear canals allows debris, wax and moisture to accumulate within the ear. This is even more likely in breeds with long, floppy ears. Ears with lots of folds and ears with lots of hair growing in the ear canal are more likely to suffer problems.

A sore ear can be particularly distressing for your pet. And an otherwise easily alleviated problem can turn into something more serious with excessive scratching and head shaking. To determine what is the cause of your pet’s problem it is best to bring them in for an examination as quickly as possible.

There are many things that can cause ear problems, including injuries, bacterial infections, fungal (yeast) infections, grass seeds, dirt or sand, excessive wax production, soap or water in the ear canal, ear mites, allergies and other skin conditions.

Clinical signs:

Your dog or cat may show any of the following symptoms…

  • Scratching one or both ears
  • Smelly ears
  • Head shaking
  • Head tilted to one side
  • A yellow, green, brown, black or cream discharge from the ear
  • Painful ears or head when touched
  • Redness and inflammation of ear flap and ear canal
  • Swelling of ear flap
  • Stumbling or circling to one side
  • Lethargy and depression.

How are ear problems diagnosed?

Because most ear problems show the same signs, the ear canal and ear drum have to be examined with an otoscope for us to make a diagnosis. A grass seed in the ear may cause your dog to have exactly the same signs as a bacterial infection so we need to be able to see into the canals to tell the difference. The ear drum must be assessed, as a ruptured ear drum is a much more serious illness which can cause balance problems and inner ear infections. The medication we choose may be altered if the ear drum is ruptured.

Sometimes a smear is taken and examined in our in-house laboratory to check the cause of the problem. Other times a sterile swab is taken to ‘culture the bacteria or yeast that are causing the infection, and thus determine the appropriate treatment.

Some dogs that have chronic recurring problems have bacteria or fungi that are resistant to the common ear ointments and drops, and need different formulations to treat the infection. This can only be determined with microbiological culture and sensitivity testing.

Treatment protocols:

Medication, in the form of drops, ear cleaning solutions and tablets, is usually all that is needed to quickly clear up ear problems. It is essential that these treatments are used correctly. Intermittent use of antibiotic solutions can favour the multiplication of resistant bacteria, which is why we always prescribe the drops only for a certain time and recommend a revisit to check the ears at the end of the course. If the ears aren’t 100%, the problem will reoccur once the medication is stopped.

Sometimes more extensive treatment is necessary. An ear that is full of pus may need to be flushed under general anaesthesia to remove as much discharge as possible and assess the ear drum. Or a grass-seed must be removed under sedation or general anaesthesia to prevent rupture of the ear drum.

Home prevention:

There are some things you can do to help prevent ear problems:

  • Cut the hair around and inside the ear flap short to allow better circulation of air
  • Get your groomer to pluck the ears regularly to prevent hair falling down into ears.
  • Use an antibacterial ear cleaner once or twice a week. These protect against a broad range of infections and also dissolves the wax and debris inside the ear and allow the dog or cat to shake it out. It also dries out the ear if your dog has been swimming or water has gone into the ear during a bath.

Fatty Liver disease

Fatty liver disease, or Hepatic Lipidosis/Idiopathic Hepatic Lipidosis, is the most common cause of  liver disease in cats. It is caused when a cat stops eating, or severely reduces its food intake, leading to anorexia. With little or no nutrient intake, the body begins to use the animal’s fat stores as fuel. These fat stores are sent to the liver, to be broken down to supply nutrients.  Sometimes, the liver becomes overwhelmed and is unable to process this fat quickly enough, which allows a build up of fat in the liver, which in turn interferes with normal liver function.

Left untreated the condition can quickly lead to death. If your cat stops eating for more than a couple of days, call the hospital for advice.

Hepatic Lipidosis can strike a cat of any age and condition, although it is seen far more more often in obese cats. Obesity is considered a major causative factor for fatty liver disease.

Other causes of hepatic lipidosis may be medical conditions which predispose your cat to the disease, including renal failure, pancreatitis, hyperthyroidism, FLUTD,  owner induced diet (without veterinary supervision). Stress leading to a loss of appetite can also cause the disease.

Around half of cases are idiopathic hepatic lipidosis, which have no known cause, hence idiopathic.

Clinical signs:

In the early stages there may be no visible signs unless you are familiar with your cat’s eating habits. Hepatic lipidosis is always preceded by anorexia. If you see, or suspect, anorexia in your cat you should make an immediate appointment for us to examine the animal at the hospital. Other signs may include…

  • Weight loss
  • Muscle wasting
  • Dehydration
  • Vomiting
  • Depression
  • Lethargy
  • Excess salivation
  • Jaundice (yellow colour to the skin and mucous membranes)

How is fatty liver disease diagnosed?

We will initially perform a physical examination and a biochemical profile to check serum levels of specific liver enzymes, which may be markedly elevated. We may also use an ultrasound or x-ray to check the appearance of the liver and eliminate other conditions. If necessary we will take a sample of liver cells to detect the presence of fat cells to confirm diagnosis.

Treatment protocols:

Appropriate treatment is dictated by the severity of disease and any underlying medical conditions the cat may have. Aggressive therapy is usually required to save the cat and may include:

Intensive nutritional support, which involves feeding a  calorie dense, high protein food via a feeding tube either directly into the stomach or esophageus.

Fluid and electrolyte therapy if the cat is dehydrated.

If caught in time approximately 70% of cases of fatty liver disease can be reversed. Untreated, hepatic lipidosis is fatal.

Prevention:

Probably the most effective prevention measure is to prevent obesity in your cat by feeding a complete balanced diet, eliminating treats and maintaining the cat’s nutrient intake at an optimal level.

Feline Infectious Anaemia (FIA)

Feline Infectious Anaemia (FIA) is caused by Haemobartonella felis, a tiny, microscopic bacterial parasite which attacks the cat’s red blood cells within the circulation. Damaged cells may die or be destroyed at an increased rate by the spleen, leading to anaemia.

Although FIA is caused by an infectious agent, it is not necessarily a problem in other cats living in the same house. The way FIA is spread between cats is uncertain; cat bites and fleas may spread the parasite but other methods may also be involved. Fighting is known to represent a risk of spread of infection. Flea infestations should be treated promptly — the presence of fleas may make anaemia of any cause more severe. Even if your other cats do become infected this may not result in any anaemia or other problems.

Clinical signs:

Cats are very good at hiding anaemia until it is quite severe. The earliest indicators may be pallor of the mucous membranes — the conjunctiva (lining of the eye), gums and tongue appear white or pale instead of their normal pink colour.

It can be difficult to assess the mucous membranes in some cats. Occasionally they may appear rather pale but there are causes of pallor other than anaemia. As the anaemia becomes more severe, the cat may become lethargic and breathless if stressed, or after any exertion.

There may be loss of condition, poor appetite, weight loss and occasionally high temperatures. Sometimes jaundice is seen, but usually only in severe, acute cases.

How is FIA diagnosed?

Diagnosis is difficult. It relies on identifying the tiny organisms microscopically on specially prepared and stained blood films. These organisms are so small they are hard to recognise, particularly if present in low numbers. Diagnosis is further hampered by the fact that parasite is sometimes found on blood cells of normal cats with no anaemia.

Finding FIA in a cat without anaemia would not necessarily be considered significant. There are many other possible causes of anaemia in cats other than FIA, and identifying it in an anaemic cat may not necessarily mean that other causes of anaemia may not also be involved.

Treatment protocols:

Treatment of FIA can also be difficult. No specific drug can be relied upon to eliminate the parasite. However, in the majority of cases, a variety of different drugs can be used to control FIA. Other diseases may also be present in cats with FIA and it is thought that the effects of these other diseases/factors, including stress, may contribute to enabling FIA to cause disease.

Successful treatment may depend on resolving any other disease problems the cat is showing. In severe cases, where the cat becomes very weak, a blood transfusion may be life-saving.

Feline Infectious Peritonitis (FIP)

Feline Infectious Peritonitis (FIP) is a common disease in cats caused by Feline Coronavirus. The infection normally causes a mild intestinal disease, which may either be asymptomatic or cause temporary diarrhoea.

In general, cats don’t usually catch the mutated form of the virus directly. FECV is the form that is transmitted between animals, usually through the saliva or faeces of an infected cat, either via grooming or contact with contaminated objects such as feeding bowls, litter trays or bedding. Once FECV infection has become established, mutated forms (FIPV) may arise during viral replication.

Cats that live in close proximity to each other, especially in catteries, are much more likely to be exposed to FECV, and are also more likely to go on to develop FIP because there is simply more virus replicating in their systems. Certain genetic lines of cats are also more susceptible. Typically, young cats from about 6-months to 2-years of age tend to get FIP, although cats of any age can contract the disease.

In some cats FECV can mutate to a form called Feline Infectious Peritonitis Virus (FIPV), which can infect certain white blood cells. The virus then spreads throughout different organs within these white blood cells.

Normally, the body mounts an immune response to eliminate viral infections. Some cats can fight this mutated form before signs become apparent. The immune systems of other cats seems unable to kill the virus and it is, in fact, this inflammatory reaction that actually creates damage to the body’s tissues and causes the signs seen with FIP.

Clinical signs:
FIP exists in two clinical forms. The ‘wet’ form is characterised by fluid accumulation in the abdomen and/or the chest, and is the most common form of the disease. The ‘dry’ form is caused by inflammation in various body systems. Many cats have a combination of the two forms. The disease, particularly the wet form, may progress over a few days, or it may wax and wane for many weeks.

Typical signs of the wet form are swelling of the belly and/or breathing difficulties, as the respective body cavities fill with fluid.

Signs of the dry form are weight loss, anorexia, fever and depression.

Depending on which organ systems are involved other signs can include jaundice, vomiting and diarrhoea, dehydration, excessive drinking and urination, inflammation in the eyes, or neurological signs.

How is FIP diagnosed?

The diagnosis of wet form FIP is fairly straightforward because of the classic yellow sticky fluid that accumulates.

The dry form, however, can mimic many other diseases and can be challenging to diagnose.

Normal blood tests may show increased protein in the blood, or changes to white cell counts, which are suggestive of FIP, but these can also be seen i many other inflammatory diseases.

Treatment protocols:
There are various treatments for FIP which can be attempted but, to date, none have been conclusively effective in treating the disease. Currently, FIP remains an ultimately fatal condition. There is also no effective vaccine available to prevent FIP.

Feline Immunodeficiency Virus (FIV)

Feline immunodeficiency virus (FIV) is the virus that causes feline AIDS, a disease similar to human AIDS, where the immune system is attacked and gradually weakened.

The virus is found in high levels in the cat’s saliva, and transmitted predominantly by bites, when an infected cat spreads the disease by biting an uninfected cat. Transmission can also occur by cats grooming each other and sharing food bowls. If an FIV positive cat is identified in a multi-cat household, a high proportion of the other cats in the household are likely to be infected.

Infection occurs in two stages. Initially the cat will have a fever lasting a few days to a few weeks, and occurring about 4 weeks after infection. Swelling of lymph nodes may also occur and can last for several months. This initial phase is often mild and may go unnoticed by the owner. Most FIV-infected cats recover from this stage of the disease after a brief illness, although in very young and very old cats even the first stage may be fatal.

The second stage occurs several months, or several years, after initial infection, presenting as an AIDS-like illness. The immune system becomes progressively weaker and infected cats are unable to fight off infection that they can normally cope with.

Clinical signs:

Symptoms in the immunosuppressed stage of the disease, when the immune system is not functioning properly, are caused by the secondary infections and are extremely variable. Infected cats may exhibit some or all of the following signs:

  • Decreased appetite
  • Weight loss
  • Scruffy appearance
  • Poor healing of wounds such as abscesses
  • Persistent or intermittent infections, particularly respiratory tract, urinary tract, skin, ear or eye infections
  • Persistent or intermittent diarrhoea
  • Infections of the mouth (stomatitis) and gums (gingivitis)
  • Anaemia
  • Behavioural changes due to neurological involvement

How is FIV diagnosed?

Diagnosis is made by a single blood test, which detects antibodies to the virus. A small proportion of FIV-infected cats may have too little antibody to be detected. Some cats in the very early stages of infection have not yet developed antibodies to the virus, and some cats in the end stages of infection are unable to produce antibodies as their immune systems are too weak.

Treatment protocols:

There is no specific treatment for feline immunodeficiency virus. Treatment is supportive and palliative, generally directed at secondary infections as they arise. Weight loss is a major feature of many clinically affected FIV infected cats, and it is important to ensure adequate dietary intake. Feeding high energy convalescent diets containing high quality protein is helpful.

An effective vaccine is available to protect your cat against feline immunodeficiency virus. The vaccine is not part of the core vaccines given to cats although we strongly recommend that all cat parents have their pets vaccinated against FIV.

The prognosis for FIV-infected cats is very unpredictable. Some cats may live for many years without any illness. Others may initially be very ill but make an excellent recovery and survive for months or years. An FIV positive result is not necessarily an indication for euthanasia, even in sick cats.

Given that FIV is primarily spread by fighting cats, it is almost impossible to prevent the spread of the disease. You can confine an unvaccinated cat at night to avoid fighting but this is not a practical way to minimise the risk of infection. The only way to completely protect against the disease, and to prevent your cat infecting other cats, is to have your pet vaccinated.

Feline Leukaemia Virus (FeLV)

Feline Leukaemia Virus (FeLV) attacks the immune system of the cat, making them more susceptible to other infections, and to developing cancer. Because the virus attacks the immune system, cats may become sick or even die from other diseases they have been vaccinated against, such as Feline Respiratory Disease (cat flu) and Feline Enteritis.

FeLV is relatively fragile in the  environment, which means it can only be transmitted by direct contact between cats. In close contact, however, it is a highly contagious disease, passed on through saliva, urine and faeces. An infected queen always infects her litter of kittens.

A study at Murdoch University in Perth found that 7-8% of healthy cats and 11% of sick cats were positive to the virus. Feline Leukaemia is thought to be the second biggest killer of our cats. All cats are at risk if they come in contact with other cats, whether wild or domestic.

Clinical signs:

Initial signs often include weight loss, lethargy and generally poor health. Signs can, however, be extremely varied as the virus makes the body susceptible to a wide range of diseases and infections.

Treatment protocols:

Unfortunately, there is no treatment available for FeLV. The emphasis is on prevention of the disease and limiting its spread through vaccination. Happily, there is an effective vaccine to control Feline Leukaemia, which threatens our entire cat population.

Kittens need two injections, which can be given at the same time as their core vaccinations. Adults also need two injections initially, but usually require blood testing first to make sure they haven’t already got the disease. After that, yearly booster shots are required, which are normally given at the same time as routine vaccinations.

FLUTD and Cystitis

Feline Lower Urinary Tract Disease (FLUTD) is a disease that results in inflammation of the bladder — cystitis — and/or the urethra (urethritis). The condition may result in partial or complete obstruction of the urinary tract. The condition is usually due to the presence of small crystals in the urine.

Urinary obstructions are life threatening and require immediate treatment.

The exact cause of FLUTD is unknown and there may be more than one cause. It is a complex condition where a number of factors contribute to the development of the disease, including age, sex, obesity, diet and urine pH. Viruses may also play a role, as the condition often occurs following a stressful period, when the immune response to various infectious agents is lowered. Stress itself may be partially to blame.

Clinical signs:

Most signs result from urinary tract irritation. Your cat may be doing one or more of the following things:

  • Spend prolonged periods squatting and/or straining during urination
  • Continuously dig holes in the garden or get into the litter tray and strain to urinate
  • Pass only very small amounts of urine
  • Attempt to urinate more frequently
  • Cry while attempting to urinate
  • Have blood-tinged (pink or red) urine
  • Urinate in unusual places, such as the bathroom sink or bath.
  • If the urinary tract is completely obstructed (blocked), your cat may show any of the above signs, and/or:
  • Be lethargic, vomit and have no appetite
  • Have pain when the abdomen is touched
  • Have a swollen lower abdomen.

How is FLUTD diagnosed?

The initial diagnosis of FLUTD is based on the identification of signs of lower urinary tract inflammation. The clinical signs displayed by the cat are often characteristic of FLUTD, but where there is doubt analysis of a urine sample in our pathology lab will confirm the presence of inflammation or blood. In some cases, x-rays may be required to assess if your cat is in a critical or life-threatening situation.

Treatment protocols:

If the bladder is blocked, a general anaesthetic is required to relieve the obstruction, flush out the urethra and place a tube (catheter) into the bladder to allow drainage. Intravenous fluids are usually required. This is usually followed by a period of hospitalisation in intensive care for several days.

When your pet returns home, it should be confined indoors for 3-4 days. Occasionally a reblockage occurs, so we need to ensure that your cat is able to pass urine daily by using a litter tray.

At home, the condition is managed by altering the diet. Only prescription veterinary diets are suitable to dissolve the crystals. There are no products available from supermarkets or pet shops that can do this. The prescription diet will also not be effective if any other food is given. Your pet should eat only the dissolving diet for 6 weeks, at which point the urine is again checked for crystals. If it is normal, the diet will be altered to a maintenance diet.

It is impossible to completely prevent diseases of the lower urinary tract. However, FLUTD is more common in cats that have a lower water consumption, and in cats that are inactive and obese. Keeping your cat lean and encouraging exercise may help in preventing FLUTD. As cats tend to drink very little, a higher water intake can be maintained by feeding at least some moist food rather than exclusively a dry cat food product.

Flea bite hypersensitivity (FAD)

FAD, or flea bite hypersensitivity, is a reaction to the bite of a flea. When a flea bites and dog or cat (or human) it injects a small amount of saliva into the skin. Normally this produces little more than a mild itching. Some cats and dogs can develop an allergy to this saliva, and, in these animals, even a single flea bite can cause intense itching and irritation. A severe flea infestation, where lots of fleas or flea dirt is found, can cause the same signs in non-allergic patients.

FAD is most commonly seen in the summer months when fleas are more prevalent but can occur even in winter because even low numbers of fleas can cause a reaction. Pets suffering from flea bite hypersensitivity will bite, chew and lick their skin continually.

Clinical signs:

Dogs typically attack their back, just above the tail, the back and insides of their hind legs, their belly and around the anus. This leads to scabs, hair loss, thickening and increased pigmentation of the skin and scaling and, eventually, to weepy skin and sores.

Cats are affected slightly differently. Some cats develop small scabs over their back or, in severe cases, over their whole body called ‘miliary dermatitis.’ Others lick until they develop bald patches, especially on their belly and the back of their hind legs.

How is FAD diagnosed?

Diagnosis is usually easily made by a visual examination. Conditions other than flea bites may cause similar signs, however, so we may recommend tests to confirm the diagnosis, especially when there is no evidence of fleas.

Treatment protocols:

FAD is not life threatening, but nor is it curable. In the short term, hypersensitive pets who have been exposed to fleas and are suffering from FAD need medication to relieve the itching, stop them scratching and give their skin a chance to heal. Some will also need antibiotics for infections that have developed in the damaged skin.

In the long term, all pets suffering flea bite hypersensitivity need to have their exposure to fleas minimised — permanently.

There are several excellent, easy to use products available that effectively kill adult and juvenile fleas both on your pet and in the environment. Where large environmental contamination with flea eggs exists separate environmental treatments may be recommended.

Other pets in the household also need constant treatment. Although they may not be allergic and no be affected, they do carry fleas which then can bite the affected pets. Please talk with our nurses regarding a specific flea prevention treatment program for your pets.

Heart disease

The heart is a unique organ, different from any other in the cat’s body. It consists of 4 chambers, all of which contain blood and are responsible for sending blood that is low in oxygen to the lungs to receive more oxygen, then sending this oxygen-rich blood throughout the body to all the tissues in need. A series of valves in the heart separate these 4 chambers, preventing blood from flowing where it shouldn’t.

In both cats and dogs, heart disease is mainly seen as a problem with the valves of the heart, or in the heart muscle itself.

Unlike dogs, however, cats rarely develop disease only in their heart valves. With felines, heart disease usually involves Cardiomyopathy, the name given to disease of the heart muscle. It is usually divided into two categories: thinning of the heart muscle (congestive cardiomyopathy); and thickening of the heart muscle (hypertrophic cardiomyopathy). In both cases, the heart fails to pump blood adequately throughout the body.

Clinical signs:

Symptoms of heart disease are usually only seen when the cat’s heart begins to fail.

Weakness, loss of appetite, weight loss, and difficulty breathing are the most common signs of heart failure in the cat.

How is heart disease diagnosed?

Diagnosis is made by physical examination, X-rays, electrocardiogram, and ultrasound of the heart. Most patients will have a heart murmur which can be readily heard by the veterinarian.

Treatment protocols:

Treatment for heart disease is most commonly by medications which assist in increasing the amount of blood pumped by the heart per beat, decreasing fluid buildup in the lungs and chest, and controlling any heart rhythm abnormalities.

Nutrient supplements added to the treatment plan may also help correct the underlying heart problem. Taurine is an essential amino acid required by cats, and has been found to be deficient in some cases of cardiomyopathy. Replacement of taurine can be quite valuable in treatment of these cases.

Hyperthyroidism

Hyperthyroidism is a relatively common disease seen in cats over 8 years old. In most  cases, it is brought on by a non-malignant growth of the thyroid gland, which causes an increased production of thyroid hormone. Hyperthyroidism is a progressive disease which, if untreated, will continue to worsen until the cat dies.

Clinical signs:

Cats with hyperthyroidism can exhibit some or all of a variety of symptoms which include:

  • weight loss
  • excessive appetite
  • poor hair coat
  • hyperactivity
  • vomiting
  • diarrhoea

How is hyperthyroidism diagnosed?

We will run a blood test in the hospital’s pathology lab to determine the cat’s thyroid hormone level. Additional blood tests should be performed concurrently to screen for other problems that might be present in older cats and which might influence treatment of choice and prognosis.

Treatment protocols:

There are a number of treatment options, each of which has advantages and disadvantages. We will discuss with you the treatment that provides the best possible outcome for your cat.

Medication: ‘Anti-thyroid’ drugs, which block the production of thyroid hormone, can be used to control the signs of hyperthyroidism. These drugs do not cure the disease, but they can effectively control it when given daily. This therapy requires giving the cat tablets every day (initially 2-3 times each day). Medication is the least complex treatment option but it depends entirely on you diligently giving your cat oral medication. If drug treatment is interrupted even for a day or two thyroid hormone levels will increase and symptoms will recur.

Surgery: Surgery can be performed to remove overactive thyroid glands, ‘curing’ the hyperthyroid state. As cats suffering hyperthyroidism are invariably older, there is an increased surgical risk. However, in the majority of cats without any concurrent medical conditions, the risk is minimal. Before surgery, we must first control your cat’s hyperthyroidism with oral medication. This takes 3-6 weeks in most cats.

In the majority of hyperthyroid cats, both thyroid glands must be surgically removed. Although this cures the hyperthyroid state, most cats will also require thyroid hormone supplementation — usually once a day — following surgery, to prevent hypothyroidism, a state of thyroid hormone deficiency (note the different spelling. Hypothyroidism is not the same as hyperthyroidism). These supplements are usually only required for a few months after surgery, but in some cases have to be given for the remainder of the cat’s life.

A serious potential post-surgical complication is hypocalcaemia, or low calcium levels in the blood. This can develop in about 5 to 10% of cats when the small parathyroid glands — located in and around the thyroid glands — are inevitably removed, or when their blood supply is temporarily damaged. Parathyroid hormone is important in regulating calcium levels in the blood. Cats must be kept in the hospital for 2-3 days following surgery so we can watch for signs of hypocalcaemia, which include muscle tremors, stiffness or convulsions. Successful treatment of hypocalcaemia includes daily administration of vitamin D and calcium. In the majority of cats, the medication can be discontinued within 2-3 months.

Radioactive Iodine: This is the least invasive treatment for your cat. Radioactive Iodine (I131) is given as a single tablet or injection. It is effective in correcting the hyperthyroid state in over 85% of cases with a single dose. I131 selectively irradiates abnormal thyroid tissue while preserving the normal thyroid gland. Because of radiation safety requirements your cat will need to be referred to a specialist clinic  and boarded for 7 days after treatment.

Hypothyroidism may also develop following radioactive treatment. However, even though the blood levels of thyroid hormone may be low, the cat often shows no clinical signs of disease and may not need to receive supplementation. Hypocalcaemia is not a complication with this form of treatment.

 

Obesity

In recent years we have seen what can only be described as an explosion in the number of obese cats and dogs coming into the hospital. Like diabetes, a serious illness in which obesity can be a major causative factor, this increase in the number of ‘fat pets’ we see is mirroring trends in the human population. And for the same reasons. Put simply, too many pet owners are either feeding their pets too much of the wrong foods, and/or failing to provide them with enough healthy exercise.

Strictly speaking, obesity is not in itself an ‘illness.’ You can call it a syndrome, a condition of excess body weight, defined as fat accumulation, where the body weight is 15% or more above the ideal weight, and great enough to cause harm to health or wellbeing. Obesity may not be an illness itself, but it is a debilitating condition that is linked to a wide range of health issues with serious consequences for you pets. Fortunately it is also very treatable.

Obesity is caused by three main factors:

Overeating. While cats and dogs can regulate the amount they eat by their energy needs, this ‘natural’ regulator can be overridden if the flavour of the food on offer is very desirable, and it is presented more often or in larger quantities than necessary.

Insufficient exercise. Animals tend to become more sedate as they age, when ill or after neutering. The less the animal exercises, the less calories they burn, and therefore the less calories they should be eating.

Decreased energy requirements. Every animal (including humans) has his or her own metabolic rate, which can be influenced by many factors including hormones, climate, reproduction status, and body weight. If an animal’s metabolic rate is decreased by any one of these factors, the body automatically stores the energy it is no longer burning as fat.

Other risk factors include gender, breed, and owner lifestyle but, basically, the root cause of obesity is the unnecessary consumption of more calories than the animal needs.

Obesity has been linked to many adverse effects and serious illnesses in both dogs and cats. Common problems caused or exaccerbated by obesity include:

  • Diabetes Mellitus (Type I and Type II diabetes).
  • Hypertension, or high blood pressure, caused by the excess workload on the heart and lungs.
  • Neoplasia. Many recent studies have found the incidence of cancer is dramatically higher in obese patients compared to lean patients.
  • Decreased immunity. The body’s ability to fight disease is greatly reduced.
  • Hepatic Lipidosis/Fatty Liver. Fatty accumulation in the liver of obese cats that stop eating (e.g., if they are unwell) can cause severe illness and possibly death.
  • Osteoarthritis is aggravated by obesity because of the extra weight on the joints.
  • Skin infections and matted hair may result from decreased grooming in obese cats.
  • Bladder infections can result if excess weight prevents a cat from grooming its hind end.
  • Obese cats may suffer from increased rates of feline lower urinary tract disease.
  • Obesity may accelerate and exacerbate kidney disease and heart disease. Note that obesity, by itself, usually does not cause heart disease.
  • Obese pets may suffer respiratory and breathing problems.
  • Obese pets are more likely to suffer complications during anaesthesia and surgery.

Just about all of these conditions can be avoided by keeping your pet’s weight at an optimum level. To assess the ideal weight for a dog, run your hands firmly down the animal’s sides and spine. You should be able to feel the rib cage and individual vertebrae, but not see them. There should also be an indentation between the rib cage and the hips — this is the dog’s waist line.

Testing a cat’s bodyweight is slightly different as the fat often hangs around the groin and belly area. The rib and spine test for dogs can sometimes be accurate, although it is important to also check for a ‘pouch’ of fat hanging around the abdomen.

Treatment protocols:

‘Treatment’ for obesity is, happily, not difficult. If your pet is diagnosed with obesity, you need to take control of his or her food intake. A balanced diet is essential for good health. But please note that we are not talking about just putting you cat or dog ‘on a diet’ here. Eliminating obesity requires a permanent lifestyle change.

The easiest and most cost efficient way of dieting your pet is with a high quality, balanced, commercial weight control premium pet food. We are happy to advise you on the most appropriate nutrition plan for your pet. You will also need to monitor the entire food intake, which means fewer treats, and only treats of the low calorie or low fat variety. Regular weigh-ins at the hospital are recommended so we can chart the progress and make adjustments if necessary. Maintaining goal weight when reached is simply a matter of adjusting the calorie level slightly and, once again, a balanced weight control diet is best.

Our recommendations for using these commercial preparations is not so we can sell you more pet food. If you can prepare complete, balanced low calorie meals at home, that’s fine. But it’s almost impossible for an individual to do that. Achieving balanced nutrient diets to bring an animal back from obesity requires a scientifically formulated diet.

Many people with obese animals tell us that they only feed their pets once a day. Perhaps, but it is not the number of meals each day, or even the quantity of food, the problem lies in the quantity of calories they are given each day. It is better for pets to get two smaller meals a day rather than one large one, as this increases the body’s metabolism, and helps to burn calories.

There’s no getting around it — if your pet is overweight, it must be getting more nutrition than it requires. The body stores only the energy it does not need. In short, there are only two ways to defeat obesity — reduced calorie intake, and higher levels of appropriate exercise, preferably a combination of both.

It is of course more difficult to get a cat to exercise, unlike a dog you usually can’t compel them to exercise. However, cat parents can encourage increased activity by offering toys that stimulate hunting instincts, and by encouraging the cat to both play and to spend time outdoors. Serving their food in a remote area of the house, or the end of the garden may reduce food intake because of the extra effort required to obtain food, and stimulate activity during the cat’s trips to and from their food bowl.

For both cats and dogs, the benefits of eliminating obesity and getting your pets back to a healthy body weight are so great that a caring owner will remain dedicated to the cause, regardless of the frustrations.

Ringworm

Ringworm (dermatophytosis) is not actually a worm, it’s a fungal infection that can affect the hair, skin or nails of both cats and dogs, and humans. The most contagious skin infection in cats, it can easily be transmitted from cats and dogs to other animals, and also to, and from, people.

Several different fungi can cause ringworm but the vast majority of cases in cats and dogs are caused by Microsporum canis, Microsporum gypseum or Trichophyton species. The fungus is commonly found in soil and often affects gardeners, who can transmit it to their pets. Once infected, an animal will shed spores into their living quarters, on brushes, bedding, furniture, or anything that has been in contact with an infected animal, and into the wider environment. Humid, warm environments encourage growth of the fungus.

Cats can be asymptomatic carriers, carrying the fungus but showing no signs of the infection themselves, and transmitting the fungi to any other pets they come in contact with. Animals thought to be chronic carriers can be routinely cultured to determine if they are carriers. Where a cat is confirmed as a carrier, if you have other pets in your home, the only way to minimise the spread of ringworm through all of your pets is by ensuring that the carrier cat is permanently kept outside.

Because ringworm spores can survive for long periods in the environment your cat can contract ringworm from just about anywhere other dogs or cats have been. Fortunately, most healthy adult cats have a degree of natural resistance to ringworm and never develop symptoms from the fungus. Young cats under a year old are most often infected. Older cats with a suppressed immune system caused by disease are also more susceptible to contracting the disease. Senior cats, free-roaming cats, and those under stress, malnourished or suffering other diseases such as parasites or viral infections are also at increased risk.

Clinical signs:

The classic symptom of a cat infected with ringworm is a small circular lesion, or spot, that is red, hairless, and often scaly in the centre. Small pustules are often also found in the lesion, which are most common on the head, ears, and tail. In some infections the fungus will not be in a circle and can spread across the face, lips, chin, or nose and look like an autoimmune disease or other generalised skin disease.

Hair loss may be mild or severe. In some cases the first sign may be excessive shedding, and hairballs may occur when large parts of the body are affected. Scratching at the ears is also common. Ringworm can also occur in the nails, often causing them to grow malformed.

How is ringworm diagnosed?

Ringworm can most easily be diagnosed by looking at a lesion under a UV lamp. The infection will glow a fluorescent color when exposed to UV light. However, not all the species of the fungus will fluoresce. A healthy animal may have spores on his or her coat that fluoresce, but not have an active infection.

If there is any doubt we will confirm, or eliminate, a ringworm infection by collecting scales and crust from the skin and coat and performing a fungal culture, but in most cases an infection is fairly easy to identify.

Treatment protocols:

Treatment is relatively simple. A fungal shampoo such as F10 or Malaseb, together with the application of a fungal cream will generally quickly clear up the infection.

Some cases of ringworm are more resistant to treatment and will also require the use of an oral antifungal medication.

Because the ringworm fungus can survive for such long periods in the environment, and be transmitted to or by humans, one of the most important elements of treatment, and the prevention of future infections, is hygiene. It is important that you thoroughly wash your hands after treating your pet, and after playing with it. You should also thoroughly clean wherever an infected animal has been to eliminate the spores.

Spores are very light and are carried in the air so wherever there is dust and hair, there may be spores. Vacuum and mop hard floors, and steam clean carpets. All grooming tools, bedding, kennels, cat carriers, and cages should also be thoroughly cleaned and disinfected.

Ringworm is a disease where prevention is definitely better than cure. If you have cats, be very careful about bringing a new kitten into your household. Cat shows, kennels, and grooming facilities can also be a source of infection. Breeders of Persians and Himalayans need to be especially cautious about bringing any new animal that has not been cultured into their facility. If any sign of ringworm is seen, make sure you isolate the infected cat and call the hospital for advice.

Solar dermatitis

Solar dermatitis, or actinic dermatitis, is a skin disease caused by exposure to the sun. It’s seen mostly in cats with white, non-pigmented areas of skin. The ear flaps, nose and lower eyelids are the most commonly affected areas. Left untreated, some cases progress to a malignant tumour, or squamous cell carcinoma (SCC).

If you have a cat with skin predisposed to solar dermatitis, you can minimise the risks by keeping the animal out of sunlight, particularly between the hours of 9am and 4pm on sunny days. An animal specific sun block cream (available at the hospital) may also be used on affected areas, although most cats will rapidly groom or lick this off.

Clinical signs:

In the early stages, the skin may look pink and scaly with some degree of hair loss. As the condition progresses the affected area becomes crusted and eroded. The cat is often irritated by the lesions and, if the ears are affected, it may shake its head and cause bleeding from the ear tips.

How is solar dermatitis diagnosed?

Initial diagnosis is by physical signs. If we are concerned the condition may be progressing to a squamous cell carcinoma, we will take a biopsy from the affected area, which will be microscopically examined to see if malignant cells are present.

Treatment protocols:

For cats with an SCC, surgical removal of the affected area and a wide surrounding margin is the most effective treatment. This is usually straightforward. If the ears are affected complete removal of the ear flaps may be necessary. This has no detrimental affect on the cat and the final appearance is often quite acceptable. Surgery is more difficult when the nose or eyelids are affected. Where it is not possible to remove all the diseased tissue, additional forms of therapy may be needed such as radiotherapy. In advanced cases, with nose involvement, removal of the nose is possible as a last resort.

The prognosis following is usually good, especially when it is the ears that are involved, and particularly if the disease if discovered early. If all the diseased tissue is not adequately removed, the tumour will recur at the operation site. This can be a problem if the nose or the eyelids are affected and is the reason additional therapies are often used in these cases.

This type of tumour doesn’t spread very rapidly to other parts of the body so removal and treatment of the site of SCC’s often produces complete remission, as long as the entire tumour is removed.

  • Addison’s Disease (Hypoadrenocorticism)
  • Arthritis
  • Atopy
  • Bloat (gastric dilation)
  • Bone cancer (Osteosarcoma)
  • Bowel disease
  • Collapsed trachea
  • Conjunctivitis
  • Cruciate Ligament Rupture
  • Cushing’s Disease (hyperadrenocorticism)
  • Dental disease in dogs
  • Diabetes
  • Discoid Lupus Erythematous
  • Dry eye (Keratoconjunctivitis sicca)
  • Ear infections
  • Entropion (inverted eyelid)
  • Epilepsy
  • Flea bite hypersensitivity (FAD)
  • Gastroenteritis
  • Heart disease
  • Heartworm
  • Hookworm
  • Hotspots
  • Hypothyroidism
  • Kennel Cough
  • Luxating patella
  • Mange (demodicosis)
  • Obesity
  • OCD (Osteochondrosis dissecans)
  • Paracetamol poisoning in dogs
  • Pancreatitis
  • Parvovirus
  • Pyometra
  • Sarcoptic mange

Addison’s disease, or hypoadrenocorticism, is a hormonal disorder. Relatively uncommon, the disease is caused by the abnormal production of cortisol or aldosterone, or both, which are normally produced by the adrenal glands. If these glands are diseased or damaged the level of hormone required for healthy function is no longer produced. The pituitary gland — another hormone producing gland, located close to the brain — can also influence the adrenals. If the pituitary gland is not functioning correctly this can also cause a decrease in the level of cortisol and aldosterone.

Note: Hypoadrenocorticism (Addison’s disease) should not be confused with hyperadrenocorticism, or Cushing’s disease.

SEVERITY: Variable to Severe. In dogs suffering an acute Addisonian crisis the disease can be life threatening. Although this is fairly uncommon, if you suspect Addison’s book a veterinary consult at Alpine Animal Doctors as soon as possible. See symptoms.

Some of the signs seen in dogs presenting with Addison’s are lethargy and weakness, loss of appetite, vomiting, weight loss, diarrhoea, shaking, increased thirst and increased urination.

The signs are variable and, particularly in patients with the chronic form of the disease, may be limited. In dogs suffering an acute Addisonian crisis symptoms may be severe and life threatening. As many of the symptoms of Addison’s can very easily be confused with non-specific diseases, particularly gastrointestinal disease or kidney disease, it’s very important that dogs showing these symptoms be examined by a vet as soon as possible to establish the true cause and to hopefully rule out Addison’s Disease.

The average age at which dogs are affected is about four years old but Addison’s can strike dogs as young as a year old as well as older pets, up to twelve years of age. Female dogs seem to be at higher risk than males.

Fortunately, Addison’s is an uncommon to rare disease in dogs, and sometimes cats. Some breeds seem more predisposed, including Great Danes, Rottweilers, Poodles, West Highland White terriers and Wheaten terriers.

A general blood test will alert us that all is not right. It is common to see electrolyte disturbances, and sometimes anaemia (low red blood cell count) or even liver enzyme elevation. A blood test will usually also reveal any disturbances with kidney function.

Although these results are indicative of Addison’s they do not provide confirmation and we will usually run a second test — an ACTH stimulation — to definitively confirm the presence of the disease. This involves taking a second blood test then injecting the patient with a hormone called ACTH, designed to stimulate the adrenals to produce cortisone. An hour later a follow-up blood test is taken to measure how much the cortisone levels have increased in the blood stream. If they have not increased at all it suggests that the adrenal glands are not working and your pet has Addison’s disease.

An animal confirmed to have Addison’s disease will need life long hormone replacement medication, and may need increased doses during periods of stress such as travel, hospitalisation and surgery.

We will usually need to take weekly baseline blood tests until their hormones are stabilised. To check their electrolytes and kidney function they will then need a blood test once a month for the first 3-6 months, then a routine blood test every 3-12 months.

Unless Addison’s has been caused by a pituitary or adrenal cancer the vast majority of patients with hypoadrenocorticism have a good to excellent prognosis following stabilisation and treatment. With proper care most Addison’s sufferers will continue to live active and healthy lives.

Osteoarthritis (also called degenerative joint disease  or DJD in veterinary medicine) is a progressive deterioration within the joints, where degeneration of the cartilage lining the ends of bones, changes of the underlying bone at the joints, and changes of the joint capsule that secretes the joint fluid leads to constant and often severe pain.

Osteoarthritis affects both dogs and cats and arthritic changes are seen in most older patients at some stage in their lives, but the disease can also affect young animals if associated with inherited or growth disorders such as hip dysplasia or patella luxation, or sometimes as a result of trauma such as a fracture, dislocation or ligament injury.

In older patients long term exercise and simple ageing are usually the cause. Working dogs and overweight pets are more likely to suffer osteoarthritis at a younger age. Other possible causes are immune-mediated arthritis — rheumatoid arthritis — infectious arthritis or a tumour.

SEVERITY: Moderate to Severe. All dogs with arthritis will be in pain. In advanced cases pain will be chronic and debilitating. See treatment.

Intermittent lameness that slowly becomes more frequent and severe is the usual sign. Stiff movement, difficulty rising and difficulty going up and down stairs are also common signs. Exercise can result in increased lameness, as can cold weather and lying down for long periods. As these signs develop very gradually it is often easy to accept them as signs of old age, rather than as signs of your pet suffering the treatable pain associated with osteoarthritis.

Because arthritis usually develops so gradually, many pet parents find it hard to remember how their pet was before arthritis set in. Your pet is definitely in pain if they are showing signs of limping, are sore after exercise, get up or down gingerly, or can’t or won’t climb steps. The pain is constant and our pets very stoic, so these signs are the only way to assess how painful the arthritis is. The pain should be treated. Response to medication in most cases is excellent, and can give an older pet a new lease on life.

X-rays are often taken, and in some cases analysis of fluid from within a joint or biopsy of tissues within the joint may be needed. For the most part though, a diagnosis can be made from a simple examination and our knowledge of the history of your pet.

The degeneration within the joint is permanent and will continue to develop, so treatment of osteoarthritis is primarily palliative, i.e., designed to relieve the clinical signs with painkillers, and in some cases slow the progression of osteoarthritis.

Two types of medical treatment exist. Straight pain relief is available in oral tablets or drop form. This type of medication gives very good results but needs daily or twice daily dosing.

A second type of medication frequently used is Pentosan, either via a course of 4 injections given at 7 day intervals or by capsules. Improvement with this treatment is usually gradual over the 4 weeks, so additional pain relief may also be given for the first 1-2 weeks.

These injections treat the pain of osteoarthritis, but they also slow the ongoing degeneration by protecting the cartilage, and improving the quality and amount of joint fluid in all the joints of the body. An excellent response in usually seen with this medication. Duration of effect is variable, often related to the initial severity of signs.

Further treatment is tailored to the individual; some patients may be extremely comfortable for 6-12 months before requiring a repeat course of injections, others may deteriorate after a couple of months and require a single injection to be given every 1-2 months. Some animals need ongoing pain relief as well as the injections.

Sometimes when the diagnosis of osteoarthritis is made an underlying cause is also diagnosed, such as hip dysplasia, cruciate ligament rupture or patella instability, which may need surgical treatment. Surgical options can range from the relatively routine (CCL) to advanced solutions such as custom made implants to fully replace arthritic joints.

Diet plays a large part in ongoing treatment. There are a number of ‘joint support’ diets available, all of which contain elements which can help reduce the effects of the disease. These can be green-lipped mussels, traditionally used in treatment of arthritis for the past 25 years and/or glycosaminoglycans, a vital joint nutrient. These formulations not only promote joint health they deliver a nutritionally balanced diet for overall patient well-being. One of the most important things you can do for a pet suffering from arthritis is to not overfeed — keep them lean.

Being overweight puts added pressure on joints, increasing pain and degeneration. It’s very easy for an arthritic patient to become overweight as their activity levels fall due to the pain. Overweight pets need to be on strict diets — please talk to us for advice about this.

Activity should be restricted to a level that doesn’t aggravate the condition. If your pet is sore after a walk, try shorter walks. If chasing the ball makes things worse, play for less time, or not at all. Swimming is an excellent form of exercise as it is non-weight bearing and puts less stress on joints, but cold aggravates osteoarthritis so a warm pool is needed.

Cold should be avoided as much as possible. Arthritis will always be worse in winter. If your pet is allowed to sleep inside this will help. Try to minimise exposure to cold by providing thick warm bedding, ideally not on concrete), a warm sheltered area and consider a warm dog coat for winter.

Atopy is an allergic condition that can affect both dogs and cats. It is thought to be inherited, and is usually first seen between 6 months and 4 years of age, although it’s possible from 3 months to 7 years.  Susceptible animals become allergic to pollens — grasses, trees, weeds — dust mites, mould and fungal spores, wool, flea saliva antigens, and become intensely itchy when exposed to their specific allergen(s). The condition is usually due to a range of allergens.

SEVERITY: Moderate to Severe. Requires ongoing treatment to ease symptoms.

The signs of atopy are generalised all over itching. Dogs are particularly itchy around the ears, under front legs, groin and inside the hind legs, feet, around the eyes and on the belly. Mild cases have only one or two affected areas, more severe cases involve them all. Cats are usually most itchy around the head.

Initially the itch may wax and wane with seasonal changes in pollen numbers and types. Over time this seasonal pattern often becomes permanent. The intense itchiness causes the animal to chew, scratch and bite resulting in inflammation/redness of the skin, hair loss, brown saliva staining of the fur from licking (especially seen on front feet), scaling, crustiness, darkening and thickening of the skin, ulceration and secondary skin infections.

Inflammation and redness of the eyes may develop from animals rubbing their irritated eyes, and recurrent ear infections are also very common as the skin lining the ears is affected.

A diagnosis of atopy can usually only be made after we have seen a pet more than once with itchiness, or in severe cases involving most of the usual affected areas of skin. Other possible causes of the itching are food hypersensitivity, flea allergic dermatitis, mange mites, yeast infection of skin, or contact irritation which must be ruled out.

We will examine your pet and usually take skin scrapings, ‘sticky tape’ preparations, and/or ear smears to examine under the microscope, or test in our pathology lab. In some cases we ask you undertake a food allergy trial to rule out this as possible or contributing cause of the itchiness.

Treatment is based on the severity of clinical signs. Mild cases can often be managed with oral antihistamine tablets. We may have to work through a few different types before we find one that works well for your pet, trialling each for around 10 days. Advantages of using antihistamines include them being non-prescription, so once an effective medication is determined they can be purchased over the counter from us without a consultation. They are administered as needed not as a course, and have few side effects apart from drowsiness — which may be seen initially but most patients develop a tolerance to this after a short time — and increased appetite.

More severe or chronic cases with inflamed irritated skin are usually treated with corticosteriod injections and tablets. These settle the inflammation and itch, allowing healing. Short term use of these prescribed medications is fine, however high doses and long term use can result in side effects of  immune suppression with an increased susceptibility to infections, increased hunger, thirst, and urination, muscle weakness, thin skin and hair loss.

If secondary skin infections are present (which are common), antibiotics and specialised shampoos may also be prescribed.

Most atopic pets are also extremely sensitive to flea bites, so a regular good quality flea treatment like monthly Frontline or Advantage is important. We recommend all atopic patients and their housemate pets be treated. Although other pets in the house may not themselves be affected by flea bites they carry fleas which will bite the affected pet. Many patients have their overall itch level reduced by good flea control, including oral fatty acid supplements in their diet, or by using special anti itch shampoos and conditioners such as Epi-soothe, Resi-soothe or Aloveen.

Ear infections are frequently present and need to be treated with ear drops or oral medication.

Atopic patients have the option of referral to specialist veterinary dermatologists. We sometimes recommend referral for severe cases, especially those with a year round pattern, and those that are difficult to control. Dermatological specialists may perform a skin patch test to identify the allergens involved, and try to reduce your pets response to these through hyposensitisation. Hyposensitisation involves a course of injections of the allergens at gradually increasing doses over a long period, to ‘train’ the pets immune system to respond normally rather than excessively when exposed to these naturally in the future.

The condition described below — gastric dilation, or bloat — is a critical, life threatening emergency. If you suspect your dog is suffering from bloat call the hospital immediately (5756 2444), and get your dog here as quickly as possible. The sooner medical treatment is begun the greater the dog’s chances of survival.

Gastric Dilation and Volvulus, or GDV, is a syndrome of dogs in which the stomach dilates and twists around itself. Most people know the condition as ‘bloat.’ A serious, life-threatening condition, GDV is seen mainly in large or deep chested breeds such as Labradors, German Shepherds, Rottweilers, Great Danes, Saint Bernards or Alaskan Malamutes. On rare occasions it can affect Daschunds and other smaller dog breeds. It can occur at any age but is most common in middle aged to senior dogs.

The exact cause is unknown. Current research leads us to suspect that it is related to the way in which the stomach digests food. Normally, the stomach contracts in an orderly and systematic fashion when digesting food. With bloat it is believed that these contractions occur in a disorderly fashion, and the pyloris — the area leading to the small intestine — is shut off, preventing food moving into the intestine. Large amounts of air are also swallowed and, with the pyloris closed, that air cannot pass through out of the stomach.

In many cases, that leads to the stomach twisting on itself. Rapid overeating followed by exercise shortly afterwards is definitely a contributing factor. Breeds prone to GDV need to be trained to eat their food slowly, and exercise should be either before food, or at least an hour afterward.

SEVERITY: Critical. Requires urgent veterinary intervention

Dogs with bloat are usually in acute pain and distress, pacing and restless and will display non-productive retching or vomiting, and excessive salivation. Progressive enlargement of the abdomen, which can start to feel like a tight balloon, is a sign bloat is well advanced. Dogs with bloat are also usually weak and depressed. Without treatment this condition can lead to death within a few hours.

Bloat is potentially fatal for a number of reasons. As the stomach enlarges and twists the blood supply to the stomach is cut off, causing the tissue to begin to die and leading to the release of harmful toxins throughout the body. The expanding stomach also puts pressure on the lungs, preventing them from expanding and depriving the body of oxygen. As the stomach expands it also puts pressure on the major blood vessels (vena cava) resulting in disruptions of blood flow to various other body organs.

Diagnosis is often based initially on breed predisposition. On examination, the stomach will be obviously enlarged and very firm and the dog will be in considerable discomfort. An x-ray is performed and usually reveals a grossly enlarged stomach full of air. Sometimes the twist in the stomach can also be seen on the radiograph.

We’ll begin treatment by placing the dog on intravenous fluids to stabilise the patient and counteract shock. Once stabilised, a long tube will be passed through the mouth into the stomach to attempt to decompress the stomach. In many cases decompression is not possible using this method, but if decompression can be achieved this way the patient is better stabilised before the next step — surgical correction of the problem.

Whether the stomach is first decompressed or not, surgery must be performed to decompress (if not achieved with a stomach tube) and untwist the stomach. Usually the stomach is then permanently stitched to the side of the abdominal wall so it is held firmly in place and cannot twist again.

In some cases, the stomach will only fill up with air and not twist on itself. These dogs still need surgery to secure the stomach position and prevent it from twisting in the future. Without surgery there is an 80 per cent chance of a recurrence of bloat, compared to 3-5 per cent after surgery.

In cases where the blood supply to the stomach has been cut off for too long the stomach will look very blue or black in surgery. Sadly, these dogs will not survive and the kindest thing is to euthanise them.

Patients who have been treated for GDV require intense monitoring for at least 36 hours after surgery. A common problem post-surgery is cardiac arrhythmia — where the heart beats irregularly — caused by the heart receiving reduced oxygen during the bloating episode, causing parts of the heart muscle to be damaged, and also due to some of the toxins released after the stomach position is corrected.

Bloat is an extremely serious condition, and frequently fatal. In most cases where we cannot save the patient it’s often because the dog’s owner was unaware of, or failed to recognise, the initial symptoms and the patient reached the hospital too late for corrective surgery.

Dog parents should familiarise themselves with the signs of bloat and act swiftly if they suspect their dog is suffering from the condition. The sooner medical treatment is begun the greater the dog’s chances of survival. With prompt and appropriate treatment most dogs suffering from bloat that undergo surgery have a  55-70 per cent chance of recovery.

Osteosarcoma is a primary cancer of the bone. It destroys normal bone, leaving in its place abnormally brittle and fragile bone prone to fracture. Osteosarcoma is an aggressive cancer that spreads rapidly to other parts of the body, usually long before it is even detected.

Large and giant breed dogs have the greatest incidence of bone cancer in their limbs. The Rottweiler In particular seems to be over-represented as a common breed predisposed to bone cancer. The average age of dogs with osteosarcoma is 7 years. Males are more commonly affected than females. Smaller dogs — and cats — can also get bone cancer, but this is much rarer.

Osteosarcoma is a terminal disease. There is no cure. The mean life expectancy of a dog with osteosarcoma is approximately 3 to 4 months. See Treatment Options.

SEVERITY: Terminal.

Symptoms can be difficult to recognise and easy for an owner to confuse with less serious issues. Dogs affected with appendicular tumours are often lame. Sometimes the limb is enlarged and firm, as well as painful. Less commonly they may be reluctant to eat or walk and can sometimes bleed from the tumour surface.

Osteosarcoma can spread quickly to the lungs and other organs so coughing and breathing difficulties are a long term complication. In about 80 per cent of cases, by the time the owner realises something is wrong and brings their pet to the hospital, it has metastasized to the lungs or heart.

While an early diagnosis cannot help us stop the cancer it can assist us in helping to control the pain and make your pet’s last months more bearable. It is worth owners being aware that what looks like a simple limp can in fact indicate something much more serious and always needs checking out.

Digital Radiography is the primary diagnostic tool. Radiographic signs of osteosarcoma include cortical lysis — where the outer margins of the bone start to dissolve —periosteal bone proliferation, which makes the edges of the bone look blurry on the radiograph, and soft tissue swelling.

Radiography does not always provide a confirmed diagnosis but when assessed with the history of the pet, its age and breed it does make a bone cancer diagnosis more likely. CT scans can provide a more accurate diagnosis but are rarely necessary to confirm Osteosarcoma.

Bone cancer is very painful and the patient will need powerful pain relief on a daily basis throughout their last months. Surgical intervention — usually the amputation of an affected limb — is possible, but amputation does not necessarily prolong the animal’s life, although it does make the pain more bearable.

Chemotherapy can extend the life span of most dogs suffering bone cancer, by approximately one year. They will then usually sucumb to the illness, or need to be euthanised for humane reasons. The drugs used in chemotherapy are powerful and aggressive and, generally, come with a cost to the dog’s quality of life.

Inflammatory bowel disease is a chronic disease of the intestinal tract, occasionally also involving the stomach. Most dogs affected by IBD will have a known history of recurrent or chronic vomiting and/or diarrhoea. During these episodes the dog may lose weight but appear generally normal in other ways. As a rule most affected dogs eat well, or even have an increased appetite, leading the owner to assume the vomiting is caused by unrelated factors.

The cause of IBD is poorly understood, there may be several factors. Whatever causes the disease, the end result is that the lining of the intestine is invaded by inflammatory cells. An allergic-type response is then triggered within the bowel lining, severely interfering with the dog’s ability to digest and absorb nutrients.

Diet and dietary components are thought to play a role in initiation of the disease in some dogs. In other cases bacterial proteins may be involved. In most instances an underlying cause is never identified.

SEVERITY: Chronic. Requires long term treatment.

The only clinical signs are persistent and recurrent vomiting and/or diarrhoea. The occasional bout of vomiting or diarrhoea every now and then is normal in dogs. Such episodes should not be assumed to be inflammatory bowel disease.

However, if your dog has frequent bouts of vomiting and/or diarrhoea for no obvious reason, i.e., they have not eaten any rubbish or scraps that may be off, and otherwise seems healthy and has a good appetite it is wise to have your pet checked for signs of an underlying problem.

Blood tests are usually taken to establish if digestion is normal and whether the digested food is being absorbed correctly, or if there is an exocrine pancreatic insufficiency. Depending on the results, before moving to more invasive treatments, we may ask you to trial specific diets based on which part of the bowel appears to be most involved. These diets are specially designed for each patient and may include hypo-allergenic foods, low residue diets or high fibre foods.

If the blood tests reveal that bacterial overgrowth is present we may also suggest drug trials involving controlled administration of specific drugs and gauging improvement.

Finally, it may be necessary to obtain biopsy samples from the bowel, either using a flexible endoscope or, in some cases, following an exploratory minimally invasive laparotomy surgery, where the bowel is examined for signs of disease and portions of the diseased tissue are secured for further tests.

Once a confirmed diagnosis of IBD is made the dog is placed on a hypoallergenic, low residue or high fibre diet for eight weeks or more. This helps identify the contribution of dietary components to the problem. Although this is not a common cause of the disease it is easier to treat if an acceptable food is found. If the dietary trial does not offer any improvement, medication is used to control, but not cure, the problem. Since not all dogs respond to the same medication, a series of drugs may be necessary.

Once we have established the most effective and appropriate diet or medication regime many dogs with IBD are maintained on these for life, although drug dosages may eventually be decreased. Occasionally, a dog will be able to cease drug therapy.

Most dogs do well on these treatments for many years. Others require regular alterations in therapy. Unfortunately, a few dogs will ultimately become totally resistant to treatment. Some severe forms of canine inflammatory bowel disease will eventually progress to incurable intestinal cancer.

Collapsed Trachea is a condition in which the tracheal cartilage sporadically loses its structural integrity, leading to a persistent honking cough. We do not fully understand how the condition develops. Because tracheal collapse is mostly seen in certain breeds of dogs, notably Chihuahuas, Pomeranians, Shih Tzus, Lhasa Apsos, Toy Poodles, and Yorkshire Terriers, a genetic factor is almost certainly involved, but an injury to the throat can also be a cause.

The trachea, or the windpipe, is an important structure that connects the upper airway — the throat — to the lower airway and the lungs. A normal trachea is tubular in shape and made up of a series of C-shaped rings of cartilage connected by a flexible membrane. If these cartilage rings and connective tissues begin to chronically flatten during inhalation or exhalation, the condition is known as tracheal collapse.

Very simply, the trachea can be visualised as a flexible straw. Pressure is applied to it as the dog breathes in and out. As long as the ‘straw’ retains its strength and structural integrity, airflow is unrestricted. However, once it collapses in one or more areas, the structure is permanently weakened and may subsequently collapse more easily and more frequently.

Studies have shown that the tracheal cartilage in dogs with chronic tracheal collapse tends to be deficient in glycosaminoglycans, the building blocks providing structural rigidity to connective tissues. The condition is usually seen in older animals, although it can occur in young dogs.

SEVERITY: Chronic. Requires long term treatment.

There are two types of trachea collapse: cervical, which occurs in the throat area, and intrathoracic, which occurs inside the chest cavity. Cervical collapse occurs during inhalation and intrathoracic collapse during exhalation. It is possible for the collapse to occur in both places simultaneously. Both are characterised by a classic honking cough, often called a ‘goose honk’ cough because of its distinctive sound.

The initial signs are usually a mild cough, becoming more persistent. Coughing may be more pronounced during the day, and is mostly seen during or after exercise, excitement, and stress, or from pressure on the trachea from a leash or choke chain. It can also affect dogs at night.

Coughing may be short lived initially but will gradually increase and episodes can frequently last hours or days, leaving the dog exhausted.

A grading system to document the severity of a tracheal collapse was devised by Drs. Tangner and Hobson to aid in determining the best way to treat the collapse. There are four stages of collapse: A grade I classification is characterised by a 25% or less occlusion of the tracheal lumen. Grade II is 25 to 50%, Grade III 50 to 75% and Grade IV is 75 to 100%. Dogs with a Grade I or II collapse are usually good candidates for medical management of the condition.

We will always consider tracheal collapse when a dog, especially one of the breeds listed in the summary, starts to show signs of a chronic cough.

Often, very light pressure over the trachea during a physical examination can raise suspicion of collapsed trachea in a small dog with a persistent dry cough, and a tracheal x-ray may identify the trachea and its shape. The most effective diagnosis is either via an endoscopy and/or a radiographic fluoroscopy of the tracheal area.

A collapsed trachea changes its diameter during the respiratory cycle so we need to see the trachea during both inspiration and expiration. This is best achieved by carrying out an endoscopy. A flexible tube with an eyepiece — or sometimes, a video camera — is inserted into the trachea allowing us to watch the trachea during inspiration and expiration, and to see any abnormal collapsing. A radiographic fluoroscopy — a machine which makes a ‘radiographic video’ — also allows us to view the trachea throughout inspiration and expiration.

Many dogs with collapsed trachea will also have heart disease, which can also be the cause of a persistent cough, so the heart is usually evaluated when carrying out a diagnosis for a collapsed trachea.

The preferred treatment for tracheal collapse is to medically manage the symptoms through weight reduction and maintenance, stress and exercise restriction, and drug therapy. Drug therapy may include antitussives (cough suppressants), anti-inflammatories, bronchodialators, sedatives, and antibiotics.

Antitussives, most commonly hydrocodone or butorphanol tartrate, are used to reduce the rate of coughing. Anti-inflammatories can help minimise airway inflammation caused by the trauma of the chronic coughing. Bronchodialators, usually theophylline based, are used to dilate the pulmonary airways, which helps decrease intrathoracic pressure during exhalation, hopefully decreasing the degree of tracheal collapse. Antibiotics can be used when the possibility of tracheal infection, known as bacterial tracheitis, exists.

Sedatives may be used in cases where antitussives fail to control coughing spasms. We have had some success using very precisely calculated low doses of ACP once a day during episodes, which have the effect of letting a dog suffering a severe coughing episode rest and sleep without coughing, allowing time for the airway inflammation to reduce.

None of the medically managed protocols are a cure. The objectives are to reduce the incidence of episodes of tracheal collapse, and to increase the intervals between collapses.

If it becomes impossible to maintain the condition medically there are surgical options. One that has had some long-term, repeatable success is the application of an extraluminal prosthesis such as a spiral prosthesis or a total ring prosthesis. The prosthesis is applied to the outside of the trachea in order to help it hold its normal cylindrical shape.

This type of surgery has a moderately high success rate, but mostly in younger dogs. Surgical intervention is a last resort but it does offer the advantage that, if successful, the condition is eliminated.

Conjunctivitis is an inflammation of the conjunctiva, the thin, pink membranes surrounding the eye, lining the globe, inner eyelids and third eyelid and forming a seal with the inner surface of the eyelids. The conjunctive membrane becomes swollen and reddened, often making it more visible. Conjunctivitis can affect one or both eyes in both dogs and cats.

In dogs, most forms of conjunctivitis are allergy based or are bacterial infections. In cats, viral (herpes), bacterial (Chlamydia) or mycoplasma (similar to bacteria) infections are more prevalent. Another cause is ‘dry eye,’ where tear production is decreased or absent. There are many other less common causes.

Long haired cats may be more susceptible to conjunctivitis as their eyes are more prominent and less protected by the eye socket, or there may be abnormalities in tear drainage causing an overflow of tears.

SEVERITY: Moderate. Can be easily treated in most cases.

  • Red eyes
  • Swollen conjunctivae
  • Mucky discharge
  • Rubbing or scratching at eyes
  • in dry eye, dogs show a thick, ropy discharge and the eyes look dry

The condition is quickly diagnosed by simple physical examination. The condition is not life threatening and can be treated. However, it does cause considerable discomfort to the animal and, left too long, the infection can persist and worsen. If your pet’s eyes are swollen and you can see a discharge you should make an appointment for an examination at the hospital. The sooner conjunctivitis is diagnosed the easier it is to treat and clear up.

In most cases, antibiotic drops or ointments applied directly into the eye are the usual treatments. For allergic conjunctivitis, anti-inflammatory drops are used, but occasionally oral antibiotics/anti-inflammatories are also required. Some conditions require a more intense action, such as swabs of the cornea to diagnose the cause.

An important part of treatment is cleaning of the eyes. Often thick discharges become stuck to your pet’s fur and are difficult to remove and uncomfortable to the patient. This can be done at home. The best way to bathe the eyes is as follows:

1. Wet a wad of cotton wool in warm saline water (warm enough that you can still touch it).

2. Squeeze out some of the water and place over the animal’s closed lids. Most animals don’t object.

3. Hold the pad there for 10 seconds to dissolve and loosen the discharge.

4. Wipe away the discharge, in one movement, from the inside corner of the eye towards the outside. Repeat as necessary until all discharge is cleaned away.

Bathing the eye in this way soothes your pet’s irritated eyes, removes discharge and the remnants of the last application of ointment, improves the blood supply to the area and aids healing.

Most ointments need to be administered at least twice a day. Drops need to be given more frequently and so are less practical. Unfortunately, some treatments cause transient discomfort after administration, meaning two people are required, one to hold the animal and the other to apply the treatment.

Liquid preparations can be applied directly onto the surface of the eye, 1 or 2 drops per eye is usually sufficient. Ointment is applied across the surface of the eye, about a centimetre per eye. The eyelids are then closed to spread the ointment over the entire surface of the eye. Some people find drops easier to administer. If you have any doubts as to how to give the medication prescribed then don’t be afraid to ask us for a demonstration.

Allergic conjunctivitis usually resolves quickly but, like all allergies, may return at certain times of year or on exposure to the allergen, especially if the allergen is pollens in the air.

Infectious conjunctivitis has a good prognosis, however dogs and cats often transfer the infection from one eye to the other, or to other dog or cat members of the household.

Dry eye requires lifelong treatment, but the eyes can be made much more comfortable.

Anybody who follows sports will know that athletes, particularly football players, will sometimes injure their ACL, the anterior cruciate ligament, putting them out of action for weeks or months.

Your dog can suffer a similar injury — Cranial Cruciate Ligament rupture, or CCL. It’s a painful, acute or chronic degenerative injury, usually of the anterior cruciate ligament, that results in partial or complete instability of the knee joint.

In the dog’s knee, or stifle, there are two cruciate ligaments — the anterior and the posterior. As they pass between the two main bones of the leg, the femur and tibia, they cross, hence the ‘anterior’ and ‘posterior names. If the knee is subjected to twisting when under load the anterior cruciate ligament can tear, destabilising the knee joint. The tear may be partial or complete.

As with athletes, the cause can be trauma related but that is not actually a common cause in dogs. More often, cruciate ligament rupture is a gradual process resulting from chronic inflammation in the knee joint. Obesity, age-related changes, poor conformation and immune-mediated diseases — where the body’s defence mechanism starts attacking the body instead of protecting it — are some of the more common causes of CCL in dogs. Whatever the cause, CCL is always painful and debilitating for the animal, and often distressing for the owner.

CCL ruptures are the most common cause of hind limb lameness in dogs. Dogs of any breed, sex or age can suffer a CCL injury but it tends to be more common in large and giant breeds, and in obese dogs. If the damage has occurred due to conformation problems of the joint, there is a strong chance of the other knee becoming injured at some stage.

It’s worth noting that overweight and obese dogs are much more prone to cruciate ligament tears. The best way to prevent a CLR in your dog is to ensure their diet is matched to their size and level of exercise.

SEVERITY: Acute to Chronic. Causes severe pain if not treated or managed.

The type of cruciate ligament injury that shows as an inability to bear weight on a leg, with the animal holding the affected limb off the ground, is generally caused by athletic or traumatic events. Signs of degenerative types of cruciate ligament injuries are much more subtle, showing as marked intermittent lameness that continues for weeks or months. Bilateral cruciate disease, where both knee joints are affected, is also common.

In some cases degenerative changes in the ligaments may predispose a dog to injury, resulting in partial ligament tears that, over time, can progress to full tears, so there is a chance that the opposite knee joint may have a similar type injury in the future.

An initial physical examination can often reveal a suspected cruciate ligament rupture. With the stabilising action of the anterior cruciate ligament ‘out of action’, the femur and tibia will move in an abnormal fashion in relation to each other, demonstrating instability. This kind of physical exam can sometimes be done when the dog is conscious but, besides being painful, the muscles of the dog’s joint will probably be in spasm, making diagnosis difficult. Usually the dog requires sedation or general anaesthesia to allow proper examination of the joint.

Tentative diagnosis of CCL is based on gait, lameness and palpation of the affected joint. There can be increased fluid in the joint, loss of definition around the ligaments or bony changes on the inside surface of the joint and pain on full extension. Movement of the joint is often increased due to a reduction in stability because of the loss of the CCL, so two different techniques can be used to ascertain joint strength — cranial tibial thrust and cranial drawer. Many dogs with CCL damage have no palpable joint instability due to muscle mass of the hind limb and swelling within the joint, particularly in acute injury. In these cases, radiographs (x-rays) are taken to determine the degree of joint effusion.

While an x-ray will not detect the damaged ligaments in your pet when they occur, they will pick up arthritic changes as they later develop in the unstable joint.

We may also use ultrasound to help confirm a diagnosis, or, in some cases, insert an arthroscope into the joint, allowing us to see if the cruciate ligament is torn or the meniscal cartilage is damaged, and the full extent of the damage.

A CCL can be treated either by medical management or by surgery.

Medical management is most suited for smaller, lighter dogs but larger breeds can show improvement as well. A conservative medical management program essentially lets nature take its course, with a little help from us. It uses a planned regime of rest, restriction of strenuous exercise, planned diet and weight loss programs, hydrotherapy, anti-inflammatory medications, and time.

At Alpine Animal Doctors, where examination does not fully confirm CCL damage, we opt for a ‘wait and see’ approach. These patients are routinely placed on a four week course of Pentosan Polysulphate (either via injection or capsules), joint supplements (Glyde or Sasha’s Blend) and sometimes pain relief, depending on the dog’s nature. The owner must then keep their pet very quiet with no exercise for the next 4-8 weeks to ensure the joint is given every chance to resolve the possible damage, rarely an easy thing to achieve.

Most dogs will show some degree of improvement with this regime, but few will return to full knee function.

The difficulty with this type of conservative management is that it is not fixing the underlying problem and, if instability of the knee remains, it will normally lead to arthritic changes in the joint, which cannot be reversed, leading to ongoing pain for the patient.

Surgery to repair the ligament and stabilise the knee joint is generally the only way in which CCL ruptures can be permanently improved. There are a number of surgical techniques available, all of which attempt to improve joint stability and reduce the progression of osteoarthritis.

Intra-capsular techniques insert devices used to stabilise the knee and require opening the joint. Extra-capsular techniques are designed to add strength to the structures surrounding the joint to compensate for the torn cruciate.

New procedures are continually being developed. The most recent, developed by Dr. James Cook of the University of Missouri in 2010, is the Arthrex Tightrope CCL repair system, which uses a very strong synthetic ligament anchored directly to the bones on either side of the joint.

In all cases there is no absolute guarantee that surgery can ensure the affected joint will function normally. Arthritis can still develop in the affected joint following surgery, but usually to a significantly lesser extent than if no surgery were performed. All of the surgical techniques are covered in the next section — Surgical options.

Surgical techniques that alter the function of the stifle joint are the most widely used. These have been developed in an attempt to reduce complications and poor outcomes of ligament replacement techniques, particularly in very large dogs. They are designed to change the anatomy of the stifle joint so that the tibia does not slide forward when weight bearing. There are four ways this can be achieved.

  • A Tibial Wedge Osteotomy (TWO) procedure reduces the tibial plateau angle by cutting a wedge of bone from the shaft of the tibia and securing the osteotomy with a bone plate and screws.
  • In a Tibial Plateau Levelling Osteotomy (TPLO) the angle of the tibial plateau is reduced by making a semicircular cut below the stifle joint. The cut bone is then rotated to a new position to reduce the tibial plateau angle, and secured with a bone plate. The procedure requires a purpose built semicircular saw blade.
  • A Tibial Tuberosity Advancement (TTA) is a procedure involving a vertical cut down the front of the tibia. The cut piece of bone is moved forward a measured distance to neutralise the forces that cause the femur to slide down the tibial plateau. The Tibial Tuberosity is secured with a purpose designed plate, comb and a spacer basket. The procedure requires specialised instruments for plate application and ready access to a complete set of plates, combs and spacer baskets.
  • The Triple Tibial Osteotomy (TTO) is a procedure that combines the TWO, the TPLO and the TTA. A vertical cut down the front of the tibia enables the Tibial Tuberosity to be advanced. A small wedge of bone is removed from the tibia behind the first cut, allowing the tibial plateau slope to be reduced. The wedge osteotomy is secured with a bone plate and screws.
  • The Tightrope Cruciate Ligament Repair system is a relatively new and minimally invasive method for extra-capsular stabilisation of the CCL. This technique does not require cutting of the bone. Instead, small holes are drilled in the femur and tibia to allow a synthetic biomaterial that closely mimics the properties of a natural ligament to be passed through a small incision to provide bone-to-bone stabilisation during healing.

The Kevlar-like synthetic material used is stronger and less prone to failure than any other suture materials being used for CCL reconstructions and is used extensively in human joint surgery, including the repair of ACL sports injuries.

The method used to repair your pet’s CCL injury will often come down to the preferences, experience and the level of skill of your chosen veterinary surgeon. At Alpine Animal Doctors we use the technique we think most suited to a specific patient.

More recently, however, we have begun to lean toward using the Tightrope system wherever we can. As effective as they are, the other techniques described above do have a downside; because the bone has been cut there is increased risk of infection or repair breakdown, and the results of such complications can be serious. They are also significantly more expensive.

The Tightrope procedure allows treatment of larger dogs who would normally require an osteotomy, but with reduced cost and risk of side effects. Our experience with this system has been that the procedure is easier and faster, with a reduced risk of both infection and/or catastrophic failure. The Tightrope system is also more easily reversed in the event of complications.

It can also be used in dogs of all sizes and both recovery rates and long term benefits appear to be comparable with other, more invasive, procedures. Importantly, because it takes less time to complete, it is also cheaper than other forms of CCL surgery.

The use of the Arthrex Tightrope Cruciate Ligament Repair System means we can reduce the operative and post-operative stress on our patients, and reduce the cost to pet owners. Savings will vary but the Tightrope procedure will generally cost between $800 to $1,000 less than other surgical methods.

How successful is CCL surgery?

There has always been some debate about the long term effectiveness of surgery for the repair of cruciate ligament ruptures. One study has found that the number of dogs who return to full normal leg function after surgery is little more than those who recover with conservative medical management.

We use both conservative management treatments and surgery, depending on the patient and the owner’s wishes.

With a substantial number of surgical procedures under our belt our experience over the years has been that, in almost all cases, our surgical patients experience a better long term outcome than those treated without surgery. Significantly, dogs who have undergone surgery for CCL appear to be far less likely to go on to develop arthritis in the affected knee, and consequently suffer less long term pain.

We will discuss with you the various treatment options available, and fully explain the pros and cons of each but, in the end, the decision on treatment options is always left to the pet owner.

Cushing’s disease, or hyperadrenocorticism, is most often seen in dogs but is also found in horses and cats. Cushing’s is a very common condition in dogs, particularly middle aged and senior pets. It is identified most often in dogs around the ages of 6 to 16 years old, although it can occur in animals as young as two. It affects all breeds and is equally distributed across male and female dogs.

Cushing’s Disease is a clinically complex chronic condition (see ‘Cushing’s Explained’) that cannot be cured but appropriate treatment can control the symptoms and give your dog a longer, more comfortable life.

There are two forms of Cushing’s Disease — pituitary dependent hyperadrenocorticism (PDH) and adrenal-based hyperadrenocorticism. Both result from the overproduction of too much glucocorticoid (cortisol/cortisone) in the body. In a normal dog the pituitary gland — a vital organ located at the base of the brain — produces a hormone called ACTH, which stimulates the adrenal gland — located adjacent to the kidneys — to produce the glucocorticoid hormones necessary for the healthy function of many important bodily systems. If something goes awry in either the pituitary or adrenal gland, resulting in too much glucocorticoid being produced, then Cushing’s disease develops.

Pituitary dependent hyperadrenocorticism involves the oversecretion of ACTH by the pituitary gland, in most cases because of a pituitary tumour. The PDH form of the disease is the most common, accounting for around 80% of all cases of canine Cushing’s disease.

Adrenal-based hyperadrenocorticism is responsible for around 15 to 20% of cases, and is usually the result of an adrenal tumour causing an oversecretion of glucocorticoids.

A third form of the disease, known as ‘iatrogenic’ Cushing’s disease, can occur if a dog has been given persistent high doses of cortisone. This is much less common and symptoms will usually disappear if the steroids are discontinued. The main reason a veterinarian will treat a dog with cortisone is to alleviate severe pain, often arthritic pain in older dogs. Paradoxically, discontinuing the cortisone treatments once Cushing’s is diagnosed will reduce or eliminate the symptoms of iatrogenic Cushing’s disease but leave the patient in constant arthritic pain if an effective alternative pain relief cannot be found. Avoiding this consequence is the reason vets are reluctant to use high doses of steroids and dogs are instead given tapering doses or every-second-day doses of cortisone treatments.

SEVERITY: Chronic/Progressive. Incurable but can be successfully treated in most cases.

There are a number of clinical signs that may lead us to suspect hyperadrenocorticism, and dogs suffering from the disease may show some or all of them. Unfortunately, because Cushing’s disease is a progressive chronic disease, the onset and development of symptoms is often so gradual that many dog owners mistake them for the normal signs of ageing. This is a natural mistake to make but, given how common the disease is in dogs it underlines the importance of ensuring that your dog has a regular annual veterinary check. One study showed that most dogs had been showing at least one symptom for between one and six years before the disease was eventually diagnosed.

Many of the symptoms are also not unique to Cushing’s and may indicate other health issues, making clinical diagnosis more complex. The main signs of the onset of hyperadrenocorticism are:

  • Increased or excessive water consumption (polydipsia)
  • Increased/excessive urination (polyuria), a by-product of increased water consumption
  • Urinary accidents in previously housetrained dogs
  • Increased/excessive appetite (polyphagia)
  • Sagging, bloated, pot-bellied appearance
  • Apparent weight gain, due to fat redistribution
  • Loss of muscle mass, giving the appearance of weight loss
  • Bony, skull-like appearance of head
  • Exercise intolerance, lethargy, general or hind-leg weakness
  • Excess panting, seeking cool surfaces to rest on
  • Symmetrically thinning hair or baldness (alopecia) on torso
  • Other coat changes like dullness, dryness
  • Slow regrowth of hair after clipping
  • Thin, wrinkled, fragile, and/or darkly pigmented skin
  • Easily damaged/bruised skin that heals slowly
  • Hard, calcified lumps in the skin (calcinosis cutis)
  • Susceptibility to infections (especially skin and urinary)
  • Diabetes, pancreatitis and seizures.

The most common reasons owners first bring their dog to the hospital for evaluation are increased water consumption — which affects more than 85% of animals with the disease and can be up to ten times their previous intake — the increased urination that follows higher water consumption, and thinning hair or hair loss.

Increased appetite is seen in around 80% of affected animals. Dogs may beg continually, begin to steal food, and become protective of their food. Often, the owner will assume that the dog is fine because he has a ‘good appetite.’ A pot-bellied appearance is also common, as is hair loss and thin skin, which usually starts around the elbows and spreads to the flanks and abdomen until eventually hair remains only on the head and extremities.

Initially, general blood and urine tests will give us a presumptive diagnosis. However, because most dogs only exhibit some of the signs of the disease, and many signs can also be caused by other metabolic diseases, further confirmatory tests are necessary. These will include a complete blood count (CBC), blood chemistry panel, and urinalysis.

Radiographs (x-rays) of the abdomen and chest may help us further define the cause. Sometimes the tumour has spread from the original site to the lungs; chest x-rays allow us to check for this. As most dogs with Cushing’s are relatively old we also check for other problems at the same time.

An abdominal ultrasound can also be helpful. It is a useful test to evaluate all of the abdominal organs. We can also use it to study the size and shape of the adrenal glands. In pituitary dependent hyperadrenocorticism the adrenal glands are usually normal in size, or enlarged. If a tumor is present however, one adrenal gland is often abnormally large or of uneven shape. Finally, if a tumor is suspected, ultrasound can help identify any metastasis to other organs such as the lungs.

There are a number of different treatment options available. Non-surgical medical management is the most common, and the treatment of choice at Alpine Animal Doctors. There are currently several different oral medications being used to treat canine Cushing’s disease and both pituitary dependent hyperadrenocorticism and adrenal-based hyperadrenocorticism tend to respond effectively to one or other of these treatments. Essentially, the oral medications used act in similar ways to chemotherapy treatments, destroying cells of the adrenal gland that produce the corticosteroid hormones.

Depending on the type of disease, particularly where an adrenal tumor is identified, surgical removal of the affected adrenal gland and the tumour causing the problem is also an option (pituitary tumors are not removed surgically).

In theory, surgical removal of an adrenal tumour will cure adrenal-based Cushing’s disease, and it is true that, once removed, these tumours tend not to recur on the remaining adrenal gland. However, an adrenalectomy is a high risk surgery and, given that most patients are elderly dogs, the risks are compounded for most Cushing’s patients. With the availability of modern oral drug treatments we see little reason to subject a senior dog to surgery when an acceptable outcome can usually be achieved with medication.

The prognosis for dogs treated for Cushing’s disease is generally very good. Symptoms such as excess drinking and urinating normally drop off quite quickly, although it can take several months for hair and skin to improve. Generally, however, patients are much more comfortable once the disease is under control, and usually live happily for a number of years.

However, it should be understood that Cushing’s disease can only be managed. We cannot cure the condition. The objective of treatment is to improve the quality of life and to increase life expectancy. We can usually achieve that but hyperadrenocorticism is a serious condition and maintaining a dog with Cushing’s disease requires vigilance and commitment on the part of the owner.

Cushing’s disease is a complex disorder involving the relationship between a number of different organs. In a normal, healthy dog the hypothalamus — an organ of the brain — ‘directs’ the pituitary gland, located at the base of the brain, to produce adrenocorticotrophic hormones (ACTH). This hormone is released into the bloodstream and stimulates the body’s two adrenal glands, which are located near the kidneys, to secrete another hormone — glucocorticoid (cortisone-like or cortisol)  — into the bloodstream.

Cortisol is necessary for life and affects a wide range of bodily functions in the dog, including blood sugar levels, fat metabolism, skeletal muscles, kidney function, nervous system, cardiovascular system, and immune response. ACTH/cortisol secretion is increased when an animal becomes stressed, from cold, heat, infection, pain, surgery, trauma etc. If blood cortisol levels rise high enough, the pituitary ceases to secrete ACTH. When blood cortisol levels fall, the pituitary secretes more ACTH. The response of the adrenal glands is to secrete glucocorticoid hormones in response to the pituitary, just as the pituitary responds by secreting ACTH in response to the adrenals. The end result is that, in a healthy dog, a balance is achieved.

In Cushing’s disease something goes wrong with this complex feedback loop. The result is a chronic excess of blood cortisol, caused by either a pituitary tumor or an adrenal tumour. Put simply, too much cortisol is poisoning the dog, who can no longer rely on its own feedback mechanism to regulate blood cortisol levels.

In the most common cause of Cushing’s, pituitary dependent hyperadrenocorticism, a tiny (usually less than 3mm in diameter), benign pituitary tumour (microadenoma) secretes an oversupply of ACTH, and ignores the response of the adrenals. The dog’s pituitary continues to pump out ACTH, causing the adrenals to keep producing cortisol, but the pituitary gland does not respond to the elevated blood cortisol levels by stopping its release of ACTH. Dogs with pituitary dependent hyperadrenocorticism are usually found to have two very large adrenal glands, both constantly working to try to keep up production of excess cortisol.

Larger pituitary tumors or macroadenomas (over 1 cm in diameter) are less common but where they do occur they can cause blindness,  seizures,or other neurological problems not directly related to hyperadrenocorticism because of the pressure their size places on brain tissue and nerves.

The less frequently diagnosed adrenal-based hyperadrenocorticism is also caused by a tumour, but located in the adrenal gland and responsible for secreting too much cortisol. The same type of faulty feedback mechanism sees the interaction between pituitary and adrenal messages get ‘lost’, and the tumour continues to secrete too much cortisol regardless of what the brain is telling it. About half of all adrenal tumours are benign (adenomas). The rest are malignant adenocarcinomas and can spread to the lungs and liver.

The extent of poor oral health in our pets has reached epidemic proportions. By the time they reach the age of three, four out of every five dogs will show some signs of dental disease. We don’t really know how bad the problem is in cats but it’s certainly even more extensive than in dogs.

The good news is that, first, most dental health issues are preventable. You can keep your pet out of the hands of the doggy dentist with some simple home care, and regular annual veterinary dental checks. If your pet already has some degree of oral health problems, caught early enough, they are most likely reversible.

Dental health problems in pets begin with plaque, which is formed by food particles and bacteria collecting along the gumline. Plaque begins to mineralise 3-5 days after it forms. If it is not removed by regular brushing and cleaning, minerals in the animal’s saliva combine with the plaque to form tartar (or calculus) which sticks strongly to the teeth. Tartar irritates the gums and causes gingivitis, an inflammation that can be seen as a reddening of the gums adjacent to the teeth. Gingivitis is what causes your pet to have bad breath. Left untreated, gingivitis will progress to periodontal disease, which is irreversible.

If tartar is not removed it builds up under the gums, separating the gums from the teeth to form ‘pockets.’ These pockets provide an ideal environment for further bacterial growth and more damage to the teeth and gums. Once infection gets into the tooth roots, where it can damage ligaments and bones, your pet has full blown, irreversible periodontal disease (periodontitis) and faces long term health problems. Periodontitis can lead to loose teeth, abscesses, and bone loss or infection and, if bacteria enter the bloodstream, to serious, life threatening heart and kidney disease. Periodontal disease is also always extremely painful for your pet.

Pet carers can prevent the development of serious periodontal disease with simple, regular oral health care at home (see Home Care tab):

  • Check your pet’s teeth regularly, including the back teeth.
  • Ensure your dog’s diet is suitable, and include raw bones.
  • Brush and clean your pet’s teeth at least twice a week.
  • Have their teeth scaled and polished once a year at the hospital’s dental surgery.

SEVERITY: Routine to Acute. Can be serious but is almost always treatable.

The first signs of tooth and gum disease are usually:

  • Yellow or brown stains on the teeth, indicating a build up of tartar
  • Persistent bad breath
  • Drooling and dribbling
  • Redness of the gums, particularly at the point where the gum meets the tooth, indicates gingivitis

The signs of more serious levels of gum disease include:

  • Signs of pus (purulent exudate) around the tooth
  • Gums that bleed easily
  • Sensitivity around the mouth
  • Pawing at the mouth
  • Inflamed or receding gums
  • Loose or missing teeth
  • Loss of appetite
  • Stomach or intestinal upsets
  • Difficulty chewing or eating
  • Irritability or depression

We use a number of criteria to assess the oral health of a dog, including the level and distribution of plaque and tartar, the health of the gums (gingiva), radiologic (x-ray) appearance, and the depth of the gingival sulcus (the ‘pocket’ formed between the tooth and the gum). After assessing these parameters, we can establish the extent and severity of disease and make a prognosis.

Tooth and gum disease are graded according to severity:

Grade I: An animal showing a mild degree of plaque and only minor redness of the gums with no radiologic change is classed as having Early Gingivitis. The prognosis is good, with appropriate treatment able to fully reverse the disease with no lasting effects.

Grade II: Advanced Gingivitis. At this stage, animals will show subgingival (below the gum line) plaque, with oedema and redness of the gums as well as as some radiologic changes. With treatment, the prognosis for full reversal of symptoms remains good.

Grade III: At this stage, the animal will be showing signs of Early Peridontitis. Subgingival calculus will be present, the gums will show redness and edema, some degree of recession (receding gumline) or hyperplasia and will bleed when gently probed. Radiology will show a 10 to 30% loss of bone support. Changes are irreversible and the prognosis is for palliative treatment.

Grade IV: Established Periodontitis will show larger amounts of subgingival calculus, severe inflammation and recession of the gums, pus, loose and/or missing teeth, deep pockets and gums that bleed easily. X-rays will reveal bone losses of greater than 30%. Prognosis is for palliative treatment.

Treatment depends upon the severity (grade) of the disease but in every case your pet will undergo a pre-surgical examination. This may include some pre-anesthetic blood testing to assess their health. In most cases we will also prescribe antibiotics several days before any scheduled dental procedures. Because animals are somewhat reluctant to ‘open wide’ and allow us to poke around in their mouths, your dog must be anaesthetised and monitored during any dental procedure.

Treatment for Grade I or Grade II involves a routine professional prophylaxis (dental cleaning and polishing). Plaque and tartar will be removed from the teeth, above and below the gum line, with handheld and ultrasonic scalers. Each tooth and the entire oral cavity is checked for disease. We will then polish the teeth to remove any microscopic scratches that would otherwise assist plaque formation and tartar build-up.

Following Grade I and II dental procedures we will suggest a regular home care program to control plaque. An individual patient plan might include the removal of plaque through brushing with specially formulated toothpastes, gels and rinses, and a nutrition plan.

Grade III and Grade IV periodontal disease requires more extensive treatment that may include root planing and subgingival curettage, periodontal debridement, gingivectomy, periodontal surgery, special therapeutics, and tooth extraction.

Root planing is a complex and difficult procedure that involves removing residual calculus and diseased cementum or dentin, and smoothing the root surface. Subgingival curettage removes diseased epithelium and connective tissue. This is also a difficult procedure.

An alternative to root planing and gingival curettage is periodontal debridement. In this procedure we use special ultrasonic scalers to remove irritants from the tooth and root surface such as bacteria and endotoxins produced by the bacteria.

In a gingivectomy hyperplastic or excess gingiva is removed. The area between this excess tissue and the tooth is a perfect habitat for bacteria. Periodontal surgery involve opening a flap of the gingiva over the root of the tooth to be able to reach the deeper structures. In some cases, a tooth cannot be saved and extraction is the only alternative.

Home aftercare for pets with Grade III or IV periodontal disease is critical. Pain relief and anti-inflammatory medication will need to be administered post-operatively and for several weeks following the procedures. Antibiotic therapy, usually for one or two weeks after surgery, will also be prescribed. You may also need to apply products containing zinc ascorbate, stannous fluoride, chlorhexidine or plaque preventives to the teeth on a regular basis.

Pets with periodontal disease will need frequent check-ups to assess their continuing oral health. Routine examinations at least twice a year will be needed.

There are a number of things you can do at home to help prevent your dog or cat from ever developing serious oral health issues.

Brush your pet’s teeth regularly, two or three times a week. Yes, it can be difficult if your cat or dog is uncooperative, but they can be trained and regular brushing is the most effective way to reduce plaque and prevent the build-up of tartar.

You can gradually condition your pet to accept brushing by just using your finger, with a little bit of liver paste or something on the tip, to gently rub the outside of the teeth. Gradually replace the paste on your finger with a bit of pet toothpaste and slowly work up to using a brush. We also have finger brushes available, like a thimble with bristles. Many pet carers find these easier to control. It may take a few weeks but eventually your pet will come to accept having their teeth brushed as just part of the routine.

You’ll need a product designed for animals; human toothpaste should not be used because of their foaming action and high levels of fluoride. The taste is also unpalatable to most pets. There are a number of pet pastes and gels available, often flavoured to be acceptable. Only the outside of each tooth needs to be brushed. That rough tongue does a good job of cleaning the inside of the teeth.

Diet is a crucial part of dental care. Some researchers and vets believe that the reduced amount of bones pets get in these days of convenient packaged pet foods may account for the dramatically increased incidence of tooth and gum disease we are seeing in dogs and cats today. Whether that’s true or not, there is no doubt that feeding your dog or cat raw bones a couple of times a week is very beneficial for the health of their teeth and gums. Chewing through bone and cartilage, and tearing the meat off the bone, exercises and cleans the entire tooth, right up to the gum line. Bones must be raw, not cooked.

Bones need to be part of a balanced diet that may include special dental diets for dogs at risk of tooth and gum disease. Contrary to popular belief, feeding your dog exclusively on dry food (or kibble) is no better for their teeth than any good commercial packaged food, wet or dry. The exceptions are the ‘dental’ diet dry foods. The kibble used in these is designed so that your pet has to chew each piece. The structure of the kibble remains intact longer than other dry foods, providing a degree of ‘scrubbing’ against the teeth that helps reduce plaque buildup. Dental foods are also often mildly medicated with chemical plaque cleansers.

The regular use of good quality artificial chews such as ‘Greenies’, which are designed to stimulate natural cleaning of teeth and gums, can also be helpful.

The single most important thing a dog or cat parent can do to keep their pet’s teeth and gums healthy is to schedule an annual dental checkup, and a scale and polish every year. Periodontal disease is painful, irreversible and can be very expensive to treat. There is no need for you or your dog to have to go through it if you get your pet on a good oral health care program now. We are happy to provide free dental checks and to advise you on the best home care plan for your particular pet.

Diabetes mellitus is a serious disorder caused by an insulin deficiency. Insulin is a hormone produced by the pancreas and secreted into the blood after eating. Its roles are varied: to promote the storage of glucose, as ready to use glycogen and for later use as fat, and to stop glucose formation from stored body fats; and to promote the uptake of amino acids and the manufacture of these into proteins.

There are two types of diabetes mellitus. Type I, also called insulin dependent diabetes, is caused by very low or absent insulin secretion. Patients with Type I diabetes will die if not treated with insulin. Type II, or non insulin dependent diabetes, is caused by inadequate or delayed insulin secretion relative to the needs of the patient. Type II diabetes is by far the more common and most patients with this non-insulin dependent form of the disease will also require insulin. In a few cases it may be possible to control the condition by diet alone but this is not the norm.

Diabetes mellitus can be caused by infection or inflammation of the pancreas, by genetic causes, immune system attack of the pancreas, as a by-product of other diseases, and, rarely, as a side effect of some drugs. Obesity vastly increases the risk of your pet contracting diabetes.

In Australia, vets are seeing more and more cases of Type II diabetes, mirroring the experience of our colleagues in human medicine, and probably for much the same reasons — just like us, our pets are becoming fatter and more sedentary than ever before.

SEVERITY: Chronic to potentially life threatening in some cases but generally can be managed.

Diabetes mellitus is usually first seen in middle age (4-14 years). Diabetic  pets usually eat a lot but lose weight, drink a lot and urinate a lot. Cataracts develop quite rapidly in dogs with poorly controlled diabetes, leading to blindness.

Left untreated, as the disease progresses pets develop ketoacidosis and become very sick, developing anorexia, vomiting, weakness and lethargy. At this stage the diabetes has become a serious life threatening illness.

Diagnosis of diabetes mellitus involves measuring glucose levels in urine and blood. A full urinalysis and blood screen is performed to diagnose the diabetes, assess the severity of the disease, look for underlying causes of diabetes and to examine for secondary problems associated with the diabetes, for example, urinary tract infections.

Treatment of pets with uncomplicated diabetes involves dietary changes, the use of injected insulin, and regular monitoring of blood glucose levels in the hospital. Insulin therapy is lifelong for most patients, and can involve once or twice daily injections (administered by the owner — we teach you how to do this).

Soft, moist foods are avoided. Pets that are not overweight are fed a diet that has a constant calorie content that they will reliably eat. Overweight pets are fed on special weight reducing diets. The ideal diet is high in fibre as this helps stabilise blood glucose levels, and will be low in fat and high in complex carbohydrates. Set feeding times and amounts are determined for your pet and are fixed in relation to insulin injection times. Exercise levels for your pet need to be kept consistent or will interfere with the stability of your pet’s blood glucose levels.

Initial stabilisation on insulin therapy requires day hospitalisation and the taking of blood glucose levels throughout the day. This may need to be repeated every few weeks until things are stabilised, then every few months or whenever a problem arises.

At home monitoring involves assessing levels of drinking and urinating, eating and body weight.

Hypoglycaemia, or low blood glucose, can occur if too much insulin is given or if your pet doesn’t eat normally or uses more glucose than usual, for example, through excessive exercise. The most obvious sign is weakness or lethargy, which will improve rapidly if a readily absorbed glucose source such as honey is given. Left untreated hypoglycaemia can result in coma or death.

Ketoacidotic diabetic patients — the really sick ones — need hospitalisation and intensive treatment with intravenous fluids and medication to get them past this life threatening stage. These patients are harder to initially stabilise on insulin, but once past this are treated in the same way as uncomplicated diabetic patients.

Entire females should be speyed when stable, as cycling makes management of their diabetes difficult, and pregnancy is strongly recommended against, it is difficult to maintain and can cause many problems for the mum.

Discoid Lupus Erythematous (DLE) is one of a number of autoimmune diseases seen in dogs, in this case affecting the skin. An auto-immune disease occurs when the body’s tissues are attacked by its own immune system. Normally the complex organisation that is the immune system will ‘seek out and destroy’ any invaders, including infectious agents. Patients with autoimmune diseases frequently have unusual antibodies circulating in their blood that instead target their own body tissues.

Discoid Lupus Erythematous is caused by an animals’s own immune system developing an immune response towards antibodies attacking the skin. Usually this occurs on the nose and/or the lips, resulting in lesions (wounds), but in can also be seen on the ears, feet, genitalia or around the eyes. Exposure to sunlight will often worsen these lesions, and may eventually lead to skin cancer.

SEVERITY: Moderate. Can lead to skin cancer if not treated/managed.

DLE results in hair loss, inflamed skin, loss of pigmentation (dark coloured skin lightens, eventually turning pink), scaling and crusting, ulceration, infection, tissue loss and scarring. DLE is most often seen in Collies, German Shepherds, Siberian Huskies and Shelties but it can occur in any breed. Female dogs seem to be more frequently affected than males.

Because most of the clinical signs of DLE can also be caused by other diseases, including other autoimmune diseases, infections and tumours, a definitive diagnosis of the condition can usually only be made after taking ‘punch’ biopsies — small circular tissue samples — of affected areas and assessing the pathology. Biopsies are normally obtained while your pet is under a general anaesthetic.

We will usually initially prescribe antibiotics. These are used for both their antimicrobial effect and anti-inflammatory affects. Vitamin B3 (nicotinamide/niacinamide) may also be a part of your pet’s treatment plan. We may also use immunosuppressive doses of oral corticosteroids.  These are usually very effective but side effects will often prohibit their long term use.

Appropriate treatment of DLE will achieve a remission of the disease in about three out of four cases. Following successful treatment, your pet needs to remain on maintenance doses of oral Vitamin E and will need to be kept out of the sun as much as possible. The use of an SPF15+ waterproof sunscreen on exposed skin is also helpful for patients predisposed to DLE.

Dry eye is the common name for Keratoconjunctivitis sicca (KCS), a disease affecting dogs, and less frequently, cats, where the eye(s) have either limited or no tear production. The cause is not known but infections and auto-immune diseases may play a role. Small breed older dogs are most at risk and some of the ‘flat-faced’ breeds are more susceptible because of their very large eyelid openings.

SEVERITY: Moderate.

Any of the following symptoms may indicate a case of dry eye.
• An eye that looks dry
• Recurrent eye problems
• Ropy yellow discharge from the eye
• Red conjunctivae

We will initially conduct a Schirmer Tear Test (STT), which involves placing a small piece of absorbent paper over the lower lid. The dogs tear film moves down the paper and is measured after 60 seconds. In this time a dog should produce 15 mm of wetting of the paper, and cats 10 mm. Any less than this and your pet has dry eye. A full eye and body examination are also performed to check for underlying causes.

There are two phases to treatment of dry eye. Initially we will prescribe ‘artificial tears’ to try to replace the tears and keep the eye moist. These are applied up to 6 times a day, and thicker preparations are sometimes also used at night.

The second phase of treatment is to encourage the eyes to start naturally producing tears again. Medications such as Cyclosporin or Optimmune will usually significantly stimulate natural tear production, often returning tear production to normal. Occasionally other drugs may be added to the treatment plan to assist with tear production. Any other underlying or associated diseases would also be addressed.

Medications are not always effective. In cases that do not respond medically there are a number of surgical options. For dogs with large eyelid openings a procedure called paria closure can help reduce evaporation of tears. Sometimes we can reposition a salivary duct from the mouth to produce a tear film.

If your pet has dry eye but still has some tear production, the prognosis is better than if there is no tear production at all. Your dog will need regular STTs to monitor his response to therapy and we may suggest adjustments to medication based on the results. Unfortunately, we can often only control the disease, not fully cure it so treatment may have to be ongoing.

Sore ears are common in both dogs and cats. The design of their ear canals allows debris, wax and moisture to accumulate within the ear. This is even more likely in breeds with long, floppy ears. Ears with lots of folds and ears with lots of hair growing in the ear canal are more likely to suffer problems.

A sore ear can be particularly distressing for your pet. And an otherwise easily alleviated problem can turn into something more serious with excessive scratching and head shaking. To determine what is the cause of your pet’s problem it is best to bring them in for an examination as quickly as possible.

There are many things that can cause ear problems, including injuries, bacterial infections, fungal (yeast) infections, grass seeds, dirt or sand, excessive wax production, soap or water in the ear canal, ear mites, allergies and other skin conditions.

SEVERITY: Moderate.

Your dog or cat may show any of the following symptoms…

  • Scratching one or both ears
  • Smelly ears
  • Head shaking
  • Head tilted to one side
  • A yellow, green, brown, black or cream discharge from the ear
  • Painful ears or head when touched
  • Redness and inflammation of ear flap and ear canal
  • Swelling of ear flap
  • Stumbling or circling to one side
  • Lethargy and depression.

Because most ear problems show the same signs the ear canal and ear drum have to be examined with an otoscope for us to make a diagnosis. A grass seed in the ear may cause your dog to have exactly the same signs as a bacterial infection so we need to be able to see into the canals to tell the difference. The ear drum must be assessed, as a ruptured ear drum is a much more serious illness which can cause balance problems and inner ear infections. The medication we choose may be altered if the ear drum is ruptured.

Sometimes a smear is taken and examined in our in-house laboratory to check the cause of the problem. Other times a sterile swab is taken to ‘culture’ the bacteria or yeast that are causing the infection, and thus determine the appropriate treatment.

Some dogs that have chronic recurring problems have bacteria or fungi that are resistant to the common ear ointments and drops, and need different formulations to treat the infection. This can only be determined with microbiological culture and sensitivity testing.

Medication, in the form of drops, ear cleaning solutions and tablets, is usually all that is needed to quickly clear up ear problems. It is essential that these treatments are used correctly. Intermittent use of antibiotic solutions can favour the multiplication of resistant bacteria, which is why we always prescribe the drops only for a certain time and recommend a revisit to check the ears at the end of the course. If the ears aren’t 100%, the problem will reoccur once the medication is stopped.

Sometimes more extensive treatment is necessary. An ear that is full of pus may need to be flushed under general anaesthesia to remove as much discharge as possible and assess the ear drum. Or a grass-seed must be removed under sedation or general anaesthesia to prevent rupture of the ear drum.

Home prevention:

There are some things you can do to help prevent ear problems:

Cut the hair around and inside the ear flap short to allow better circulation of air

Get your groomer to pluck the ears regularly to prevent hair falling down into ears.

Use an antibacterial ear cleaner once or twice a week. These protect against a broad range of infections and also dissolves the wax and debris inside the ear and allow the dog to shake it out. It also dries out the ear if your dog has been swimming or water has gone into the ear during a bath.

Entropion is a condition in dogs, and occasionally in cats, in which either the upper or lower eyelid is inverted. When an eyelid is inverted it turns inward toward the eye and eyelashes and hair will irritate the cornea (the clear, front part of the eye) causing the signs of entropion. These vary in duration and with the severity of the condition, but can range from excessive tear production to a mucoid discharge with pigmentation or ulceration of the cornea. In severe cases corneal perforation (rupture of the eye) may result. Secondary infection of the eye is also common.

SEVERITY: Moderate. Good prognosis.

There are two forms of entropion, primary (anatomical) and secondary (spastic).

Primary entropion is the result of a a structural abnormality (birth or genetic defect) of the eyelid/tarsal plate and is usually seen in dogs with short noses and extra folds of skin around the nose and face such as Chow and Shar Pei dogs, or in any breed with heavy facial skin, including the Bloodhound, Newfoundland, Mastiff, or Saint Bernard. Primary entropion is usually found in young dogs, less than a year old, and can be seen in pups as young as 2-6 weeks of age.

Secondary entropion is generally found to be the result of excessive blinking associated with an eye irritation — a foreign body in the eye, extra hairs on the inside of the eyelid, or conjunctivitis — and can be seen in dogs of any age.

Both primary and secondary Entropion are diagnosed by physical examination.

Primary entropion must be surgically corrected. Young dogs with the condition have sutures placed to hold the eyelid everted in the correct position, until the puppy is around 6 months old and of a more mature size. In some individuals these everting sutures may be the only treatment needed but in many cases the sutures will need to be repeated if entropion recurs when sutures are removed or they loosen. Using this method and waiting until the dog is more mature gives the best chance of success for the more permanent surgery that follows.

Second phase surgery is undertaken once the puppy reaches a more suitable age/size, and also for mature dogs who present with chronic entropion. During this procedure skin is removed to permanently evert the eyelid edge to the point where it sits normally on the eye. The amount of skin to be removed needs to be finely judged to prevent the eyelid being everted and not correctly resting on the cornea. Surgeons will err on the side of caution and remove less rather than more skin so the procedure may need to be performed twice to achieve the optimum outcome.

The procedure is fairly routine and the outcome, in the vast majority of cases, is good.

Treatment of secondary entropion is simpler, involving correction of the underlying problem, for example, by removing the foreign body causing the entrpion. In some cases, once the primary problem has been resolved, surgical correction will be needed to prevent a recurrence, particularly in breeds predisposed to primary entropion.

In both primary and secondary conditions the treatment is fairly straightforward and the long term prognosis is excellent.

Epilepsy is a disorder of the brain where, although the brain is normal in structure, it functions abnormally. Generally, the disease occurs in animals between 6 months and 5 years of age, causing seizures that vary in intensity and frequency depending upon the dog. The condition is controlled by medication.

Where epilepsy develops in dogs  under 2 years of age it is often the case that medication may not fully control the seizures. As the animal ages, the seizure activity usually increases and it may become difficult to maintain control over the disease.

SEVERITY: Moderate to Severe. Generally controllable with medication but can become life threatening. See Treatment Options tab.

An epiletic seizure will begin without warning. A single seizure may be brief or may consist of multiple events with no break (Status epilecticus. See Treatment Options tab).

The dog will fall on its side, become stiff, chomp its jaw, salivate profusely, urinate, defecate, vocalise, and/or paddle with all four limbs and may become unconscious. These seizure activities generally last between 30 and 90 seconds. Dogs can display abnormal behaviour after seizuring that may include periods of confusion and disorientation, aimless wandering or pacing, eating or drinking more. Recovery from a seizure may be immediate or may take up to 24 hours.

Epilepsy is often first diagnosed by eliminating the possibility of other contributing diseases, for example, brain tumours, or toxicity. Blood tests are required initially to exclude such causes and also to give a baseline for future referral after initiating medication.

The two determining factors in diagnosis are age at onset and the pattern of seizures, i.e., the type and frequency. As a rule, the younger the age at onset, the more severe the epilepsy and also the increased likelihood of the disease being resistant to medication.

There are a number of therapies available for epilepsy and we may need to trial a few before the best option for your pet is found. During the initial stages of medicating, several blood tests may be required to determine the correct dosage.

The need for treatment is based on the severity and frequency of seizures, and the level of owner concern. Isolated instances of mild seizures every 2-3 months are usually not thought to warrant anti-epileptic medications initially. However, seizures often gradually increase in frequency so medication may need to be started in the future.

Therapy is initiated if there is either more than one generalised seizure, acute ‘clusters’ of more than one seizure within a 7 day period over a period of a month, or in the case of acute status epilepticus. Status epilepticus is defined as a series of seizures with no intervening states of consciousness. This stage of epilepsy is considered a medical emergency as it can cause irreversible nerve damage and/or be fatal. Once therapy has begun it must be maintained for the life of the dog. Cessation of treatment can induce seizures.

Human anti-epileptic medications are not appropriate for use in animals.

Anti-epileptic medications will decrease the severity, frequency and length of seizure activity but perfect control is rarely achieved. The normal goal of treatment is to achieve a balanced state of less than one seizure over 6 to 8 weeks. Epilepsy can become resistant to medication despite trialling the appropriate therapies available.

There are very few indicators that will help warn you of impending seizures in animals but reducing stress and avoiding ‘hot’ foods may help in reducing the likelihood of a seizure. If a seizure does occur try to prevent your pet from injuring themselves on surrounding objects, and avoid placing fingers in their mouths as muscle spasms may cause severe bites.

Flea bite hypersensitivity, or FAD, is an excessive reaction to the bite of a flea. When a flea bites and dog or cat (or human) it injects a small amount of saliva into the skin. Normally this produces little more than a mild itching. Some cats and dogs can however develop an allergy to this saliva, and, in these animals, even a single flea bite can cause intense itching and irritation. A severe flea infestation, where lots of fleas or flea dirt is found, can cause the same signs in non-allergic patients.

FAD is most commonly seen in the summer months when fleas are more prevalent but can occur even in winter because even low numbers of fleas can cause a reaction. Pets suffering from flea bite hypersensitivity will continually bite, chew and lick their skin.

SEVERITY: Incurable but treatable.

Dogs typically attack their back, just above the tail, the back and insides of their hind legs, their belly and around the anus. This leads to scabs, hair loss, thickening and increased pigmentation of the skin and scaling and, eventually, to weepy skin and sores.

Cats are affected slightly differently. Some cats develop small scabs over their back or, in severe cases, over their whole body called ‘miliary dermatitis.’ Others lick until they develop bald patches, especially on their belly and the back of their hind legs.

Diagnosis is usually easily confirmed by a visual examination. Conditions other than flea bites may cause similar signs however, so we may recommend tests to confirm the diagnosis, especially when there is no visible evidence of fleas on the animal.

FAD is not life threatening, but nor is it curable. In the short term, hypersensitive pets who have been exposed to fleas and are suffering from FAD need medication to relieve the itching, stop them scratching and give their skin a chance to heal. Some will also need antibiotics for infections that have developed in the damaged skin.

In the long term, all pets suffering flea bite hypersensitivity need to have their exposure to fleas minimised — permanently.

There are several excellent, easy to use products available that effectively kill adult and juvenile fleas both on your pet and in the environment. Where large environmental contamination with flea eggs exists separate environmental treatments may be recommended.

Other pets in the household also need constant treatment. Although they may not be allergic and no be affected, they do carry fleas which then can bite the affected pets. Please talk with our nurses regarding a specific flea prevention treatment program for your pets.

Gastroenteritis is an inflammation of the stomach and intestinal tract, presenting as vomiting and/or diarrhoea. Potential causes are many and varied, with the main culprits being intestinal worms, bacterial or viral infections, or eating something that upsets the gut such as ‘garbage,’ cooked bones and excessively fatty meals.

While most episodes of gastroenteritis do not turn out to be serious, regular worming, vaccination and care with your pet’s diet will greatly reduce the chance of it reoccurring.

SEVERITY: Generally minor. Treatable. NB: Rare cases of the more severe Paroviral enteritis require ICU hospital treatment.

Clinical signs are usually obvious, presenting as abnormal episodes of vomiting and/or diarrhoea. Other than vomiting the dog will usually be perfectly happy and normal. Owners will also often be aware that their pet has eaten something they shouldn’t. You may also see signs of lethargy.

Diagnosis is usually straightforward at a visual examination/consult. Patients suffering gastroenteritis will often still be quite bright and happy. If they present with lethargy/depression as well as vomiting/diarrhoea, other more serious conditions may be present and blood tests and/or other diagnostic tests may be needed.

The exception is parvoviral enteritis.  Parvovirus causes a potentially very severe, and possibly fatal, form of enteritis in dogs, which requires the patient to be hospitalised under intensive care conditions, often for a week or more.

Treatment involves first ruling out worms as a cause. This is done by knowing the patient has recently been wormed with an effective dose of an effective drug, or by worming them at the time. Then we give the stomach and intestines a rest. The most common problem we see is that patients have been fed too much too soon after the signs started, which triggers another bout of vomiting or diarrhoea.

In most cases the best course of action is to feed nothing for 24 hours (allowing free access to water) and then introduce regular, small, bland meals. ‘Bland’ means food that is not rich enough to upset the gut and is easily digested. Suitable foods may be boiled chicken — no skin, no fat, no bones — and boiled white rice, or one of the commercially prepared prescription diets available from the hospital. Small meals ensure the volume of food will not over stretch the gut and cause further irritation.

Once the initial vomiting/diarrhoea has stopped meals are gradually increased in size and the frequency reduced, then normal food is added little by little over a period of a week. See the ‘Diet’ tab for an example of an effective recovery diet.

Many patients will get better if they revert to normal feeding earlier than this but some will return to vomiting/diarrhoea. You cannot know which ones will relapse until it happens so it’s best to treat all patients as if they will be one of the more sensitive ones. If at any time the vomiting or diarrhoea recurs it is very important to let us know so we can reassess the diagnosis and treatment.

An injection may be given to settle the nausea/vomiting and sometimes a course of antibiotics will be prescribed if bacterial infection is suspected. Antibiotics are often held in reserve as most patients improve rapidly without them.

This is an example of a daily recovery feeding regime pet carers can use and which we have found very effective. In our experience the most common cause of a recurrence of gastroenteritis is owners not following this regime precisely and either feeding too much or reverting to a normal meal size before the seven days are up.

Day 1.
Water only.

Day 2.
Food: Veterinary Diet, canned or dry.
Frequency: 4-6 times per day
Amount: one-eighth of normal meal size

Day 3.
Food: Veterinary Diet, canned or dry
Frequency: 3-4 times per day
Amount: one-quarter normal meal size

Day 4.
Food: three-quarters Veterinary Diet, plus one-quarter normal food (mixed together)
Frequency: 3-4 times per day
Amount: one-quarter normal meal size 

Day 5.
Half Veterinary Diet, plus half normal food (mixed together)
Frequency: 2-3 times per day
Amount: one-third normal meal size

Day 6.
Food: Veterinary Diet, plus three-quarters normal food (mixed together)
Frequency: twice a day
Amount: half normal meal size

Day 7.
Feed normally

The heart is a unique organ, different from any other in the dog’s body. It consists of 4 chambers, all of which contain blood and are responsible for sending blood that is low in oxygen to the lungs to receive more oxygen, then sending this oxygen-rich blood throughout the body to all the tissues in need. A series of valves in the heart separate these 4 chambers, preventing blood from flowing where it shouldn’t.

For dogs suffering heart disease it is generally the valves of the heart that become affected rather than the heart muscle. The four chambers in the heart are divided into right and left sides, each with its own set of valves. In some breeds of dogs — Poodles, Cocker Spaniels, Yorkshire Terriers — there is a predisposition, possibly genetic, towards developing problems with the heart valves although any breed can be affected.

The heart valve itself becomes blunted, or rounded, and fails to close properly, leading to ‘back-flow’ of blood, which results in an overload of blood in the chamber receiving the back-flow. Over time, this overload of blood can stretch the receiving chamber, causing it to enlarge. Chamber enlargement may put pressure on the main airway in the lungs (the trachea or windpipe), resulting in a dry, hacking cough. Stretching of the chamber may also disturb the heart’s rhythm, resulting in an arrhythmia.

Disease of the heart muscle is much less common than that of heart valve disease. Heart muscle disease can be inherited, and is often seen n Cocker Spaniels, Boxers, Great Danes, and Doberman pincers. Other breeds can also be affected.

For dogs affected by this form of heart disease, the heart muscle shrinks, or atrophies. This reduces the effective pump of each heart beat, meaning the  weakened heart muscles fail to pump blood adequately to the tissues of the body.

SEVERITY: Chronic. Treatable with appropriate medication

A cough is often the first sign of heart disease. Over time, the overload of blood in the chamber can stretch the receiving chamber, causing it to enlarge and resulting in dry, hacking cough. Stretching of the chamber may also disturb the heart’s rhythm, resulting in an arrhythmia.

Most commonly, the overflow of blood can also back up into the lungs, resulting in a ‘wet,’  productive sounding cough.

Patients can also become weak, stop eating, or eat less or much more slowly, and sleep a good portion of the day. Sometimes they will collapse, showing signs of stiffness in the legs and arching back their head. During these collapsing episodes, the patient may also lose control of their bladder, bowels, or both. Clients often refer to these episodes as ‘seizures,’ although technically they are not. When this occurs, the patient is considered to be in heart failure and both owner and vet will be faced with an emergency. Much better to bring in your dog for an examination if or when you see the earlier telltale signs. Diseased heart muscles can cause similar signs to those seen in heart valve disease — weakness, decreased appetite and cough.

Diagnosis is by physical examination, X-rays, and  auscultation, which, in larger animals, helps us to pinpoint the affected valve (PAM on the left and Tricuspid on the right side of the chest).

If the condition is non responsive to medication, or a more definitive diagnosis is required we will refer the patient to a veterinary cardiologist, who may use an electrocardiogram (ECG) or ultrasound of the heart. Specific tests are then used to determine what form of heart disease exists.

Appropriate treatment is aimed at reducing the backflow of blood, reducing the size of the heart if enlarged, strengthening the heartbeat itself, correcting abnormal heart rhythms, and clearing fluid back-up in the lungs.  We have a number of medications available to us that have been specifically designed to treat these conditions, either singly, or in combinations with other drugs.

In some cases of heart muscle disease nutrient supplements may be used. L-carnitine, and Taurine are two essential amino acids that are often found to be deficient in some cases of heart muscle disease in dogs, which has prompted the development of specialised cardiac diets designed to help dogs and cats with heart disease. These can be very helpful in controlling symptoms.

These days most canine heart disease can be successfully treated with long term medication but surgery is sometimes an option in some cases.

Heartworm is a large parasite — up to 30 cm long — that lives in the heart and the main blood vessels of the lungs of infected dogs. Transmitted from dog to dog by mosquitoes, the parasite begins its growth within the mosquito and is injected into the dog’s bloodstream as the mosquito bites.

It then develops into an adult worm within the dog, migrating to the heart and lungs and, occasionally, the liver. The adult heartworms then breed and can increase to large numbers, interfering with circulation and damaging tissues and leading to heart and/or liver failure and lung disease as the lungs develop a severe reaction to the worms. Left untreated this will result in the death of your pet.

While heartworm disease is more prevalent in areas where the mosquito population is high it is not confined to these areas. It takes only one infected pet moving into a new area to introduce heartworm into a dog population previously free of the parasite. Also, because many people take their dogs with them on outings and holidays, and even for walks in different parks, the level of heartworm disease is gradually increasing.

As it is transmitted by mosquitoes, dogs do not have to come in direct contact with other dogs to catch the disease.

SEVERITY: Highly transmissible and life threatening if left untreated.

In the early stages of infection no symptoms are evident, and dogs may remain symptom free for months or even years despite carrying the parasite. The first signs may include coughing, the animals may become progressively more lethargic with exercise, and/or experience reduced appetite and weight loss. In the later stages of the infection collapsing episodes occur, as well as coughing up blood.

NB: These are all similar signs to heart disease.

Heartworm disease is diagnosed by a number of methods. A simple blood test is performed in the hospital’s pathology lab and checked under the microscope for antigens and microfilaria in the blood. If the test result is negative prevention for heartworm disease can be commenced (see ‘Prevention’ tab). If the test is positive, a second blood test is taken to confirm the result.

If this second result is also positive, confirming our diagnosis, then we will perform a series of tests to determine how advanced the infection is and how much damage the heartworm have caused so far. These include further blood tests and radiographs (x-rays).

Treatment is drug-based and varies depending on the degree of heartworm disease present in the infected dog. The drugs we use to kill the heartworm are dangerous in themselves. Some are arsenic based so great care is required. Dogs with no clinical signs have a good prognosis. Those with advanced and/or severe heartworm disease have a guarded to poor prognosis.

Fortunately, heartworm is a disease that is easily avoided with routine modern preventative medications. Prevention should begin from 6-8 weeks of age. Puppies are started on a monthly chewable treat or spot-on treatment that contains the heartworm medication. They can then stay on this medication for life without any adverse effects. Alternatively, fully grown adults can be give an annual injection that will provide them with 12 month protection against heartworm.

If you have a dog that is older than 6 months, a blood test is required before heartworm prevention can be begun.

The parasitic hookworm gets its name from the hook-like mouthparts they use to attach themselves to the intestinal wall. Despite being very small — about 3 mm long — they can suck substantial amounts of blood from tiny vessels in the intestinal wall, and a large infestation can cause anaemia. Hookworm is most commonly seen in puppies but adult dogs can become infected.

Dogs can get hookworms by four routes: through the mothers placenta before birth; via the mother’s milk; by swallowing hookworm larvae in the milk, or; through the skin.

If a pregnant dog has hookworm, the pregnancy may reactivate the larvae. The larvae will then enter the female’s circulatory system and pass to the puppy through the placental blood flow. Infection through the mothers milk is another source, and considered an important route of infection for puppies. Finally infection may also be by the infected larvae penetrating the skin and then migrating to the bowel where the worms complete their life cycle.

SEVERITY: Moderate to Serious if left untreated.

The presence of pale gums, diarrhoea, or weakness might suggest the need to specifically determine the dog’s red blood cell count. Some dogs experience significant weight loss, bloody diarrhoea, or failure to grow properly with hookworm infection.

Skin irritation and itching can be one of the common signs of a heavily infested environment. The larvae burrow under the skin and cause the dog a great deal of itching and discomfort. Kennels can sometimes have a problem with hookworms.

Hookworms are easily detected by a microscopic examination of a small sample of the suspect animal’s faeces. In puppies large numbers of worms must usually be present before eggs are shed into the faeces, hence faecal examination may be less reliable in very young puppies than in adult dogs.

There are several very effective drugs that will kill hookworms. These are given by injection or orally and have few, if any, side-effects. However, these drugs only kill the adult hookworms. It is necessary to treat again in about 2-4 weeks to kill any newly formed adult worms that were larvae at the time of the first treatment.

In some dogs with severe anaemia caused by heavy, untreated infestations, a blood transfusion may be necessary.

The dog’s environment — kennel, bedding, runs etc. — can also be laden with hookworm eggs and larvae and will need to be chemically treated to avoid recurring infection. There are some available that are safe to use on grass.

Hookworm is fairly easy to prevent with modern broad spectrum worm treatments. All pups should be treated at 2-3 weeks of age and periodic deworming is advisable to prevent any cross-contamination to other household pets, particularly if they are walked frequently. Nursing females should be treated concurrently with their pups, as nursing can reactivate the infection in the mother.

Good hygiene is also important, particularly the prompt disposal of dog faeces.

Adult hookworms do not infect humans. Where bare skin has come into direct contact with moist, hookworm infested soil it is possible for the larvae to burrow into human skin and cause itching. The worms do not mature into adults. In rare cases, the canine hookworm will penetrate into deeper tissues and partially mature in the human intestines.

The presence of pale gums, diarrhoea, or weakness might suggest the need to specifically determine the dog’s red blood cell count. Some dogs experience significant weight loss, bloody diarrhoea, or failure to grow properly with hookworm infection.

Skin irritation and itching can be one of the common signs of a heavily infested environment. The larvae burrow under the skin and cause the dog a great deal of itching and discomfort. Kennels can sometimes have a problem with hookworms.

Hookworms are easily detected by a microscopic examination of a small sample of the suspect animal’s faeces. In puppies large numbers of worms must usually be present before eggs are shed into the faeces, hence faecal examination may be less reliable in very young puppies than in adult dogs.

There are several very effective drugs that will kill hookworms. These are given by injection or orally and have few, if any, side-effects. However, these drugs only kill the adult hookworms. It is necessary to treat again in about 2-4 weeks to kill any newly formed adult worms that were larvae at the time of the first treatment.

In some dogs with severe anaemia caused by heavy, untreated infestations, a blood transfusion may be necessary.

The dog’s environment — kennel, bedding, runs etc. — can also be laden with hookworm eggs and larvae and will need to be chemically treated to avoid recurring infection. There are some available that are safe to use on grass.

Hookworm is fairly easy to prevent with modern broad spectrum worm treatments. All pups should be treated at 2-3 weeks of age and periodic deworming is advisable to prevent any cross-contamination to other household pets, particularly if they are walked frequently. Nursing females should be treated concurrently with their pups, as nursing can reactivate the infection in the mother.

Good hygiene is also important, particularly the prompt disposal of dog faeces.

Adult hookworms do not infect humans. Where bare skin has come into direct contact with moist, hookworm infested soil it is possible for the larvae to burrow into human skin and cause itching. The worms do not mature into adults. In rare cases, the canine hookworm will penetrate into deeper tissues and partially mature in the human intestines.

‘Hotspots’ is a common term for a painful, itchy, moist skin rash, a form of dermatitis, that can affect any part of the dog’s body. Any breed can develop a hotspot although certain breeds, particularly Golden Retrievers, Labradors and Rottweilers, seem predisposed to developing this kind of rash.

The most common underlying cause leading to the development of a hotspot is fleas. Other possible causes include ear infections — if the hotspot occurs on the face or neck — anal sac disease, allergies, dry skin, fatty acid deficiency or an underlying medical condition. In some cases the underlying cause cannot be determined.

SEVERITY: Moderate. Painful and debilitating if left untreated.

Hotspots begin with an irritating itch, which leads to severe scratching which results in damage to the skin. Consequently, the skin begins to ooze serum resulting in matting of the hair, which promotes bacterial growth. The hotspot becomes even itchier, which leads to more scratching which in turn leads to further damage to the skin. A destructive cycle is begun where the hotspot progressively worsens.

Hotspots are diagnosed by their distinctive appearance. They appear as a moist, itchy lesion with slimy discharge and matted hair on the surface. Hotspots often have a sudden onset and can sometimes occur within a matter of hours so it is important to seek treatment as soon as any signs are observed.

Treatment usually involves clipping of the matted hair over the hotspot to allow it to be cleaned with a mild antiseptic to assist in removing the discharge and surface bacteria. A topical antibiotic/anti-inflammatory cream is then applied. Systemic antibiotics are often needed to help fight the infection within the deeper layers of skin.  Cortisone, an anti-inflammatory, is often also prescribed to reduce the itchiness of the hotspot and break the destructive cycle of itching.

If an ear infection is suspected then a thorough ear examination will be performed and appropriate medications dispensed.

It is important that the underlying cause is addressed to avoid a recurrence of the issue. Dogs should be treated monthly with an effective flea treatment such as Frontline Plus or Advantage. Flea collars and shampoos will not provide adequate flea control. Early detection leads to quicker resolution of hot spots.

Hypothyroidism is caused by an inactive thyroid, often following the destruction of the thyroid gland, usually by the patient’s own immune system. Genetics play a role in the condition, which is more common in certain breeds.

SEVERITY: Chronic. Requires ongoing medication.

Visible symptoms usually develop during middle age, from about 2 to 6 years of age. Signs are quite variable and depend, in part, on the age of the dog. In the adult, the most consistent signs are related to changes in activity and mental status. The most common signs seen are those related to skin and hair coat.

Some or all of the following signs may be seen:

  • lethargy, mental dullness, inactivity, weight gain, cold intolerance
  • hair loss, ‘rat tail,’ dry brittle hair coat, coat that is slow to grow, scales and dandruff, hyperpigmentation (where skin changes to black colour), skin infections, ear infections.
  • reproductive abnormalities
  • seizures, weakness, difficulty maintaining balance
  • eye problems
  • bradycardia (slow heart rate), cardiac problems
  • diarrhoea, constipation
  • anaemia, bleeding disorders.

Fasting blood tests are required for diagnosis. Generally we recommend a full blood profile at the same time to assess general health and to monitor for changes that are secondary to the hypothyroidism itself.

Hypothyroidism is treated by supplementing the thyroid gland with a synthetic hormone. An initial dose is calculated based on the patient’s bodyweight and tablets are given twice daily. Because of the variability in response to the treatment in individual dogs this initial dosage and frequency may require adjusting before a complete clinical response is observed.

After a month on a set dose blood tests are repeated to check for blood levels, measuring hormone levels just before tablets are given then again several hours after to measure the lowest and highest levels achieved in the bloodstream.

An increase in mental alertness, activity and appetite are the initial signs of improvement and are usually seen in the first week. Noticeable changes in the skin and some hair improvement should be seen in the first month. However, the cost often looks worse before it looks better as the old hair is shed in large quantities. It may take several months before complete regrowth occurs.

Once a dose is decided on and the patient stable blood tests are repeated every 6-12 months as dose requirements can change over time. The prognosis for dogs suffering hypothyroidism is excellent.

 

Kennel cough, or infectious canine tracheobronchitis, is a highly contagious* condition in dogs that affects the throat, trachea (windpipe) and, on rare occasions, the lungs. It is most commonly characterised by a persistent, dry, harsh hacking cough, often producing white frothy saliva, which can lead pet parents to mistake the coughing for vomiting.

Kennel cough can be caused by a variety of different viruses or bacteria, but is most commonly triggered by the presence of both the parainfluenza virus and the bacteria Bordetella bronchiseptica. Although dogs can be largely protected by routine annual vaccinations, unfortunately, we can only vaccinate against these two most common strains. Some dogs vaccinated for kennel cough can still contract the disease. However, in vaccinated dogs the disease is generally a lot milder and not as prolonged as in those animals not vaccinated. It is also much less likely that the disease will progress to pneumonia if the dog is vaccinated.

Kennel cough is highly contagious. It is transmitted by respiratory secretions by direct nose to nose contact or via aerosol transmission — sneezing or coughing. It occurs most frequently in kennelled dogs because they spend a lot of time barking at each other, hence the name. However, dogs can also contract it just by sniffing noses through a fence or while on a walk.

In many cases the infection may run its course and the dog recover without treatment. However, due to the risk of pneumonia, we do strongly recommend that all dogs suffering kennel cough are treated, and vaccinated if necessary. The infected dog must be isolated from other dogs while the condition remains.

*Important! If your dog has any kind of cough and you are making an appointment to bring him or her to the clinic for a checkup, please inform us about the cough. We will ask you to wait in the car for a vet to check your dog before he/she comes into the hospital. You’ll be helping us avoid exposing other patients to a potentially infectious condition. 

SEVERITY: Potentially serious but rarely life threatening.

The cough normally has a sudden onset and occurs within 3 to 10 days of being in kennels or having been exposed to an infected dog. Most dogs will appear healthy apart from the annoying cough.

The majority will also have a fairly normal appetite although some may refuse food, become lethargic and may have a fever. On very rare occasions, and depending upon the bug which caused the disease, kennel cough can spread to the lungs and cause pneumonia.

Diagnosis is generally straightforward and requires only a visual and auditory examination together with a history of any time spent in a boarding kennel or shelter. In some cases we may take bacterial cultures, viral isolations, and bloodwork to identify the specific pathogens causing the exact type of kennel cough the dog has. X-rays, which can show bronchitis, are occasionally used.

Treatment for kennel cough is designed to give your dog relief, shorten the course of the disease and help prevent pneumonia. Treatment may include a short course of antibiotics and, if required a cough suppressant. An anti-inflammatory injection may also be given to help ease the throat.

The cough should start to improve within 3-5 days of beginning treatment, although sometimes the cough can get a lot worse before it improves. Any deterioration in your dog’s health should be brought to our attention as it may be a sign of developing pneumonia and we may recommend chest radiographs to confirm this.

Due to the extremely contagious nature of this disease it is important to keep your dog away from other animals for 2 weeks after the cough has resolved. It is also important to keep your dog confined and limit exercise as activity tends to irritate the windpipe further, which in turn worsens the cough. Also make sure your pet is kept warm, especially at night as cold air also tends to irritate the throat further.

The patella is the bone commonly called the knee cap. In the dog, normal movement is for the patella to slide up and down within a groove on the femur — the thigh bone, the bone above the knee joint — as the knee is bent and straightened. Sometimes a patella will luxate (dislocate), meaning it slips out of this groove and to the side.

A luxating patella will always cause discomfort for the dog and in chronic cases the pain will be severe. Left untreated arthritis will develop, leading to further chronic pain. 

SEVERITY: Initially mild developing to severe. Treatable by surgical correction.

A luxating patella may be caused in one of two ways. It may be an inherited (genetic) problem. Depending on the breed and size of the dog in most small dogs the patella usually luxates medially (towards the mid line of the patient, i.e., to between the legs rather than towards the outside of a leg). In large dogs the patella generally luxates laterally.

The degree of patella luxation caused by poor conformation of the leg varies from a looser than usual patella that generally stays in place but luxates when being examined, to a patella that is permanently luxated and cannot be manually replaced.

The signs of patella luxation therefore also vary — from occasional ‘skipping,’ where the leg is carried with the knee bent for one or more steps, to significant lameness. These signs are usually evident from a young age, although mild cases may not be noticed by owners until arthritis develops. This occurs at a younger than usual age causing more obvious significant lameness.

Conformation problems cause luxating patellae most commonly in toy and miniature dog breeds, for example chihuahuas, toy and miniature poodles, maltese and shih tzus. As this is a genetic problem, breeding of dogs with this problem should be avoided. Around 50 per cent of patients will have the problem in both knees, and around 15-20 per cent of those patients with chronic patella luxations will also rupture their cranial cruciate ligament. Patella luxation of this type usually worsens over time.

A second cause is trauma, such as a hard knock to the knee. In these cases the patella will luxate to one or both sides and the patient will show signs of sudden onset lameness, pain and swelling.

A luxating patella is usually obvious after a simple physical examination and a visual observation of the dog’s gait. We will however take radiographs prior to surgical correction to establish the extent of surgical repair required.

Apart from the mildest cases, all patella luxations require surgical correction. More severe cases suffer constant pain and lameness. All cases, even the mild cases that don’t appear as much of a problem for the young pet, will lead to early arthritis from the constant abnormal rubbing of bone, gradually wearing away the cartilage lining the bone. Arthritis can be very painful and early prevention by surgical correction of the problem is the best way to protect your pet from future pain and discomfort.

The degree of patella luxation determines the degree of repair performed. For a simple lateral ligation your dog will be home the same day. Severe cases requiring groove deepening and lateral ligation involves an overnight stay in the hospital for monitoring.

Procedures performed include:

  • Surgically deepening the patella groove
  • Tibial crest transposition
  • Medial desmotomy, which involves releasing tissues on the medial side of the joint.
  • Lateral imbrication, or the tightening of tissues on the lateral side of the joint.

Pain relief is given to minimise pain and encourage movement.

Home care after surgery is an important part of recovery. If your pet is not using the leg, physiotherapy by gently bending and straightening the leg is recommended. Short, gentle, slow leash walks on even ground surfaces should also begin a few days after surgery. These should initially be only for a few minutes, gradually increasing in length over the following 6 to 8 weeks.

The aim is to encourage early but controlled use of the leg. Exercise must be carefully restricted to prevent injury. After the first two weeks, swimming is excellent exercise for the leg, as it is non-weight bearing and reduces the chance of slipping and injuring the leg as well as forcing those reluctant patients to start using the leg again.

Healing time ranges from around 4 to 10 weeks depending on the surgical techniques needed for repair. Overweight pets put extra stress on all joints and should be brought back to normal weight. Please discuss your pet’s weight with our staff to ensure a desired weight loss is achieved.

We will begin a course of pentosan (anti-arthritis) injections one week after surgery. This involves a course of 4 weekly injections followed by monthly or 3 monthly boosters depending on whether arthritis is already present.

 

Demodex canis is a mite that lives in the hair follicles of canine skin and is present in small numbers in most healthy dogs, horses and even humans. Transmission occurs from the bitch to nursing pups by direct contact during the first 2 or 3 days of neonatal life. However, in certain dogs these mites can multiply on the skin to become a condition known as demodicosis, or demodectic mange. The condition is more common in purebred dogs and certain breeds have far more frequent disease than others, but any breed can develop the disease.

SEVERITY: Mild to Severe depending on the type of infection.

There are two types of clinical conditions seen with demodicosis:-

1. Localised demodicosis is usually seen in young dogs and commonly occurs on the face and forelegs. The condition often heals spontaneously but this type of demodicosis may also progress to the generalised state.

2. Generalised demodicosis covers a large area or large patches of the body and secondary bacterial infection is common. There are three forms of generalised demodicosis:

i) juvenile onset: usually starts between 3 and 18 months of age. If the lesions do not resolve spontaneously or receive adequate treatment, the patient carries the disease into adulthood.

ii) adult onset: the dogs are generally over 5 years of age and an underlying disease or immunosuppression (with the demodex as a secondary problem) should be suspected.

iii) chronic pododermatitis where the disease is confined to the paws. These cases are particularly resistant to therapy.

A complete cure for the generalised form of the disease is not always possible. In these cases, lifetime therapy may be necessary for control.

Demodectic mange is diagnosed by taking a skin scraping to demonstrate the nature and quantity of mites in the skin of the dog. The microscopic debris and hair collected are examined under the microscope where the immature forms and eggs are visible. Sometimes a skin biopsy may be required under general anaesthesia.

Localised demodicosis is a mild disease that usually heals spontaneously in 6-8 weeks, but may wax and wane in a localised area for months.

Generalised demodicosis is a more serious form of the disease and is difficult to treat. In the case of adult onset generalised demodicosis it is always necessary to search for predisposing factors such as Cushing’s disease, neoplasia, or other medical diseases causing immunosuppression etc. The dog will be placed on a nutritionally balanced diet with vaccinations and heartworm preventatives kept current. Any significant stresses should be removed. Corticosteroids are contraindicated in all cases of demodectic mange.

Strictly speaking, obesity is not in itself an ‘illness’ or a disease. You can call it a syndrome; a condition of excess body weight, defined as fat accumulation, where the body weight is 15% or more above the ideal weight, and great enough to cause harm to health or wellbeing. We include it here amongst our disease fact sheets because of the rapidly growing problem of overweight dogs (and cats) in Australia. Dogs and cats are covered here.

In recent years we have seen what can only be described as an explosion in the number of obese cats and dogs coming into Alpine Animal Doctors. Like diabetes, a serious illness in which obesity can be a major causative factor, this increase in the number of ‘fat pets’ we see is mirroring trends in the human population. And for exactly the same reasons. Put simply, too many pet owners are either feeding their pets too much of the wrong foods, and/or failing to provide them with enough healthy exercise.

 Obesity may not be an illness itself, but it is a debilitating condition that is linked to a wide range of health issues with serious consequences for you pets. Fortunately it is also very treatable.

SEVERITY: Potentially serious and, in some cases, may be life threatening.

Obesity is caused by three main factors:

Overeating. While cats and dogs can regulate the amount they eat by their energy needs, this ‘natural’ regulator can be overridden if the flavour of the food on offer is very desirable, and food is presented more often or in larger quantities than necessary.

Insufficient exercise. Animals tend to become more sedate as they age, when ill or after neutering. The less the animal exercises, the less calories they burn, and therefore the less calories they should be eating.

Decreased energy requirements. Every animal (including human) has his or her own metabolic rate, which can be influenced by many factors including hormones, climate, reproduction status, and body weight. If an animal’s metabolic rate is decreased by any one of these factors, the body automatically stores the energy it is no longer burning as fat.

Other risk factors include gender, breed, and owner lifestyle but, basically, the root cause of obesity is the unnecessary consumption of more calories than the animal needs.

Obesity has been linked to many adverse effects and serious illnesses in both dogs and cats. Common problems caused or exacerbated by obesity include:

  • Diabetes Mellitus (Type I and Type II diabetes).
  • Hypertension, or high blood pressure, caused by the excess workload on the heart and lungs.
  • Neoplasia. Many recent studies have found the incidence of cancer is dramatically higher in obese patients compared to lean patients.
  • Decreased immunity. The body’s ability to fight disease is greatly reduced.
  • Hepatic Lipidosis/Fatty Liver. Fatty accumulation in the liver of obese cats that stop eating (e.g., if they are unwell) can cause severe illness and possibly death.
  • Osteoarthritis is aggravated by obesity because of the extra weight on the joints.
  • Skin infections and matted hair may result from decreased grooming in obese cats.
  • Bladder infections can result if excess weight prevents a cat from grooming its hind end.
  • Obese cats may suffer from increased rates of feline lower urinary tract disease.
  • Obesity may accelerate and exacerbate kidney disease and heart disease. Note that obesity, by itself, usually does not cause heart disease.
  • Obese pets may suffer respiratory and breathing problems.
  • Obese pets are more likely to suffer complications during anesthesia and surgery.

Just about all of these conditions can be avoided by keeping your pet’s weight at an optimum level. To assess the ideal weight for a dog, run your hands gently down the animal’s sides and spine. Even with a light touch you should be able to feel the rib cage and individual vertebrae, but not see them. There should also be an indentation between the rib cage and the hips — this is the dog’s waist line.

Testing a cat’s bodyweight is slightly different as the fat often hangs around the groin and belly area. The rib and spine test for dogs can sometimes be accurate, although it is important to also check for a ‘pouch’ of fat hanging around the abdomen.

‘Treatment’ for obesity is, happily, not difficult. If your pet is diagnosed with obesity, you simply need to take control of his or her food intake. A balanced diet is essential for good health. But please note that we are not talking about just putting you cat or dog ‘on a diet’ here. Eliminating obesity requires a permanent lifestyle change.

The easiest and most cost efficient way of dieting your pet is with a high quality, balanced, commercial weight control premium pet food. We are happy to advise you on the most appropriate nutrition plan for your pet. You will also need to monitor the entire food intake, which means fewer treats, and only treats of the low calorie or low fat variety. Regular weigh-ins at the hospital are recommended so we can chart the progress and make adjustments if necessary. Maintaining goal weight when reached is simply a matter of adjusting the calorie level slightly and, once again, a balanced weight control diet is best.

Our recommendations for using these commercial preparations is not so we can sell you more pet food. If you can prepare complete, balanced low calorie meals at home, that’s great. But it’s almost impossible for an individual to do that. Achieving balanced nutrient diets to bring an animal back from obesity requires a scientifically formulated diet.

Many people with obese animals tell us that they only feed their pets once a day. Perhaps, but it is not the number of meals each day, or even the quantity of food, the problem lies in the quantity of calories they are given each day. It is better for pets to get two smaller meals a day rather than one large one, as this increases the body’s metabolism, and helps to burn calories.

There’s no getting around it — if your pet is overweight, it must be getting more nutrition than it requires. The body stores only the energy it does not need. In short, there are only two ways to defeat obesity — reduced calorie intake, and higher levels of appropriate exercise, preferably a combination of both.

It is of course more difficult to get a cat to exercise. However, cat parents can encourage increased activity by offering toys that stimulate hunting instincts, and by encouraging the cat to both play and to spend time outdoors. Serving their food in a remote area of the house, or the end of the garden may reduce food intake because of the extra effort required to obtain food, and stimulate activity during the cat’s trips to and from their food bowl.

For both cats and dogs, the benefits of eliminating obesity and getting your pets back to a healthy body weight are so great that a caring owner will remain dedicated to the cause, regardless of the frustrations.

Osteochondrosis dissecans, or OCD, is a common bone condition occurring in young, rapidly growing dogs and seen in many breeds, particularly the larger breeds including the Rottweiler, Bernese Mountain Dog and Labradors.

The bones of growing puppies lengthen from each end, at the growth plates. As the bones grow, soft cartilage is deposited continuously at the growth plates and, over time, this cartilage is transformed into bone. OCD occurs when cartilage fails to transform into bone, continuing to grow and to become abnormally thick. This often leads to cracks in the cartilage which can result in a whole flap lifting up.

SEVERITY: Causes inflammation & pain, frequently Severe. Surgical correction required.

OCD causes considerable inflammation and pain in the joint, and affected dogs begin to display a limp. The most commonly affected areas are the shoulder and elbow, although the knee and hock can also be affected.

The exact cause of OCD is unknown but contributing factors include:

Rapid growth. The rarity of OCD in breeds weighing less than 23 kg indicates large size and rapid growth are important factors.

Genetics. The condition appears to hereditary.

Diet. Overnutrition with a high intake of calcium during growth is a contributing factor.

Trauma may also play a role in the development of OCD.

If your dog has a limp, we will conduct a complete physical examination to try to localise the pain to one or two joints. Once this is determined, x-rays will help us rule out other reasons why your dog may be limping, such as a fracture. 

OCD rarely heals by itself and in the majority of cases surgery to remove the cartilage flap is required.

Ingestion of human medicines is one of the major causes of poisonings in dogs and cats, accounting for 25 per cent of all poisoning cases. You should never give human pain relief medications to your domestic pets.

In particular we see an alarming number of dogs (and cats) who have been given paracetamol (sold in supermarkets and pharmacies under brand names such as Panadol, Panamax etc.) for pain and fever relief. While you may be tempted to try to provide some relief for your pet by using your own medicines, or other over-the-counter painkillers you might have, the reality is you may be killing your pet. Paracetamol is particularly toxic to cats and dogs. If it doesn’t kill them immediately the toxic substances can be stored and accumulated in internal organs.

Other common over-the-counter pain killers such as Ibuprofen are also toxic to pets. In humans, Ibuprofen works to block the chemicals in the body that cause pain, fever and inflammation. The drug is a component in many anti-inflammatory products and it can be potentially toxic to dogs, especially smaller breeds, and to cats. Giving even one tablet of Ibuprofen to pets can cause gastric ulceration.

It’s also important to be aware that using any human medications prior to visiting your vet can create difficulties in treatment. For example, if surgery is necessary we will be unable to provide appropriate pain relief if there are potentially toxic human drugs in your pet’s system.

Animals have a different physiology to humans and products that may be safe for us can be very harmful to them. There are more appropriate drugs for animals that have been tested and approved by the relevant authorities.

In an emergency it is best to contact the hospital for advice. At Alpine Animal Doctors an emergency vet can be contacted 24 hours a day and we would prefer that you woke us up to ask for advice rather than self medicate your pet.

SEVERITY: Can be fatal. Do not give pets paracetamol.

Paracetamol causes extreme breathlessness by dangerously altering the red blood cells. It changes the red pigment of the blood (haemoglobin) into a compound known as methaemoglobin. This substance does not carry oxygen and results in the animal’s gums and lips turning blue. Fluid-like swelling occurs around the face and the pet begins gasping for breath.

A mild case of paracetamol poisoning can give your dog an upset stomach, and they may appear to be tired. Other symptoms can include laboured breathing, swelling on the face, neck or limbs, gums turning brownish-grey and jaundice. More serious cases can include signs of staggering, vomiting and restlessness. There can be blood in the stool and the urine will seem unusually dark-coloured. In these cases you need to get your dog to a vet as quickly as possible.

We will need to conduct a full physical examination and a biochemistry panel, complete blood count, urinalysis and faecal examination to determine the level of toxicity currently in your dog’s body. We will closely monitor your dog’s liver and kidney function for any changes that may indicate their liver or kidneys are damaged and failing.

Hospitalisation is usually required while undergoing treatment for paracetamol poisoning. The first step is decontamination, then blood tests to check your dog’s liver and kidney functions. A gastric lavage may also be carried out, or vomiting induced to rid your dog of any paracetamol still in their stomach. Further care generally includes oxygen, intravenous fluid therapy, vitamin C supplementation, cimetidine and N-acetylcysteine. Cysteine is an amino acid that aids in repairing any potential damage that has been done to the liver.

Once your pet is able to go home we will need to continue to monitor the kidney and liver function to ensure that there are no long-term effects.

Pancreatitis is a failure of the pancreas, an organ in the abdomen, to do its job. The pancreas has two main functions — to produce digestive enzymes and insulin. The digestive enzymes pass down a duct and empty into the intestine. Eating stimulates the pancreas to increase production of digestive enzymes, which digest meat, carbohydrates and fats. In dogs, this duct enters very close to the opening of the bile duct, which brings bile from the liver to the intestine. In cats, it shares the opening of the bile duct.

Occasionally some of these enzymes may leak into the pancreas and surrounding tissues and the pancreas starts to ‘digest’ itself, causing pain and inflammation — pancreatitis — and sometimes peritonitis.

Pancreatitis may occur simply because a patient has eaten a particularly fatty meal, causing large amounts of digestive enzymes to be released, or there may be no obvious explanation. However, once patients have had one episode of pancreatitis they are more prone to experiencing repeated bouts of the condition. Pancreatitis is much more common in middle-aged, overweight, female dogs but it can occur in male or female dogs of any age or weight.

SEVERITY: Can be serious and, occasionally, fatal without proper treatment.

Patients with pancreatitis may just present as lethargic but vomiting is common, as is diarrhoea. Your pet’s appetite may or may not be affected but eating will usually cause vomiting. Thirst may also be increased and drinking will often also cause vomiting. There may be fever and dehydration and, often, abdominal pain which the dog may attempt to ease by getting into a ‘praying’ position — standing on hind legs, but with elbows on the ground.

Jaundice — yellowing of the skin and other surfaces — may be seen as an inflamed pancreas can block off the bile duct and cause yellow pigment to build up in the blood.

Diagnosis is usually made on a combination of clinical signs and the results of blood and urine tests. The blood tests assess the level of two enzymes (amylase and lipase) that the pancreas releases into the blood. Also examined are the level of inflammatory response, electrolytes, and liver and kidney function, to give a measure of severity of the disease process and help us rule out other diseases with similar signs.

Treatment involves preventing further damage and reversing the signs, generally using a five-pronged approach.

  • We will first place the animal on a ‘no food or water by mouth’ regime to reduce the activity of the pancreas.
  • Provide intravenous fluids, often with a vitamin supplement, to maintain hydration and flush out toxins.
  • Antibiotics will be prescribed to prevent the damaged pancreas becoming infected.
  • Anti-vomiting drugs will be used to settle the stomach and reduce nausea.
  • Pain relief will be given to reduce abdominal discomfort.

As the vomiting and pain subside — usually over 1-5 days — small amounts of water are offered. If no vomiting occurs then bland food is offered. It is very important not to feed too early or too much; if vomiting recurs you can be back to square one. Gradually meals are made larger and mixed with normal (low fat) food.

Occasionally the pancreas can develop one or more abscesses (pockets of pus) after inflammation. This may require ultrasound examination and surgery to diagnose and fix. Rarely, usually after repeated bouts of pancreatitis, the pancreas may have lost so much tissue that it can no longer produce enough enzymes. If insulin production is affected this will make the patient diabetic. If digestive enzyme production is affected special supplements will need to be added to the food so proper digestion can occur.

Pancreatitis is a quite serious condition. Not all cases require hospitalisation but the majority do. Most patients pull through with proper care and treatment but occasionally a serious case is fatal. The longer a sick animal is left without treatment, the more likely it is to be one that will not survive.

Most dogs will never get pancreatitis, but there are things you can do to improve the odds of your pet suffering the disease. Always avoid very fatty meals and ensure your dog is not overweight.

If a dog has had pancreatitis once this should be taken as a very serious warning to feed only reduced fat food from then on. It takes only one dim sim, or one sausage from the barbecue to potentially put the patient back into hospital in a serious condition. This may mean warning visitors not to feed your dog, or even locking him up until visitors leave. Special prescription diets are often recommended to reduce the risk of another bout of the disease.

Canine parvovirus (CPV) infection is a highly infectious disease that was first identified only in 1978. Despite that, it is relatively well known amongst dog owners because of the severity of the disease and its rapid spread through the canine population. The virus that causes it is very similar to feline enteritis and the two diseases are almost identical.

The main source of the virus that causes CPV is the faeces of infected dogs, which can carry a high concentration of viral particles. Susceptible animals become infected by ingesting the virus, which is carried to the intestine where it invades the intestinal wall and causes inflammation.

Unlike most other viruses, CPV is stable in the environment and resistant to the effects of heat, detergents and alcohol. CPV has been recovered from dog faeces even after three months at room temperature. This stability helps the virus to be easily transmitted via the hair or feet of infected dogs, contaminated shoes, clothes, and other objects. Direct contact between dogs is not required to spread the virus. Dogs infected with the virus and showing clinical signs will usually become ill within 7-10 days of the initial infection.

Affected dogs must be isolated to prevent the disease spreading. This is usually best done in the hospital’s Isolation Ward.

SEVERITY: Highly infectious and potentially fatal without prompt treatment.

The clinical signs of CPV disease can be variable but generally take the form of severe vomiting and diarrhoea. The diarrhoea may or may not contain blood. Additionally, affected dogs will often exhibit a lack of appetite, depression, and fever. Many dogs may not show every clinical sign but vomiting and diarrhoea are the most common, with vomiting usually the first indicator.

Parvo can affect dogs of all ages but is most common in dogs less than a year old. Young puppies less than five months of age are often the most severely affected, and the most difficult to treat.

The clinical signs of CPV infection are very similar to other diseases causing vomiting and diarrhoea, making the diagnosis of CPV a challenge for the veterinary surgeon. A positive confirmation of CPV infection requires the demonstration of the virus in the faeces, or the detection of anti-CPV antibodies in the blood serum. Occasionally a dog will have parvovirus but test negative for virus in the faeces. Fortunately, this is rare.

A tentative diagnosis is often based on the presence of a reduced white blood cell count (leukopaenia). If further confirmation is needed, faeces or blood can be further tested at the hospital. The absence of leukopaenia does not always mean that the dog cannot have CPV infection. Some dogs that become clinically ill may not necessarily be leukopaenic.

As with any viral disease there is no treatment to kill the virus once it infects the dog. However, the parvo virus does not directly cause death. It causes loss of the lining of the intestinal tract which results in severe dehydration, electrolyte (sodium and potassium) imbalances, and infection in the bloodstream (septicaemia). When the bacteria that normally live in the intestinal tract are able to get into the blood stream it becomes more likely that the animal will die.

The first step in treatment is to correct dehydration and electrolyte imbalances by the administration of intravenous fluids containing electrolytes. Antibiotics and anti-inflammatory drugs are given to prevent or control septicemia. Antispasmodic drugs are used to inhibit the diarrhoea and vomiting that perpetuate the problems.

Most dogs with CPV infection recover if aggressive treatment is used and if therapy is begun before severe septicaemia and dehydration occur. For reasons not fully understood, some breeds, notably the Rottweiler, have a much higher fatality rate than other breeds.

Parvovirus is almost fully preventable with routine vaccinations. Puppies receive a parvo vaccination as part of the vaccines given from 6 and about 10 weeks of age. After the initial series of vaccinations all dogs should receive an annual booster shot. Dogs in high exposure situations — kennels, dog shows, field trials, etc. — are particularly at risk of CPV. Bitches should be boosted before mating or immediately before whelping in order to transfer protective antibodies to the puppies.

If you have had an unvaccinated dog unfortunate enough to be infected with CPV, the stability of the virus in the environment makes it critical to thoroughly disinfect contaminated areas by cleaning food bowls, water bowls, and other contaminated items with a solution of 250 mL of chlorine bleach in 5 litres of water. The hospital can assist you with advice on the correct disinfection regime.

 

Pyometra is a life-threatening infection within the uterus. Most commonly seen in female animals over the age of six, pyometra is caused by repeated exposure of the uterus to the hormones of the normal reproductive cycle (when pregnancy does not occur), which stimulate glands in the inner uterus wall. These glands produce increased fluid that accumulates in the uterus and this becomes an ideal environment for bacterial growth.

In dogs we see pyometra 1 to 12 weeks after they have been in season. In cats pyometra is a lot less common and the timing can be a lot more variable. It is possible (though uncommon) for pyometra to develop in the very small end of the uterus remaining in place after a spey operation. Generally, however, desexing will prevent pyometra infections.

SEVERITY: Life threatening. Requires prompt treatment.

Pyometra can be open or closed, meaning the cervix is either open or closed. If it is open, there is usually a visible vaginal discharge of pus that may be bloody or blood tinged.

If the cervix is closed there is no drainage of the infection and the animal will often become much sicker, much faster. This is much more difficult to see and diagnose as there is no discharge. Pets with pyometra will go off their food, become lethargic, drink a lot, urinate a lot, and possibly start to vomit.

If there is a vaginal discharge and a good history, diagnosing open pyometra may be relatively easy after a clinical examination. Closed pyometra can be more challenging. It is important for us to rule out pregnancy. Sometimes radiographs are required, and a blood test is also important to confirm the diagnosis and assess kidney function.

Treatment must be prompt if we are to save your pet’s life. The treatment of choice is intravenous fluids, antibiotics, and to anaethetise the patient and remove the infected uterus surgically. As there is no further need for the animal to cycle following surgery, ovaries are also removed, as is done in a normal spey operation.

Recovery after surgery is usually reasonably rapid. A 1 to 3 day stay in hospital is usually all that is required, after which care can be continued at home with oral antibiotics, close monitoring, and some revisits to the hospital.

 

Sarcoptic mange, commonly called scabies, primarily affects dogs but can also temporarily affect cats and humans. Caused by a mite called Sarcoptes scabiei, scabies is extremely contagious and the mites are transferred to other dogs, and humans, by direct contact. Dogs can contract the mite by contact with foxes, wombats, rodents or a carrier dog. Symptoms may take 2 to 6 weeks to show after contact with an infected animal.

Note that Sarcoptes scabiei can be transmitted to people in close contact with an affected dog. An itchy rash may develop on the arms, chest or stomach area. The rash is usually transient and should resolve once the affected dog(s) is treated, however, if you appear to be affected you should seek medical advice.

SEVERITY: Mild to Moderate. Treatable.

The mites cause an intense itch as they burrow beneath the skin to lay eggs and secrete substances which cause a reaction in dogs. Infected animals develop a rash and hair loss, particularly in areas such around the eyes, elbows, hocks, abdomen and ear flaps. Often yellow crusts also form on these areas of the body. Long term infections of Sarcoptes scabiei can lead to the development of a deep bacterial skin infection.

Diagnosis is made by examination of skin scrapings under a microscope, or confirmed by response to treatment.  Skin scrapings are always performed, however, the mites live deep in the skin and can be extremely difficult to find. If a skin scraping is negative but sarcoptic mange is suspected, medication will be used and the diagnosis confirmed, or eliminated, based upon the response to treatment.

It can take up to a month for an asymptomatic carrier to show any signs, so all in-contact dogs should be treated, even those showing no clinical signs.

Treatment for Sarcoptes is usually via a specifically designed medication such as Revolution, a liquid applied on the back of the neck. The product is very safe and highly effective. Dogs should have a repeat dose two weeks after their first. Some severely affected dogs may also need antibiotics, anti-inflammatories and/or medicated shampoo.

Response to treatment should be seen within two weeks. Dogs at high risk of becoming infected with Sarcoptes, for example, those that live in areas of fox habitat, may need to be treated monthly.

  • Choke
  • Colic
  • Dental disease
  • Founder & Laminitis
  • Greasy heel
  • Rain scald
  • Seedy Toe
  • Strangles
  • Tetanus

Choke

Choke is a relatively common condition in horses. It occurs when a mass of food or a foreign body blocks the horse’s oesophagus (gullet), the tube that takes food from the back of the mouth (pharynx) to the stomach. Choke may be partial or complete.

The most common causes are a greedy horse attempting to swallow hay without chewing it thoroughly, swallowing food or other material which is either too dry or too coarse (most commonly hay), or food which swells rapidly once chewed (typically sugar beet) so that its passage down the oesophagus is slowed or stopped. It can also occur in foals who are given access to dry, coarse hay or straw. Any pre-existing condition which interferes with the horse’s ability to swallow, such as sedation, an injury to the neck or oesophagus, grass sickness, botulism, etc., can predispose a horse to choke.

Clinical signs:

The most visible signs are usually a discharge of saliva and feed material from the nostrils and/or mouth, depression and apparent difficulty in swallowing. When first ‘choked’ some horses will panic, making repeated unsuccessful efforts to swallow, or will cough and ‘gag’ as though trying to clear something from the back of the throat.

If the condition has gone unnoticed by the owner, the horse may become dehydrated and severely depressed. Although many cases clear on their own, and few are serious, it is possible for the oesophagus to rupture, and for death may follow due to shock and infection. While this is not common, the possibility does mean that the condition is classed as an emergency. if you think your horse has choke, please call us immediately.

How is choke diagnosed?

We will normally pass a stomach tube via a nostril into the oesophagus to confirm that something is obstructing passage into the stomach. This examination will determine at what level the blockage has occurred, how solid it feels and if it can be gently massaged to pass on into the stomach.

Treatment protocols:

In most cases the saliva that is continually produced in the mouth will gradually lubricate the offending obstruction, eventually allowing its passage to the stomach. We may help speed this passage by administering a sedative or a spasmolytic injection to help relax the oesophagus. Sometimes, this is all that is required.

In other cases the obstruction can be gently encouraged to move on down into the stomach with the help of the stomach tube. If this cannot be achieved easily the horse is sedated and the obstruction flushed with water and lubricant via the stomach tube. Fluid is gently pumped in via the stomach tube and allowed to run out, gradually flushing some of the obstructing material out.  This can be a long process and patience is needed to avoid damaging the oesophagus.

In some uncooperative or solidly-obstructed cases, or where a horse is panicked, it may be necessary to anaesthetise the horse to allow flushing to be performed safely and thoroughly. Once the choke is cleared the horse should be fed sloppy feeds or grass for several days to allow any local swelling to subside.

The most important complication is oesophageal rupture either as a direct result of the obstruction or following attempts to dislodge it.

Another possible complication is pneumonia. Occasionally, the horse may aspirate (breathe in) fluid and/or food material into the trachea (windpipe) and lungs, causing infection which may be fatal.

Horse owners can help prevent choke by:

  • Soaking dry feed thoroughly to allow them to swell before they are eaten and swallowed.
  • Ensuring regular routine dental care to allow the horse to chew food thoroughly and effectively before it is swallowed. Injuries to the insides of the cheeks, caused by sharp teeth, will cause discomfort and may discourage a horse from chewing food properly.
  • Provide permanent access to clean water to encourage the horse to drink normally.
  • Some horses tend to choke on a particular feed. Once this is recognised, access should be avoided.

Colic

The condition described below — colic in horses — can be a critical, life threatening emergency. If your horse is suffering from any of the symptoms described call the hospital immediately (5756 2444) for advice.

The term ‘colic’ means simply pain in the abdomen — a tummy ache basically. However, while a tummy ache in people usually means little more than a couple of hours or days of discomfort, because of the complexities of the equine stomach, in the horse a bellyache is often much more serious, potentially leading to death within 12-48 hours. All horse owners should be constantly vigilant for signs of colic, and contact the hospital immediately if your horse shows symptoms.

There are many causes for the pain of colic, from mild to life-threatening or fatal. It can be very difficult in the early stages to distinguish the mild from the potentially fatal so all cases of abdominal pain should be taken seriously. When colic develops it is vital that we can quickly identify the cause in order to instigate appropriate treatment.

There are many types of colic but the major types include:

Impaction colic is when the intestine becomes blocked by a firm mass of food. Impactions most commonly occur in the large intestine at one of the flexures. This is a fairly common type of colic which can usually relatively easily treated. However, an impaction may also be just the first obvious sign in a more complicated case.

Sometimes gas will build up in the intestine, most commonly in the large intestine and/or caecum, causing gas colic. The gas stretches the intestine, leading to pain. Gas colics usually resolve fairly easily with appropriate treatment, although, again, it is important to ensure that there is no underlying reason for the problem.

Some cases of colic are due to increased intestinal contractions, the abnormal spasms causing the intestines to contract painfully. Known as spasmodic colic these cases usually respond well to treatment.

Displacement/volvulus/torsion colic, or ‘twisted gut’ colic is where a portion of the intestine has moved to an abnormal position in the abdomen. An incidence of ‘volvulvus’ or ‘torsion’ colic occurs when a piece of the intestine twists. The suspension of the small intestine from the mesentery and the fact that the anatomy of the horse is such that much of the large intestine is not fixed predisposes horses to intestinal displacements and torsions.

Except in rare cases these types of colic cause a total blockage of the intestine and require immediate surgery. In the early stages of a displacement/torsion colic, the signs may be similar to those of a horse showing one of the milder forms of colic. This is why it is vital that horse owners take all cases of colic seriously and seek veterinary advice as early as possible.

Some cases of abdominal pain are due to inflammation of the small (enteritis) or large (colitis) intestines. These are also serious clinical cases and require immediate veterinary attention.

When a horse gorges itself on grain or any feed which expands when dampened, the contents of the stomach can swell, causing a gastric distension/rupture. The horse’s small stomach and its inability to vomit mean that in these circumstances the stomach may burst. Death is inevitable once the stomach bursts. The intake of such feed should be strictly controlled but if you suspect your horse may have overeaten concentrate feeds, seek veterinary advice immediately.

In many cases of colic we can never fully determine the reason for the pain. Symptomatic treatment, close monitoring and attention to any adverse developments usually lead to resolution of the problem.

Clinical signs:

The signs of colic in horses range from almost imperceptible in mild cases to extremely violent in severe cases but the symptoms for any form of colic are the horse indicating it is in pain. The animal may sweat, paw the ground, purse its lips, kick out and perhaps try to bite at its stomach region. They may roll, sometimes assuming unusual positions in doing so. Classically, they may stretch out in a bid to relieve the discomfort. They may also appear drawn up at the flank. The onset is often sudden in colic. In some cases, depending on the cause, their pulse may be rapid, possibly over 100 beats a minute.

Some of the most common signs include:

  • Lying down more than usual
  • Repeatedly getting up and lying down again
  • Standing stretched out
  • Standing frequently as if to urinate
  • Turning the head towards the flank
  • Repeatedly curling the upper lip
  • Pawing the ground
  • Kicking at the abdomen
  • Rolling

What should I do if I suspect colic?

If a horse showing signs of colic is behaving violently call the hospital right away. Violent behaviour usually indicates severe pain, which usually equates with a serious case of colic. Time is of the essence in severe cases. Some horses may become quite violent with a relatively mild case of colic but it is best to err on the side of caution and assume the worst.

If the signs of pain are less extreme you can afford to take a few minutes to observe the horse’s appearance and behaviour before calling us. If you can do so easily, take his temperature, pulse and respiration rates. Note what his appetite has been like in the past day or so, and the consistency and frequency of defecation. Also note the colour of the gums. Ask yourself if the animal’s water intake has been normal in the previous day or two, and if he has had access to any unusual feed, whether any medications have been administered, and whether there have been any changes in management.

Armed with that information, call the hospital for advice. We will almost certainly arrange to come and see the animal unless we are confident it is a very mild case. Before the vet arrives it is important that you remove all food. Try to get the horse to a pen where you can make sure he does not eat.

Walking the horse can help distract him from the pain, but he should not be walked to exhaustion. If the horse insists on rolling, there is little you can do to prevent it. Try to get the horse to an area where he will do himself the least damage when he rolls.

Do not administer any drugs, remedies or ‘DIY treatments’ until we have seen the horse, or we tell you it is safe to do so.

How is colic diagnosed?

Initially we will attempt to identify the cause of the colic, and the stage it is at. An accurate assessment is critical to deciding on the most appropriate treatment. Generally we will check the pulse, temperature, and overall condition of the horse. A lower than normal temperature may indicate that the animal is in shock and in need of rehydration. We will listen for bowel noise, and may perform a rectal examination, and/or pass a stomach tube into the horse.

We are trying to determine if the pain is being caused by an overactive intestine or due to a blockage. The outcome in both these cases is usually positive, provided we are called in quickly enough. If the cause is a twisted bowel, the prognosis is less positive, unless the horse is well enough to undergo urgent abdominal surgery.

Treatment protocols:

Treatment depends on the outcome of our diagnosis and the cause of the colic. Treatment may involve administering oil and a wetting agent via a stomach tube to assist in lubrication. We may also administer a worming agent. Painkillers will almost certainly be given to ease the horse’s discomfort. Depending upon the status of the intestine, we may administer drugs to relax the bowel. Fluids may be given intravenously if the horse is in shock.

Colic caused by a twisting of the bowel is the most serious. Surgery is the only option to save the horse in these cases, provided it is strong enough to withstand an abdominal operation. Without surgery a horse is unlikely to survive beyond 24 hours.

The type and combinations of treatments will very much depend upon our diagnosis and the condition of the animal. The most important thing is early intervention and diagnosis.

How can I prevent colic? 

For a horse, colic is probably an unfortunate fact of life. Good management can reduce the incidence however. Horses which fall into high-risk categories, such as stabled horses in intense training and fit horses recently injured, should be monitored particularly closely.

  • Allow as much natural paddock grazing as possible
  • Maintain a regular and controlled feeding schedule
  • Ensure constant access to clean water
  • Provide at least 60% of digestible energy from forage
  • Do not feed excessive digestible energy
  • Do not feed mouldy hay or grain
  • Feed hay and water in preference to grain
  • Provide access to forage for as much of the day as possible
  • Do not overgraze pastures
  • Do not feed horses immediately after work, or give cold water while a horse is still hot following exercise
  • Maintain a consistent exercise regime
  • Make all changes in diet, exercise level and management slowly
  • Control intestinal parasites by worming horses regularly (90 per cent of colic cases are caused in the first instance by parasites — mostly strongyles)

Dental disease

Periodontal disease in the horse is similar to dental issues in other domesticated animals and in humans. The disease begins when bacteria in the mouth proliferate around the teeth and gums, most commonly due to the trapping of food between or around the teeth and the formation of plaque. The body’s defences are overwhelmed by bacteria which attack the gingiva or gums, leading to inflammation and gingivitis.

As the gum becomes more inflamed it swells and begins to bleed, allowing bacteria to enter the bloodstream and thus placing all of the vital organs at risk of infection. As the disease progresses the attachment between the gum, the teeth and the jaw is lost. At this point periodontal disease can be stopped but cannot reversed. If the disease process continues it will lead to the loss of periodontal attachment around the tooth, which becomes loose and must be extracted. This can have life long consequences for the horse, who relies on healthy teeth to gain adequate nutrition from feed.

As a herbivore, the horse has teeth designed to grab and grind forage (grasses). To do this efficiently there are some important differences in the anatomy of their teeth compared to ourselves and other domesticated animals such as dogs. The teeth of the horse are are constantly worn away by the actions of chewing and grinding. To combat this wear and tear, horses have two sets of teeth during their life. The horse possesses extra tooth or reserve crown below the gumline in the jaw and this grows out (erupts) at a similar rate to the rate at which tooth is worn away. There is enough of this reserve crown to last the average horse well into their twenties, and often their thirties.

This system of tooth being worn away and replaced must remain in balance or problems will quickly occur. Just like people, horses can experience severe dental pain and pain is the most common reason the system become imbalanced; in response to pain the horse will alter the way it eats and chews, which rapidly affects the wear patterns of the teeth. Dental pain in the horse has a number of sources. The maxillary branch of the infraorbital nerve innervates the teeth of the upper jaw, while the mandibular branch of the trigeminal nerve innervates the teeth of the lower jaw, leading to severe pain caused by sensation to these nerves.

Horses also experience pain due to oral ulcers, impacted teeth, feed packing into the diastema — the spaces between the teeth — fractured teeth, unopposed teeth erupting into soft tissues and tooth root infections causing abscesses.

Horses evolved to graze with their heads down at ground level, eating grasses for many hours a day. Altering their diet and their living conditions can mean we also alter the way their teeth wear. Feeding of hard feeds, grains, chaff and pelleted feeds alters their normal grazing patterns and forces shorter feeding times. It also often forces them to eat with their head up. These management changes can all affect the dentition of the horse.

Horses have another important anatomical difference from people. Seen from the front, their upper jaw is wider than the lower jaw. This means that the outer surface of the upper teeth, closest to the cheeks, and the inner surface of the lower cheek teeth, closest to the tongue, are both unopposed. Through the continual process of eruption the edges, or cingulae, of these teeth grow but are not worn away but form razor sharp points along the arc of teeth.

When the horse eats it is forced to bring its cheeks into contact with these points. Unlike us, the horse does not chew in a simple up or down or side to side motion but in a complicated four part cycle, so these sharp points act on the horse’s cheeks and tongue like a saw, causing ulceration, cuts and, if bacterial infection occurs, abscesses. Fortunately, these cingulae, or sharp points, are formed of enamel, meaning these points can be removed relatively easily, providing relief for the horse and, usually, a return to a normal chewing cycle.

Clinical signs:

The early signs of periodontal disease in horses can be difficult to spot. More advanced signs are easier, but once disease is advanced it cannot be reversed so it is important that horse owners make themselves aware of the normal signs of healthy dentition and carefully note any changes from the norm. Call the hospital if you see signs that your horse is experiencing discomfort, is not eating normally or has sharp points on the upper and lower teeth.

The most effective way to prevent equine periodontal disease is to have the teeth of your horses checked annually.

How is equine periodontal disease diagnosed?

Dental disease is primarily diagnosed by visual exam. It is important that a thorough examination is made of a horse’s mouth as there are many other conditions that commonly occur in the mouth, many of which cannot be diagnosed without visualisation.

Treatment protocols:

Treatment is aimed at assisting the body’s natural defence systems by removing excessive bacterial growth and maintaining a balance of the bacterial population in the mouth. How this is achieved depends on the location and severity of the disease, as well as other factors such as the age of the horse and any other health conditions they may have.

Our dental care is based on an oral examination, then a dental rasp to grind the apposing surfaces of their sharp hooks and ramps. In a few rare cases, mostly in old horses, ‘wave formations’ — where the upper or lower arcade has lost a tooth and because of the continual dental growth, the apposing tooth has grown down to the point of preventing chewing — we will usually anaesthetise and then use gigli wire to saw through the offending lower portion, and trephine out the upper portion.

We will remove plaque and where food is trapped in the spaces between teeth remove the offending material to prevent bacterial population, followed by an antibacterial solution to kill any remaining bacteria. This treatment is practical only for incisor teeth. For cheek teeth the space where the food is packed may need to be widened to allow the food to fall out before it is overrun by bacteria. Patients may also need to be on systemic antibiotic therapy, and x-rays may be required to assess the structures below the gum.

Founder & Laminitis

Founder, or laminitis, is a common condition affecting the feet of horses. An extremely painful condition, founder/laminitis can affect every breed and is a major concern of caring horse owners. Though easy to diagnose and generally treatable, left untreated the condition can result in permanent severe lameness, with euthanasia sometimes the most humane option.

Founder occurs when there is inflammation of the laminae, the folds of tissue connecting the pedal (or coffin) bone to the hoof. When there is subsequent degeneration of the laminae, laminitis results. To properly understand how your horse might be affected it may help to understand what  the terms ‘laminitis’ and ‘founder’ mean, and how they interact.

Laminitis is the painful swelling of the laminae, the substances that attach the hoof wall to the pedal bone. There are four stages a horse with laminitis may progress through:

The Developmental Phase is seen for approximately the first 20-72 hours, in the time after the ’cause’ and before the first signs of lameness.  There are few clinical signs during this phase and the hoof wall should appear as normal.

This is followed by the Acute Phase, approximately 36-72 hours after the first signs of lameness. Heat will generally be found in the hoof at this point but the hoof wall will still appear as normal.

The Sub-Acute Phase begins some 72 hours after the onset of lameness. This is the recovery stage when proper treatment has been given, pain is receding and proper care must be maintained to prevent any further damage. It may take up to 12-14 months for full recovery. The hoof wall may appear to have a ‘ring’ of protruding hoof growth that will continue to grow downwards from the coronet during this stage.

Chronic Laminitis is the stage that can be avoided if the disease is caught and treated early enough. At this point, the structures inside the hoof have been altered permanently and the pedal bone has rotated downwards in angle, front tip first.  This is where the terminology combines and we begin to use the word ‘founder’ to describe the condition.

Founder is the actual misalignment of the bone structures inside the hoof due to destabilisation of the hoof capsule. The inner hoof wall has torn its attachment to the pedal bone, destabilising the bone structures. At this point, the pedal bone is no longer supported within the hoof and rotates toward, sometimes through the sole.

The terminology is used in various ways within the veterinary profession. For simplicity, at Alpine Animal Doctors we will generally use the term ‘founder’ to denote those horses with pedal rotation, and ‘laminitis’ for those horses that suffer from bouts of inflammation with no rotation.

Founder may represent itself on the outside of the hoof wall in the form of ‘rings’ of protruding growth, signs of stress that can occur over time. These rings will grow from the coronet downwards.  The hoof wall may begin to curl upwards at the toe, and the sole may become flatter due to the misalignment of the hoof structures.

Although founder is still poorly understood, and the exact mechanisms that cause laminitis are yet to be determined, we do know that there many causes of the condition. Classically, founder would occur in an overweight pony, complete with cresty neck, with a good appetite and access to plenty of lush pasture. In any horse, access to excessive amounts of pasture, hay, grain or pelleted feed is the most common cause of founder.

Other definitively known causes include blood poisoning following a systemic infection, severe cases of colic or enteritis, physical trauma to the soles of the hooves, ingestion of large amounts of cold water, retained placenta following foaling (particularly in shire breeds), and excessive weight bearing on one limb.

A sudden onset of founder is known as acute laminitis, and is considered a potentially life threatening emergency. Ongoing low grade laminitis is often referred to as chronic founder. Any horse that has suffered a previous episode of founder is more susceptible to a future attack.

Clinical signs:

Although founder can occur in one or all limbs, in most cases it is both forelimbs that are most commonly affected. A foundered horse will be reluctant to move, and will often lean back in an attempt to take weight off the front of the hooves. The pain can be so severe that the horse will often prefer to lay down. There is generally a noticeable increase in the pulse to the hooves, pain on compression of the sole and often the horse’s weight is shifted from one limb to the other.

If you have any suspicions that your horse may have foundered you should call the hospital for advice or seek veterinary attention.

How is founder/laminitis diagnosed?

The condition is relatively easy to define by visual examination. If the laminitis has already progressed to the chronic stage, x-rays may be needed to determine the degree of rotation.

Treatment protocols:

The most important initial treatment is to remove the suspected cause, which may involve restricting and controlling diet and the removal of any other relevant factors.

Ongoing veterinary treatment is most often via the use of anti-inflammatory medication, generally the nonsteroidal antiinflammatory drug (NSAID) phenylbutazone (bute). It is also vital that corrective farriery be employed as specialised trimming and shoeing is almost always necessary. In some cases no shoes and permanent soft footing may also help.

There are many products on the market claiming to assist the foundered horse. Some are useful, just as many not. Before administering any over-the-counter or DIY treatments or supplements please consult us for advice.

How can I prevent founder?

Founder is often a result of ‘killing with kindness.’ A well balanced, appropriate diet with restricted access to excessive feed intake will go a long way to avoid the condition. Commercial vitamin/mineral supplements and some hay should be fed even if strict feed restriction is necessary.

  • Regular, controlled exercise is important to keep the animal in good condition.
  • Monitor body condition and weight and avoid excess weight gain and obesity.
  • If the risk of founder occurring (or recurring) is high, consider adding a founder preventative agent to the feed, but only on veterinary advice.
  • Ensure the farrier visits regularly and you provide regular hoof care, including trimming, hoof dressing and feeding supplements to enhance healthy hoof growth.
  • Avoid access to unaccustomed pasture, hay, grain or commercial feed pellets and make sure your horse has no chance of ‘binge eating’ of hard feed. Any change in diet should be instituted gradually.

Greasy heel

Greasy heel, more commonly known as ‘mud fever’, is a painful infection caused by the same bacteria that causes rain scald. It usually affects horses with white feet but it can also occur in animals with dark feet and in endurance animals. In severe cases it can extend as far up the leg as the cannon.

Contrary to popular opinion, greasy heel is infectious so it’s very important to quarantine affected horses to prevent transfer to paddock mates. It is most common in damp, winter weather but the bacteria can be present in moist, poorly drained paddocks all year round.

Clinical signs:

The basic signs of greasy heel are probably familiar to all horse owners: red, crusty, cracked lesions with a greasy yellow weeping around the heels. These weeping sores are extremely painful and require prompt treatment, both to relieve the suffering of the horse and in an effort to prevent the spread of the disease to other horses.

Treatment protocols:

Initial treatment is aimed at reducing the pain and fighting the infection with antibiotic/cortisone creams. In very severe cases, a shot of penicillin may be required.

In all but the most severe cases ongoing treatment can be carried out by the owner. The method described below is a very effective treatment you can do at home.

Warning! Remember that greasy heel can be very painful. Your usually mild mannered horse may become a bucking bronco at the mere scent of anyone approaching those very sore heels. It’s best to have a ‘second in command’ to hold the horse for at least the first round of treatment to make sure it’s performed safely and effectively.

Be sure that you don’t become the agent of transmission. Before beginning treatment, wash your hands thoroughly, and always care for the ‘clean’ horses first, then deal with the infected animals.

Check all heels, starting with the dark feet. For affected heels clean off the grease with warm salty water, then carefully clip the hair over and around the lesions, following any cracks.

Very gently, massage Yellow Lotion (available at the hospital) on the area with a soft nail brush. Avoid causing any further abrasions as these can allow easy access for the bacteria. Don’t forget to clean in the deep cracks, and don’t be too concerned if they bleed, as the skin will initially be very raw.

If the heel is very sore and swollen follow the Yellow Lotion with some prescription antibiotic/cortisone cream.

This process must be repeated every day for four or five days. Once the affected areas look less raw and swollen use Cetrigen (available at the hospital) every morning and night to dry it out.

There are some paddocks rife with greasy heel bacteria. There are also some horses predisposed to this nasty infection. In these animals, a thick, zinc based cream such as Filtabac or Derisal can be applied to the susceptible areas each morning to try to prevent the return of the infection.

Important! Please, do not use any of the ‘natural’ or ‘home made’ treatments you may have heard about for the treatment greasy heel. There are dozens of these so-called ‘therapies’ and treatments, from metho to sump oil. None of them work, none of them will do anything to combat the infection, and many of them are downright bad for the horse.

Rain scald

Rain scald, or Dermatophilosis, is a skin condition in horses that most owners will be familiar with. It’s caused by a bacteria — Dermatophilus congolensis — which lives on the skin of ‘carrier’ horses. A carrier horse will be prone to developing rain scald, but may not always show clinical signs. They are however the source of infection for other horses.

Young horses, and senior horses with poor skin immunity, or animals introduced from a property with low contamination are more likely to develop acute or chronic rain scald.

For a horse to be infected there must not only be a carrier animal close by, moisture and skin trauma must also be present. When a carrier horse’s skin is soaked, such as occurs during rain, the bacteria release zoospores, the infective stage of the bacteria. Flies carry these zoospores from the carrier to a non-infected horse. Brushes and rugs may also be responsible for transmitting the zoospores.

If the skin of a non-infected horse is healthy and intact the bacteria cannot invade and cause rain scald. If the skin is damaged or broken, by fly bites, other skin diseases, or trauma, the bacteria are able to invade the skin and infect the animal.

Clinical signs:

The most commonly affected areas are the croup, loins, saddle area, neck and face, the areas of the body that take the brunt of rain. In severe cases the disease may become generalised over the entire body and limbs.

The visible signs of infected skin is a discharge which dries and causes the hair to become matted and crusted. The skin underneath these crusts is often pink, moist, and occasionally bleeding. A greenish-yellow pus will often accumulate on the under surface of the crust. Affected areas can be sore to touch.

Rain scald can also affect the limbs, with white, non-pigmented skin being more susceptible. When the legs are affected, they can become painful and swollen, causing lameness.

Severely affected horses may also develop a fever, be lethargic/depressed, have poor appetite, and swollen lymph nodes. For a horse to become this severely affected there are generally other contributing factors, such as an underlying disease that’s causing suppression of the immune system.

How is rain scald diagnosed?

The characteristic appearance of the condition makes diagnosis by visual examination quite straightforward. It can be confirmed by taking smears of the discharge from the lesions and testing them to establish the bacteria present. Acute lesions can be sampled and cultured.

Treatment protocols:

Treatment is straightforward, involving the application of antibacterial agents to the skin, usually in the form of washes. Chlorhexidine and povidone-iodine scrub are effective. Chlorhexidine is the active ingredient in Malaseb, a dog shampoo which is also very effective against rain scald in horses.

Wet the affected areas and apply the antibacterial scrub, gently massaging the affected areas to remove the scabs. Leave the wash on for ten minutes before thoroughly rinsing off. Repeat this every day for 5-7 days, and then twice a week until all the lesions have healed. Severely affected horses may benefit from systemic antibiotics, but the majority of cases do not need this treatment.

Although relatively easy to treat, unless the underlying cause of the skin trauma that allowed the infection in the first place is addressed, the condition may not resolve fully. It is important that, after treatment, any affected horse has a waterproof rug to prevent maceration of the skin and a recurrence of the disease. Affected horses should have their own rugs, saddle cloths, brushes, etc., which should not be shared with other horses.

Horses that have not previously had rain scald can get some protection by being rugged, although, if there is damage to the skin, they can still contract the disease. It is difficult, if not impossible, to prevent the flies that transmit the disease from landing on and biting the horse, allowing the bacteria to enter the skin. Rugging offers some protection, as does the regular application of fly repellents also helps, but it is virtually impossible to completely prevent flies from landing on your horse. Owners should be vigilant in checking for the signs of rain scald and treating it early.

Seedy Toe

Seedy Toe, sometimes called White Line Disease, is the necrosis of the sensitive laminae, usually on the anterior wall of the hoof. It can however affect all distal areas of the laminae, extending proximally towards the coronary band. The condition is caused by cracks in the white line, leading to infection and chronic laminitis with separation of the laminae in the area of the white line, usually at the toe. In some cases, anaerobic fungi have also been incriminated.

Clinical signs:

  • A horse with seedy toe will generally show slight to moderate lameness although this is not seen in every case.
  • A chalky, crumbly white line region, which can be easily gouged out with a foot knife, will be present on examination of the foot.
  • The hollow behind the adjacent hoof wall may show grit and other foreign matter.
  • Overt sepsis is occasionally present, with a discharging tract at the coronary band proximal to the region affected.

How is seedy toe diagnosed?

Diagnosis is confirmed by the characteristic clinical signs listed above. In some cases a lateral radiograph (x-ray) will be taken and will clearly show air and foreign matter wedged into the area between the wall and the pedal bone.

Treatment protocols:

Small areas of seedy toe can be pared back to healthy tissue with a hoof knife and, if the defect is spotlessly clean, it can be packed with synthetic filler. If not clean, the cavity will need to be tightly packed with a swab soaked in povidone-iodine or Metronidazole. The swab will need to be replaced daily.

More severe cases require the wall over the seedy portion to be completely removed and a small area of wall removed back to sound laminar tissue, either by a vet or a skilled farrier. The defect is not filled permanently until all risk of haemorrhage is past and the area is clean and infection free.

Some defects are very wide and as high as the coronet and these need to be extensively debrided, after a shoe with supportive side clips has been fitted to the foot to stabilise the distal margin. The shoe is then removed and the defects are debrided

Once all the necrotic and infected tissue has been removed the defect can be filled with an acrylic filler, the shoe reset and the horse rested in a stable. The animal should have only light walking for two weeks following treatment. The shoe should be regularly reset every four weeks and a close watch kept on the repair patch to make sure it remains firm and uninfected.

Strangles

Strangles is one of the most important equine diseases. It is a notifiable disease in Victoria under the Livestock Disease Control Act 1994. If your horse has Strangles you have a legal obligation to report it to the DPI within 7 days. If you suspect the disease is present please contact the hospital immediately for advice.

Strangles is a highly contagious infectious disease of the upper respiratory tract in horses. Caused by a bacterium called Streptococcus equi (S.equi), the disease affects horses, ponies and donkeys. Strangles is endemic and is easily recognised (see clinical signs). The disease tends to be more prevalent in the colder months and outbreaks may follow the introduction of new horses onto a property, or the mixing of groups of horses, at studs, shows or camps, or those on agistment properties with other horses. Horses of any age can become infected but strangles is generally more common in younger horses, whose clinical symptoms will be more severe.

The incubation period of the disease is usually about a week but it can be up to 3 weeks. Transmission is via both oral and nasal routes. Infection can also occur by direct contact between individual horses and/or through indirect contact through contaminated feed, water, bedding, stables, stable utensils and transport vehicles.

The S. equi bacteria is tough and can survive in pus and purulent discharges for many months and in nasal discharges for several weeks and for long periods in the environment. Bacterial shedding usually ends rapidly after clinical recovery and can be confirmed by negative culture of swabs from the back of the throat (nasopharyngeal swabs). Occasionally a horse will become a long-term carrier.

Clinical Signs:

The classic signs of Strangles are usually visible within 3 to 8 days of an animal becoming infected and include:

  • The rapid onset of a high temperature (39.5 to 40.5 C)
  • Loss of appetite
  • Discharge of yellow pus from the nose
  • Symmetrically enlarged glands of the head and neck that often form abscesses
  • Coughing and difficulty swallowing

A discharge of mucus from the nostrils is sometimes the only symptom seen. A carrier state without any obvious clinical signs is also possible. When abscesses burst a thick yellow pus is discharged. Abscesses rupture and drain within 2 weeks and drain and recovery is generally without incident. Symptoms can last for days or for months and, even after infected horses have recovered and appear healthy, affected horses will remain infectious for at least 4 weeks.

How is Strangles diagnosed?

We will confirm a suspected Strangles diagnosis by taking a swab from the back of the nasal cavity. Definitive diagnosis is based on a culture of S. equi, which may take several days to confirm a diagnosis, and a culture may fail to isolate S. equi if antibiotic treatment has already commenced.

Treatment protocols:

Any suspected case of Strangles must be treated immediately to try to control infection. Each case is assessed and treated individually but the primary treatment in less severe cases is isolation and good nursing care to keep the horse comfortable and quiet until the abscesses burst and the horse acquires a natural immunity. We may prescribe non-steroidal anti-inflammatory medication to reduce a high temperature and/or to assist with painful swellings. Once abscesses have burst they are treated by washing the site with warm water and antiseptic.

Affected horses must be kept isolated for 6 to 8 weeks to prevent the spread of the disease. Good biosecurity and hygiene must be observed when handling infected horses.

Complications are uncommon, and most horses recover uneventfully but some horses will contract Bastard Strangles, where bacteria spread through the body and cause abscesses in the lungs, liver, spleen, kidneys, brain, and lymph nodes. These cases can be fatal.

Purpura Haemorrhagica – where red spots form on skin and mucous membranes, caused by bleeding from smaller blood vessels — is also possible and is generally fatal. In these cases swelling of the limbs and head will be present.

A small number of horses can develop Chronic Carrier Status. These animals carry Strangles in the guttural pouch — an out-pouching of the back of the throat — for months after they have recovered from the disease. Carrier horses appear healthy but will shed bacteria in nasal discharges and are a source of infection for other susceptible horses.

Disease control:

Infection can be controlled through the isolation of infected horses until they are free from infection. The spread of the disease can be limited by the early detection of shedders amongst newly-affected horses and their contacts by taking three nasopharyngeal swabs over a two week period and culturing the swabs for S. equi. In the majority of cases a series of 3 negative swabs provides strong evidence of freedom from infection.

All infected horses, and their in-contacts, need to be under veterinary supervision while in strict isolation, with the highest possible standards of hygiene. Horses should not enter an affected stud unless they are vaccinated and can be kept in strict isolation from all sources of infection.

Disease prevention:

A vaccine is available for Strangles. While vaccination will not always prevent disease in individual horses, it can assist control by reducing the spread of disease in an outbreak and limiting the severity and duration of the disease in individual animals. An initial course of 3 injections two weeks apart is given, followed by at least annual booster vaccinations. We strongly recommend that all horses, but particularly those travelling to shows, events or mixing with other horses, are vaccinated. Please call the hospital for further information.

Reporting strangles:

Under the Livestock Disease Control Act 1994, a person must report Strangles if they know or have reason to suspect that strangles is present in horses or horse products. There is no regulatory action if strangles is detected on your property. Properties are not placed under quarantine, bio-security measures are put in place to prevent further spread.

Notification can be made by telephone to the District Veterinarian or Animal Health officer at your local Department of Primary Industries office or to the Disease Watch Hotline on 1800 675 888. Or you can call the hospital and we will advise the authorities.

Tetanus

A bacterial disease, tetanus can affect most animals but horses are particularly susceptible because of the environment they live in and their tendency to incur injuries. Tetanus is caused by the bacterium Clostridium tetanii which is found in soil and droppings. It’s found everywhere and survives in the environment for long periods.

The bacterium enters the body through wounds, particularly puncture wounds, and especially if the wound is dirty. Puncture wounds on the sole of the horse’s foot are common sites of infection.  Animals can also be infected via the intestines after eating contaminated soil or droppings, or through gastric or intestinal ulcers.

In foals, infection can be transmitted via the umbilicus (navel).  The tetanus bacteria are classified as ‘anaerobic’ bacteria, meaning they do not need oxygen to survive and multiply rapidly in the damaged tissues at the site of the injury. The bacterium produce a potent neurotoxin (tetanus toxin) which causes the classical signs of tetanus.

Clinical signs:

Tetanus toxin attacks the nerves controlling the muscles of the body, causeing progressively worsening muscular stiffness and spasm. An affected horse will become stiff and have difficulty moving and eating.

The third eyelid (membrana nictitans, a membrane which can be easily seen at the inner corner of the eye) will begin to protrude across the eye, particularly if the horse is startled.  The tail is often held out straight and the horse develops an anxious facial expression because of facial muscle spasm. Any stimulus such as a loud noise, bright light or touch can exacerbate the signs.  The horse may sweat.  In advanced cases the horse will collapse, showing spasms, convulsions. Death will usually result from respiratory failure.

Treatment protocols:

Sadly, we cannot do much for a horse with infected with tetanus bacteria. Most cases of tetanus will result in the death of the affected animal.  Diagnosed early enough, treatment is targeted at destroying the bacteria so that no more toxin is produced, and reducing the effects of the toxin that has already been produced.  Large doses of antibiotics, usually penicillin, are used in conjunction with Tetanus Antitoxin injected usually intravenously and intramuscularly.

If the horse is able to eat, food should be offered at a height where it can easily reach it.  In severe cases, slinging may be necessary.  Intravenous fluids and/or catheterization of the bladder may also be necessary.  Unfortunately, the chances of recovery are extremely poor if such measures are necessary and euthanasia on humane grounds is usually more appropriate.

Can tetanus be prevented?

The good news is that Tetanus is an easily preventable disease, with a readily available vaccine. Vaccination with ‘tetanus toxoid’ should be used for all horses and ponies. The initial course consists of two injections given approximately four to six weeks apart, with booster shots at two-year intervals.

Foals cannot respond to vaccine given before approximately four months of age but they should start a course of vaccine very soon after reaching this age.  As protection before this age foals usually receive tetanus antitoxin soon after birth. They also receive some protection via colostrum (first milk) if the mare is well vaccinated.

Good first aid can also help prevent tetanus. Wounds should be cleaned as soon as they occur and steps should be taken to encourage drainage of deeper injuries. Paddocks, stables and stable yards should be kept safe, clean and clear of dangerous items such as old tractor parts, corrugated iron sheets and building materials that may cause injury.

  • Avian Heart Disease
  • Behavioural enrichment
  • Bumblefoot
  • Cannibalism in backyard poultry flocks
  • Coccidiosis
  • Coccidiosis in poultry
  • Egg bind in poultry
  • Feeding backyard poultry
  • Polyomavirus (PVD)
  • Poultry: Moulting
  • Scaly Mite

Just like humans, pet birds, particularly the long-lived species, can suffer from heart disease (atherosclerosis), falling victim to hardening of the arteries, high cholesterol and heart attacks. Heart disease is fairly common in caged birds, affecting many species.

Birds known to be prone to developing atherosclerosis include waterfowl, pigeons and doves, chickens, turkeys, and related birds), and Psittaciformes (parrots). Psittacine birds most commonly affected include Amazona species, especially blue fronted amazons (Amazona aestiva), African Grey parrots (Psittacus erithacus), cockatoos, Monk or Quaker parrots (Myiopsitta monarchus), and macaws.

The causes of atherosclerosis in pet birds may be genetic, or a factor of age, but avian heart disease is now believed to be primarily an inflammatory disease, resulting after a long period of chronic inflammation. Similarly to the development of heart disease in people, diet plays a large part. A ‘perch potato’ is not much different to a ‘couch potato.’ And eating ‘junk food is just as bad for pet birds as it is for people. The type of dietary fat eaten most likely affects the development of atherosclerosis more than the total amount of fat consumed.

Unlike wild birds which must constantly work to feed themselves, caged birds are frequently compromised by lack of exercise, nutritional deficiencies and unnatural climate conditions. Combined with the bird’s naturally high blood pressure, these factors make the risks of heart disease more significant. The avian heart is much like our own heart, having four chambers and being strongly muscled to pump blood to the rest of the bird’s body.

Clinical signs:

Signs of heart disease in birds are often nonspecific and can also be accompanied by other diseases which disguise the clinical picture. The most frequent signs seen are…

  • Weakness
  • Lethargy
  • A bluish discoloration of the skin around the eyes, particularly in African Grey parrots
  • Abdominal distension
  • Difficulty breathing
  • Exercise intolerance

How is avian heart disease diagnosed?

Diagnosing heart disease in birds is considerably more complicated than in people. There has been little clinical research into avian heart disease, and normal reference values for most tests do not exist. Diagnostic techniques are also compromised by the size of the patient, the bird’s naturally fast heart rate, and the degree of stress placed each test may put on the patient.

Diagnosis is most often based on examination, but if the patient is in a stable condition radiographs can be used to visualise the heart and evaluate its position, size, and shape.

Treatment protocols:

Few studies of avian heart disease exist and many of the drugs we routinely use in mammals have not been tested in caged and aviary birds. Drugs that might work well in one bird species may not necessarily work well in another. The end result is that heart disease has a poor prognosis for affected birds. The disease is often not recognised until the heart fails or the heart is showing advanced disease changes.

However, just as it is with people, prevention of heart disease is far better than treatment after the fact. Factors that have been linked to the development of atherosclerosis in birds include long-term diets high in fat or cholesterol and a lack of exercise. Bird owners should reduce the fat in your bird’s diet, avoid fatty foods that can cause cholesterol plaques in your bird’s arteries and make sure your bird gets plenty of exercise. Proper diet can go a very long way to reducing or preventing heart disease in your pet bird. Our staff at the hospital are happy to advise on the optimum diet for your birds.

Regular exercise is absolutely vital. You must get your parrot out of its cage and moving regularly and frequently. And make sure your bird has an annual checkup at the hospital so any diseases can be caught early.

Behavioural enrichment

Bringing a bird into your home should be a happy and rewarding experience. With many of our native parrots enjoying long lives — some can live for 80 to 100 years — a well cared for bird can be a companion and keep you entertained for a lifetime. But having a bird as part of your family is also a big responsibility. Any pet bird will be entirely dependent upon you — for its food, its water, shelter and its continuing good health. Birds on a poor diet and in a non-stimulating environment will become depressed, miserable and ill, dramatically reducing their life span.

Feeding

A balanced diet is the most important part of a healthy bird’s life. The most common problems seen at the hospital include obesity, egg binding and vitamin A deficiency.

Many pet parrots eat nothing but seed. It’s easy for the owner to buy a packet of seed mix from the supermarket, and the birds might love it, seeds are often interesting to open, and also high in fat and therefore very tasty. But dry, packaged seeds are also low in all vitamins, especially vitamin A. Some seed mixtures do contain added vitamins and minerals but this is rarely an improvement as most birds simply eat around these ‘vitamin balls.’

Many birds will also pick out just one or two preferred types of seeds from the whole mix and refuse to eat any of the other food or seed. Most commonly selected are sunflower seeds or oats — which are like chocolates for a bird, and equally unhealthy in quantity. To provide a balanced diet many other foods need to be offered in addition to seeds. Varied fresh foods should be given daily. Vegetables, especially green vegetables, are extremely important. Good examples include spinach, silver beet, endives, parsley, celery and cabbage. Fresh grasses, milk thistle, dandelion and fresh fruit are also great.

Environment

Birds need a full spectrum of light to remain healthy and happy. Sunlight through a closed window is inadequate because vitamin D is absorbed only from natural sunlight. Bird owners need to allow their feathered friends regular time outdoors, on the deck or the patio, where they can enjoy the sun. Conversely, birds also need at least 10-12 hours in total darkness each night. If your bird is kept in a room that you use at night then a blackout cover over their cage is needed, but it’s better that your bird sleeps in a room that’s not subjected to high levels of noise during the night. For many species of bird the period of darkness should be varied during the year to mimic the natural seasons.

A lot of birds enjoy baths or showers, and even enjoy going into the shower with their owners, but avoid using soaps if your bird is with you. Alternatively, spray lightly with a spray bottle or a hose on a fine mist spray. Bird baths can be provided, but it is best not to leave them in cages all the time as birds will defecate in them.

Smoking is extremely hazardous to birds. Their lungs, in combination with their air sacs, are an extremely efficient breathing system. The skin can also become irritated by the smoke, which is considered one of the causes in self-mutilation.

Cages

Cages should be a home to protect birds, not a prison. A cage should be rectangular, and deep enough for two birds to be able to fully extend their wings. Birds are not helicopters so tall cages are not appropriate. A bird’s cage may be its home but that should not mean the bird should never ‘go outside.’ Birds should spend as much time as possible outside of their cage and moving in order to have healthy exercise. Without these ‘outings’ your bird will become overweight and depressed. If you are concerned about catching the bird or if it is reluctant, simply dim the lights and the bird will be much easier to catch.

Poor perches commonly lead to sores on a bird’s feet and sometimes potentially serious issues such as bumblefoot. A good perch is made of natural branches of varying sizes, never smooth timber dowelling or plastic.

Food and water bowls should be placed in cages where the bird will not defecate in them. Parasitic and bacterial infections are not going to improve if the bird constantly eats and drinks their own faeces. It’s also best not to place any food or materials — sandpaper, grit or food — on the floor of the cage that will encourage the bird to eat there. Instead, use a grill to allow faeces to fall through to the cage floor, or line the floor with plain paper.

Avoid using metal toys or plastic coated bag ties in the cage as these can lead to heavy metal poisonings. Heavy metal poisoning is an extremely common problem in cage birds, many of whom are continually exposed to metals in their cage or environment. Lead, zinc and copper are the most common metals involved in poisoning. They are found in galvanised wire, paint, copper wires, metal ties, rusty metal toys and backs of mirrors, etc.

Cages and aviaries should be made of stainless steel, powder baked, or polymer-covered wire. If you cannot provide a cage of these materials, scrub galvanised wire with vinegar and a wire brush, then rinse the vinegar off and repeat. This will help minimise zinc toxicity but will not eliminate it. Weathering the wire (leaving it outside to the elements) will not detoxify the metal either.

Most parrots live in large flocks, not small cages. Birds, unlike dogs and cats, become bonded to one single other bird, or human, as a mate. A bird that is always calling, talking, or singing is in fact demanding the attention it craves. It is very rare for a bird to be happy on its own. Many bird diseases and various syndromes are related to stress, frustration and boredom. Television and radio may be occasional comforts but they are not long term substitutes for the company of their own species. If you are not in a position to provide pretty constant companionship for your pet bird, please consider keeping more than one bird. They’ll both be healthier, happier — and twice the entertainment value.

Bumblefoot

Bumblefoot is the common term used for a bacterial infection — ulcerative pododermatitis — causing  inflammatory or degenerative reaction on the weight-bearing surface of the feet of birds (and rodents). The infection is common and occurs in all species of birds, but is primarily a problem of cage birds. Obese and inactive birds are particularly prone to developing bumblefoot. Pet birds typically suffer from bumblefoot due to an underlying problem with some aspect of their husbandry and environment.

Any bird species can be affected but the condition is most common in budgies, cockatiels, galahs and ducks. Birds of prey (raptors) are also prone to the condition.

Clinical signs:

The symptoms of bumblefoot may not be initially apparent. Lameness is usually the first sign to be noticed – the bird may appear unwilling to land, stand, or grasp normally with one or both of its feet. Initially there may simply be a loss of the normal scale on the feet and the skin may be seen to be red and thin.

The resulting cracked skin is susceptible to bacterial colonisation, which can lead to abscess formation. In more severe cases, the foot is swollen and there are plugs of necrotic (dead) tissue on the weight bearing foot surface. Ulcers may form on the pads of the feet and, in the most severe cases, the bones of the feet will become infected Once infection spreads to the bones and joints, birds become severely lame and the prognosis is poor. Surgery is usually needed if there is to be any chance of recovery.

How is bumblefoot diagnosed?

A history of lameness, combined with obvious redness and swelling of the feet are clear indicators of bumblefoot. X-rays of the feet may be required to determine the extent of the infection. X-rays will indicate if the infection has spread to the bones of the feet.

We may also take a swab from the site of the infection and for a culture and sensitivity test in our pathology lab. This determines which strain of bacteria are present and helps us choose the most appropriate antibiotic to treat the bacteria.

Treatment protocols:

Veterinary treatment for relatively mild cases showing redness of the skin and minimal swelling is via an antibiotic ointment applied at least twice a day to the affected feet. Oral antibiotics may also be used as a systemic infection control.

Ball bandages may be used to relieve pressure on the foot and to allow the bird to stand comfortably – bandages need to be changed regularly.

Severe cases may need surgery to remove plugs of inspissated pus and dead tissue.

Very mild cases can clear up within a few days, but severe cases of bumblefoot can take weeks to months to resolve. The bird(s) may also be left with permanent damage to the feet so the sooner treatment is initiated, the better the prognosis for recovery.

As the primary cause of bumblefoot is most often environmental factors, there is a great deal the bird owner can do to minimise the possibility of your bird(s) contracting the infection. Good sanitation, a balanced diet and appropriate perches will help reduce the incidence of the disease.

Provide different perch sizes and types, as well as of a variety of materials. Branches of different sizes from native trees make good perches because the bird’s feet are not constantly held in the same position. Perches of uniform size and made of improper materials such as smooth dowelling or hard plastic, or perches covered with sandpaper are detrimental to the birds’ feet and should not be used. You can also soften perches by wrapping bandages or strips of cloth around them, but these should be changed regularly and frequently.

Ensure good cage hygiene. Perches that are infrequently cleaned can accumulate bacteria and spread to a foot that has already been damaged and lost its protective scales.

Check the bird’s diet. Poor nutrition – fatty foods, a dietary deficiency of vitamin A, and diets high in cholesterol and low in calcium are all associated with bumblefoot. Improve the diet by moving your birds onto a high quality pellet or crumble food and including, more dark green vegetables for increased vitamin A. Our staff are happy to advise on suitable balanced diets. For ducks, ensure they have access to grassed areas and a clean deep wading pool.

Obesity and inactivity are a major contributor to bumblefoot so ensure your bids are not overfed, or fed fatty foods, and give them plenty of exercise outside their cage. Get those wings and feet working.

Birds that have concurrent illnesses are more susceptible to disease and more likely to contract bumblefoot due to a compromised immune function. Have your pet bird checked at the hospital at least annually to pick up underlying health issues before they become severe. Check your bird’s feet at home at least once every two weeks.

Cannibalism in backyard poultry flocks

Your backyard chooks won’t truss up an unpopular flock member and pop them in the pot, but they may exhibit cannibalistic behaviour by pecking and tearing at their unfortunate flock mates.

Outbreaks of cannibalism happen mostly in larger flocks but it can occur in small groups and in even the best of housing systems, including cages, floor pens, aviaries and in outdoor, free-range flocks. It happens in all types of poultry, including chickens, ducks, turkeys, quail and pheasants. Birds will mostly show cannibalistic behaviour when they are crowded or when feed is restricted.

The act of pecking at each other is natural amongst birds as they establish dominance — the ‘pecking order’ — and sort out their respective positions in the flock. Cannibalism is not dominance behaviour, however, and may occur long after dominance relationships have been established. Cannibalism may begin with feather pecking and is usually directed toward the body, toes, tail and especially the vent area. Cannibalism differs from dominance in that it actually causes physical harm, sometimes even death. Preventing cannibalism amongst your birds is much easier than treatment.

Cannibalism is believed to be caused by a combination of genetic and environmental factors and outbreaks can occur in the best-managed flock. Some of the more common causes include:

  • The propensity of different breeds to feather peck.
  • Overly large flocks in crowded conditions.
  • Insufficient feed, water, perch or nesting space.
  • Dietary deficiencies, particularly vitamin deficiency or a lack of salt.
  • Bright lighting.
  • Injured birds left exposed in the flock.
  • Keeping different ages or colours together.
  • Injuries that bleed as a result of enclosures in poor repair or from fighting among newly mixed birds.
  • Diseased birds or individuals with low body weight.
  • Barren environments that restrict or limit natural behaviour, especially normal nesting and foraging behavior.
  • Sudden changes in diet, e.g., to a less-preferred food.

Prevention:

Because cannibalism is a learned behaviour that, once established, will spread throughout a flock there is no ‘cure’ as such. Apart from the mechanical debeaking carried out in some commercial flocks prevention is the only way to reduce or eliminate the behaviour. The first step is to select a genetic stock (breed) that is not prone to cannibalism.

Other than genetic factors, limiting the size of the flock and giving your birds the most natural environment and best care possible is the most effective way to prevent cannibalism. This includes providing an adequate diet, safe housing and environmental enrichment. In caged or permanently housed flocks careful lighting control will reduce incidences of cannibalism.

Providing a diet that meets all the nutrient needs for the age and the type of flock is critical. Cannibalism has been linked to deficiencies in protein, sodium and phosphorus. As chicks grow their protein requirements change and need to be adjusted based on a recommended feeding schedule.

Ensuring adequate feeder space for all birds to eat simultaneously will help avoid having underweight birds, who are invariably at the bottom of the pecking order and are therefore the most likely victims of cannibalism.

Light intensity and the period of light and dark can influence cannibalistic behaviour. In commercial flocks photostimulation programmes are often used to encourage laying but these should be delayed until the birds are physically mature, after 20 weeks of age. Dim lights are often used to prevent cannibalism. If reduced lighting is used, 5-10 lux (about 0.5 to 1.0 foot candles, just enough to read a newspaper) from incandescent or fluorescent bulbs is recommended.

Chickens are attracted to blood and any injury that bleeds can lead to an outbreak of cannibalism as flock mates subsequently peck at the injury, encouraging others to follow suit. It is important to keep enclosures in good repair to prevent injury from sharp wire or from a bird becoming trapped in mesh and injuring itself in its struggle to get free.

Any bird that shows cannibalistic tendencies should be culled, and injured birds, victims of cannibalism and dead birds should be immediately removed from the flock.

Environmental enrichment is the most effective way to prevent cannibalism. The best way to provide an environment in which birds are less prone to pecking other birds is to give your flock the opportunity to free range in an environment that mimics as closely as possible their natural foraging behaviour. That means giving them sufficient space to forage about for much of the day amongst trees and plants with dense ground cover and insect-rich mulches. A chicken’s natural behaviour includes spending a large portion of the day searching for food. In an environment that prevents the expression of this normal foraging behavior, such as an enclosed house or shed, the instinct to peck can be redirected toward flock mates and may lead to cannibalism.

Failing a natural free range environment, you can try putting objects in their pen or henhouse that will provide a greater variety of behavioural outlets. For hens, the kind of environmental enrichment that reduces the incidence of cannibalism include nest boxes, perches and other objects that might redirect pecking away from flock mates.

The cloaca is highly attractive to hens prone to pecking and some outbreaks of cannibalism begin during oviposition. A dark nest box provides a safe place for egg laying and prevents exposure of the everted cloaca during oviposition to potentially cannibalistic flock mates. Hens can also escape aggressive vent pecking by being able to perch high enough off the ground that they cannot be reached by hens on the floor. Pullets are more likely to use perches as a refuge if they are reared with them from an early age.

Pullets reared on a litter type that is attractive for scratching and pecking are less likely to develop cannibalistic behaviour. It is important to provide foraging opportunities such as straw, green leafy vegetables or grass clippings, or to feed small grains in deep litter. A mash diet, rather than pelleted feed, may also help prevent outbreaks of cannibalism because chickens sift through the variety of ground particles and take longer to consume their feed. One popular trick is to hang a couple of cabbages in the pen, which encourages the birds to peck at the leaves rather than each other.

Control:

Carefully selecting a breed that is docile, together with the management practices described above, should prevent most cannibalism. However, if an outbreak does occur, it is essential to stop the behaviour before it spreads. It’s difficult to stop a substantial outbreak altogether but some options include:

  • Separate any birds seen to severely peck at others, especially if directed at visible injuries or the vents of other birds.
  • Remove victims of cannibalism and care for them separately.
  • Add enrichments, especially foraging-related devices.
  • Add additional feed and water space.
  • Add nest boxes and perches.
  • Use an antibiotic wound spray on the everted cloaca of hens with vent trauma (in small flock situations).

Beak Trimming:

Beak trimming, or debeaking, is the practice of cutting off a small portion of the bird’s upper beak, blunting the sharp point and reducing the damage a bird can do by pecking. It is generally only carried out in larger commercial flocks of either caged or barn-reared hens.

Debeaking is associated with both acute and chronic pain and should never be undertaken indiscriminately, or by an inexperienced poultry keeper. Neuromas — tangles of severed nerve endings in the amputated beak stump — can result from beak trimming carried out incorrectly or with inadequate tools.

The avian beak, like the human hand, is very sensitive to touch, temperature and pressure. Chickens use their beaks not just to gather food but to explore and manipulate objects in their environment, to preen, dust bathe and in nesting and social interactions. Beak trimming is not only painful, it deprives the bird of important sensory information.

Like many large scale farming practices it is something that makes any animal lover, including the farmers themselves, uncomfortable. There is little doubt that, like battery cages, debeaking will eventually be banned in Australia (it already is in Europe). Genetic research and selection of more docile breeds less likely to practice cannibalism will in the future have to replace the practice of debeaking.

In the meantime however, until alternatives to beak trimming are developed the commercial poultry farmer must balance the potential painfulness of the practice against the likelihood of a cannibalism outbreak.

If the decision is to beak trim, the procedure should be done between seven and 10 days of age and only by properly trained personnel, preferably a veterinarian, under controlled conditions. Beak trimming errors can add additional pain and suffering to an already stressful procedure, and may fail to prevent cannibalism. Even for larger flocks, there are management and environmental alternatives to debeaking and we are happy to assist commercial operators to make the transition.

Coccidiosis

Coccidiosis is an infection of the intestinal epithelium caused by microscopic protozoan parasites. It is characterised by an invasion of the intestinal wall by the Coccidia parasite (Coccidia isospora and C. elmeria) which then goes through various stages of growth and multiplication, during which damage occurs to the mucosal and submucosal tissues of the intestine.

Depending on the Coccidia strain involved, the disease can range from subclinical infections to infections that include serious intestinal problems with severe lesions in the gastrointestinal tract, to diarrhoea, resulting in high mortality and/or a negative impact on production in commercial poultry flocks.

All domestic animal species can be infected, as can wild populations, but coccidiosis is especially prevalent when birds or mammals are grouped together. Coccidia can be seen in any species of bird but are more commonly seen in budgerigars, pigeons, doves, chickens, turkeys and geese. They are less frequently seen in canaries, finches and lorikeets. Coccidia oocysts (eggs) are passed in the bird’s droppings and are often ingested through food or soil contaminated with infected droppings. The hard-shelled oocysts are highly resistant to environmental conditions and disinfectants, making coccidiosis control difficult.

Clinical signs:

Symptoms of coccidiosis infection may include any or all of the following signs…

  • Diarrhoea, with or without blood
  • Weight loss and depression
  • Lack of appetite or cessation of eating
  • Poor growth and/or death in young or unwell birds

Some birds can carry the organisms and may not show signs of illness.

How is coccidiosis diagnosed?

Examination of fresh, warm faecal smears of the droppings will usually display the eggs (oocysts). Eggs do not show up in every faecal smear and flotation of a dropping sample in the laboratory may be necessary to find the coccidia.

Treatment protocols:

There are a number of different options for the control of coccidiosis. One of the more efficient treatments is Baycox (Toltrazuril) added to drinking water for two days. A repeat treatment may be necessary. Cage should be cleaned thoroughly daily and then disinfected. Treatment of secondary infections caused by bacteria may also be required.

The possibility of coccidiosis can be minimised or prevented by good hygiene, including frequent cleaning of the aviary or cage. New birds should also be quarantined until they have been examined for coccidia and introduced to flocks only after they have been given a clean bill of health. Cage bird owners should also have their bird’s regularly checked for coccidia.

Coccidiosis in poultry

Coccidiosis is an infection of the intestinal epithelium caused by microscopic protozoan parasites. It is characterised by an invasion of the intestinal wall by the Coccidia parasite (Coccidia isospora and C. elmeria) which then goes through various stages of growth and multiplication, during which damage occurs to the mucosal and submucosal tissues of the intestine.

Depending on the Coccidia strain involved, the disease can range from subclinical infections to infections that include serious intestinal problems with severe lesions in the gastrointestinal tract, to diarrhoea, resulting in high mortality and/or a negative impact on production in commercial poultry flocks.

All domestic animal species can be infected, as can wild populations, but Coccidiosis is especially prevalent when birds or mammals are grouped together. Coccidia can be seen in any species of bird but in poultry is more commonly seen in chickens, turkeys and geese. It’s also found in budgerigars, pigeons and doves in cage birds. They are less frequently seen in canaries, finches and lorikeets.

Coccidia oocysts (eggs) are passed in the bird’s droppings and are often ingested through food or soil contaminated with infected droppings. The hard-shelled oocysts are highly resistant to environmental conditions and disinfectants, making coccidiosis control difficult.

Clinical signs:

Symptoms of coccidiosis infection may include any or all of the following signs…

  • Diarrhoea, with or without blood
  • Weight loss and depression
  • Lack of appetite or cessation of eating
  • Poor growth and/or death in young or unwell birds

Some birds can carry the organisms and may not show signs of illness.

How is coccidiosis diagnosed?

Examination of fresh, warm faecal smears of the droppings will usually display the eggs (oocysts). Eggs do not show up in every faecal smear and flotation of a dropping sample in the laboratory may be necessary to find the coccidia.

Treatment protocols:

There are a number of different options for the control of coccidiosis. One of the more efficient treatments is Baycox (Toltrazuril) added to drinking water for two days. A repeat treatment may be necessary. Hen housing and the flock’s environment should be cleaned thoroughly daily and then disinfected. Treatment of secondary infections caused by bacteria may also be required.

The possibility of coccidiosis can be minimised or prevented by good hygiene, including frequent cleaning of the hen house. New birds should also be quarantined until they have been examined for coccidia and introduced to flocks only after they have been given a clean bill of health. Cage bird owners should also have their birds regularly checked for coccidia.

Egg bind in poultry

A hen is said to be ‘egg bound’ (or to suffer from ‘egg bind’) when a particularly large egg becomes lodged in the oviduct and the bird cannot complete the process of laying.

Egg bind is a fairly common condition that can affect hens of any age but is most often seen in young birds laying their first eggs. It’s a serious condition that puts the hen in danger, and is invariably very uncomfortable for the bird. If you are an inexperienced poultry keeper and suspect egg bind you should contact the hospital for advice.

Clinical signs:

  • The hen’s abdomen will be swollen and, often, the shape of the egg can be felt by gentle exploration, but this diagnosis should be treated with caution. See below.
  • Egg bound hens will usually keep returning to the nest box and straining to lay, without success.
  • They will often show signs of distress around the yard.
  • Occasionally, another hen in the flock will ‘raise the alarm’ with loud squawking around the affected hen.

How is egg bind diagnosed?

The most definitive way to diagnose an egg bound bird is with an abdominal radiograph (x-ray). We appreciate that an x-ray is often neither practical nor economic for a lowly hen but backyard poultry keepers should keep in mind that a swollen and distended abdomen — the sign most people immediately assume to be egg bind — can be caused by other, more serious conditions.

If an x-ray reveals a properly formed and calcified egg we know what we are dealing with. If, however, the radiograph does not show a distinct egg it could mean you are faced with a ‘shell-less’ egg or an ‘internal layer.’ Treatment for both of these is far more complex and requires a veterinarian.

An x-ray may also reveal a fluid filled abdomen, which can be a sign of peritonitis. This might be caused by egg material that has never been passed, or by unrelated issues. In either case immediate treatment with broad-spectrum antibiotics is required.

In most cases however, egg bind will be diagnosed by sight and by feel, using a finger very gently inserted into the vent to establish by touch if there is actually an egg lodged in the oviduct. If you opt to do this yourself rather than call a vet, please be aware that cleanliness is vital. It is critical that you do not introduce bacteria into the oviduct. Thoroughly wash and disinfect your hands first. It is also often better that someone with small hands does this task. There is not a lot of room and the muscles of the oviduct can be easily damaged.

Treatment protocols:

One way or another, the recalcitrant egg must come out. There are a range of treatment options veterinarians can use, from physical manipulation to draining the contents of the egg and removing the shell. We can also place an egg bound hen in an incubator/humidicrib in a controlled environment with increased humidity that will assist the hen in passing the egg naturally.

In severe cases the only solution may be surgical removal under a general anaesthetic, sometimes resulting in a complete hysterectomy.

In practical terms that is rarely done and in fact a veterinarian is not often called in for cases of egg bind. Some vets will not even attend a hen because the cost for even simple treatments can often outweigh the value of the bird. At Alpine Animal Doctors we understand that even a lowly chook can be precious to its owner and we are happy to treat any creature if asked. But we also understand that simple economics will often mean the small scale poultry keeper will prefer to attempt a DIY treatment.

In mild cases treating the bird at home can be successful providing you proceed carefully and pay attention to strict hygiene.

In cases where the egg is just that little too large to pass easily through the oviduct you may only need to apply a lubricant such as warm olive oil or Vaseline to the vent. Try to get some lubricant just inside the cloaca but be careful not to tear the delicate tissues. If you can actually see the end of the egg through the vent, lubricate that too. Push very gently on the abdomen and the egg might come come out without too much of a problem.

After successful removal ensure that your hens have adequate protein, calcium, selenium, and vitamins A and E.  Poor condition is the primary cause of egg binding and it can largely be prevented with good diet, additional vitamins and plenty of free range in the sunshine.

For simple cases of egg binding caused by a hen’s first egg, this home remedy may be enough to save many of the birds that are lost each year at sexual maturity. If this method fails the problem is almost certainly more than minor and the egg will probably need to be drained and broken to be removed. The chances of breaking the shell and causing internal damage are quite high and we do not recommend that this be done at home unless you are a very experienced poultry keeper. Please call us for advice.

After treatment the affected bird should be kept somewhere quiet, away from other members of the flock. There will likely have been some muscle damage to the oviduct, which may never recover fully, or cause prolapse where part of the oviduct ‘hangs out’ and needs to be tucked back in again.

Feeding backyard poultry

A well fed chook is a happy chook. And a happy chook is a healthy chook. Healthy, happy chooks lay more eggs. Understanding the role of good nutrition and ensuring your little backyard flock is fed a balanced diet is the single most important thing you can do to keep your hens healthy. A good diet, together with the provision of a natural environment, will go a very long way to avoiding poultry health problems entirely.

Back in the day, about fifty years ago, when just about every household kept a few chooks, everyone knew exactly how to feed them. Or thought they did. Every home kept a smelly container under the sink — the chook bucket — into which all that day’s food scraps were thrown, from raw potato peelings up to and including the tea leaves.

Once a day the malodorous contents would be tossed to the hens. Maybe a bit of pollard mixed with water or, sometimes, skim milk, might be added. If they were lucky the hens would dine a la carte, from a trough. Mostly, people just chucked the sloppy mess on the ground and left the birds to fend for themselves. In the evening the flock would fight over a few handfuls of wheat scattered in the run. The really lucky ones, those that lived out of town, were able to get out of the henhouse and forage for a bit extra during the day.

There were almost no bags of balanced mash or pellet feeds available back then. Even if there were most people would have disdained their use, and the cost. It all sounds a bit inadequate, and it was, yet many people still look back on those days convinced that the little flock of hens they grew up feeding in this hit and miss manner were healthier, happier and laid tastier eggs than today’s pampered backyard chooks.

The reality is that they weren’t, and they didn’t. Lay tastier eggs that is. Backyard poultry were frequently underfed, suffered from dietary deficiencies and all the health problems that go with them.They were usually scrawny meat birds that laid fewer, smaller, eggs and had much shorter lives. Chooks were cheap and people didn’t expect too much from them.

There’s nothing wrong with feeding scraps to your chooks, they can play a valuable role as an addition to a balanced diet, but not as a sole source of food. And no raw potato peelings or tea leaves!

Things are different today. Not only do we have a far better understanding of the importance of diet and environment in promoting good animal health, the backyard poultry keeper has a wide range of affordable proprietary feeds available to them — pre-mixed mashes, crumbles and pelleted feeds, most containing the added vitamins and minerals needed to keep a chook well fed and healthy. And of course we still have the all important grains, including the ever dependable wheat.

Mash is basically whole grains crushed and chopped up small. Pellets are mash in a compressed ‘tablet’ form. Pellets are convenient for you but the birds eat them very quickly, which is undesirable for a number of reasons, and they can take a while to digest.

Mash is more flexible. You can feed it dry or mix it with water, milk or boiled up house scraps. It takes longer for the hens to eat so has a certain entertainment value. That extra time also comes a little closer to the bird’s natural feeding behaviour, which can help avoid cannibalism in your flock.

Most of these ‘ready-to-eat’ feeds are very good, although some are better than others. It’s generally a case of you get what you pay for. For those concerned about the feeds which add hormones to the mix, or are produced using chemically sprayed crops there are organic and ‘natural’ feeds available with few or no additives. But don’t be too concerned about added protein or vitamins and minerals such as sodium and phosphorous. Most backyard flocks need these ‘extras’ to stay healthy.

If you opt to feed a proprietary mix as your sole food source the first thing to decide is just how you are going to serve dinner. Providing you have a vermin- and bird-proof house and run, a hopper is the easiest way to provide a permanent, on-demand supply of dry food.

There are various types of hoppers and ad lib feeders available. The best let the food flow readily into the feed tray without it overflowing onto the floor. But remember, if your fowls are allowed out of their house during the day you have to be able to close off the feeder hopper to prevent birds, mice and other pests pigging out while the doors are open. Shutting the doors would mean the hens can’t get access to their nests to lay, and they’ll relish any excuse to lay away.

To foil these opportunistic pests there are feeder designs that stay closed until triggered by a fowl’s weight on a platform, which raises the lid to allow access to the food.

Whatever design of feeder you choose make sure the chooks can’t perch on top of or above it. Not only will they foul the feed tray but you will have to clean the lid of chook poo every time you need to fill it. A steeply sloping cover solves the problem.

If you don’t have a bird-proof house then the feed, whether dry pellets or moistened mash, should be fed in troughs early in the morning. Ideally, you would repeat this several times a day, removing the troughs when the flock has finished eating. Make sure all your fowls have had enough to eat before you remove the leftovers. Remember the chook pecking order — those at the bottom have to wait until the dominant members of the flock are finished before they can start. If there’s anything left for them. It’s up to you to ensure that these weaker, more timid, members of the flock get enough food.

Simple, V-shaped troughs are more than adequate, but put a couple of small drain holes in the bottom — just in case it rains.

How much feed does a hen need? There are no hard and fast rules. Laying birds are unlikely to overeat and it’s important they get all they need if they are to lay to potential. At the same time, any uneaten food will go stale and attract birds, rats and dogs so it’s best to put out roughly the right quantities to begin with.

If your birds get mash or pellets and little else, start by offering about 100 to 120 grams per bird per day for the lighter breeds, less than that for bantams — around 80 grams. For heavy breeds Increase this to about 180 grams per bird. If a cup of flour weighs about 100 grams, you are looking at a little over a cupful of mash a day for the lighter breeds and nearly two cups each for heavy breeds or your meat birds.

How often should you feed? From the bird’s point of view the optimum is two to three feeds a day. If you have six chooks they would enjoy a couple of cups of mash in the morning, at noon and again at night.

Most of us don’t have the time to spend all day trudging down to the hen house and back, although if you can find the time, it’s therapeutic to sit and watch the chooks feed. If you’re too busy, try the time honoured method of employing junior poultry managers — send the kids down to do the job before and after school. That leaves one feed for you to do at noon.

Three feeds a day is often just not possible, so cut it to two — morning and night. Too busy in the morning? Chooks will adjust to one feed a day, but it’s far from ideal. In this situation use an ad lib feeder so they can peck at mash or pellets whenever they like.

Water is also critical. Laying hens will drink up to 500 ml a day and if they run short will be stressed and could go off the lay. Make sure their water supply is always full, always fresh and keep water bowls where hens cannot foul the water with droppings and dirt. Keep it just outside the house where hens can poke their head through to drink. You don’t want a water bowl inside the henhouse. Too much water dropped on a deep litter floor will make it damp and encourage bacteria and disease.

In theory a good all-purpose food plus plenty of fresh, clean water is all your birds need to remain healthy. But mash and pellets are no more than a bare bones diet. Birds must be able to perform economically on it or all the commercial egg farms would go out of business. Still, most of those who keep chooks today like to think our fowls live really happy, healthy and well balanced lives. There’s no doubt they do better and enjoy life more with some supplements for variety. And we have the time and resources to offer that little bit extra. So keep that traditional kitchen scrap bucket. All those vege peelings can be cooked up and added to the mash.

If you have fruit trees, come preserving time there will be masses of delicious skins and seeds. Add a bit of milk to mix the mash. Stale bread, left over mashed potato … you name it, chooks will eat it.

A word of caution though. Don’t make radical, abrupt changes to an established feeding regime. If you are going to give them milk, start with just a little for a week. If the vege garden starts producing heaps of pumpkins, don’t suddenly ask your hens to eat boiled pumpkin and nothing else. Introduce anything new gradually.

Don’t reduce grain rations unless you are sure the extras you are offering are as balanced and will complete the meal, or feed the hard rations first — it’s these that will produce the eggs. Scraps will be appreciated as after dinner treats but are not nearly as nutritious as hard feeds.

The most ‘natural’ way to add supplements to the diet of your flock is by allowing them adequate free range in an appropriate, resource-rich environment. Bringing the supplements and extras to the chooks may be less work for you, and require less in the way of ‘infrastructure,’ but free range activity does a lot more than provide a source of dietary supplements, or a way to clean up the pests in the vege garden.

In the right environment, free range foraging provides huge benefits for the overall health of your flock. Not only do they pick up a lot of vitamins, additional protein (from bugs mostly) and necessary supplements like grit, they get a lot of benefit from the exercise and the time spent in the sun. A hen with regular free range access to good sized plant- and litter-rich areas will have stronger musculature, produce stronger-shelled eggs, and generally be far more resistant to disease.

Importantly, they’ll also be more content and less prone to behavioural problems. There may be the odd squabble over a particularly juicy bug but these will rarely develop into full scale fights, which can lead to injuries and, sometimes, to more serious issues. While it is not always possible to provide a large run containing lots of plant material and insect-rich soil, the more you can get your hens out into the open and allow them to express their natural behaviour the healthier they are likely to be.

So, what should you be providing as supplementary feed? For most backyard flocks the best source of health-giving extras is the vege garden. Ideally you want to fence it so you can allow your chooks in at appropriate times — to clean up after a crop has been harvested — and keep them out when you want to protect your crops from their marauding ways.

The vege garden is also an ideal way to grow your own chook feed. The trick is working out what combination will be best to give your flock the balanced diet they need. Basically, chooks need water, carbohydrates, protein and fats, minerals and vitamins. Much the same as us really.

Grains are the best hard food and there is no secret to growing wheat, barley and oats, or pulses like lentils and chick peas. Choose which grains suit you and the local climate best. But don’t feed green or immature wheat, it must be well ripened — the proverbial golden grain. Corn and wheat are both high in carbohydrates so are especially good for fattening birds for the table or keeping them warm during winter.

Most vege gardens will include some peas, beans, corn, and maybe sunflowers. Chooks love sunflower seeds. Just plant a heap more of these crops than you need for your own family, and keep the surplus for the chooks. Organise some cool, dry storage to make it last through the year.

Protein can be either vegetable or animal. It is usually the more expensive part of a feed and there is no point overdoing it — any excess will simply pass through and end up fertilising the ground anyway.

Laying hens need about 16 per cent protein in their diet to lay to potential. If your feed is of poorer quality the chooks have to eat more to get the protein they need or their productivity will suffer.

Protein can be supplied by adding meat and bone meal or skim milk powder to the feed, or mixing feed with milk. Birds on a free range regime will meet some of their protein needs with the bugs, slugs and worms they scratch up, but not all of it.

Commercial feeds often contain added minerals and vitamins, and synthetic mixes are also available, but free range birds can get most of their needs from foraging, as long as you have the right plants growing for them to pick through.

Comfrey is a terrific plant for chooks (and for enriching the vege garden). A good, deep rooted plant rich in minerals, plant comfrey where it can poke its leaves through the fence into the chook run and they will peck at it as they need it.

The brassicas — broccoli, cabbage and caulis — are also good, but don’t expect chooks to eat too much raw cabbage or other stringy greens. They can leave them with a mass of undigested fibre sitting in their crop.

Silverbeet, lucerne, the outside leaves of lettuce, even lawn clippings will be appreciated, as will a lot of weeds — sow thistle, shepherds purse, lotus, dandelions, plantains, nettles, twitch, oxalis and onion weed to name a few. If you are not using moveable arks and your permanent chook run is a decent size it’s the one place you can happily let run to weed.

One of the most important feed ingredients is calcium. All those egg shells have to come from somewhere. If your hens are not getting adequate calcium they will either go off the lay, produce soft shelled eggs or rob their own bodies of calcium supplies.

Avoid this by making sure they get bone meal, or add lime direct to their meal, about 1 per cent by weight. Or give them plenty of shell grit. Besides supplying calcium, grit is the grindstone the chooks need in their crops to help digest all those grains and lumpy bits. They will seek it out after eating so keep a container of grit next to the feeder. Ground up shell is the simplest. Failing sea shells, crush egg shells beyond recognition and feed those back to the hens. If you supply calcium in other forms the chooks may settle for small pebbles or coarse sand as grit.

That takes care of basic needs, but there are lots of treats you can offer to excite the chook palate. If your chook run is movable, keep it in the orchard so they can enjoy the windfalls. If the house is permanent, plant some grape vines over the netting. It provides shade in summer and grapes in autumn.

If you keep ducks instead of chooks, or both, note that while ducks enjoy many of the same foods there are a few differences. They prefer their feed wet so mix mash with plenty of water or milk. They also need more protein than hens, at least 20 per cent protein as a minimum.

Ducks are more creatures of habit than chooks and like a regular routine. If you want to collect eggs, the simplest system is to let them out to forage during the day, call them home about three or four pm with a good feed of mash and scraps and shut them in their pen. Give them a wet mash meal as early as possible the next morning. They should have laid their eggs by nine am and you can let them out again.

Ducks love a stream or pond to swim in but it isn’t vital as long as they have access to good clean water at all times. Just make sure their water bowl is big enough and deep enough for them to get their whole faces in for a good wash.

Like chooks, ducks need plenty of fresh greens but they are excellent foragers and will take care of most of their own requirements if you let them out of their pen onto good pasture every day. They cannot cope with dry mature grass so keep the forage area tender and sweet for them by running a lawn mower over it occasionally. If the birds are penned permanently be sure to bring them a daily selection of fresh greens. Don’t just toss greens into the run, where they’ll wilt and dry out, chop them into small pieces and add them to the drinking water. Your ducks will have fun all day fishing them out.

Whatever feathered fowl you choose to make part of your family, remember that you can learn best from the birds themselves. Watch what foods they scramble for, observe when they lay best and what they were eating at the time. Watch for changes in their feathers when their diet alters.

And remember the pecking order. Sometimes younger birds are bossed away from the trough and have to survive on what falls to the ground. Eventually they will starve unless you feed them separately or put out enough feed to ensure everyone gets their fill.

If you are new to keeping poultry and you practice just some of the tips in this article you will have healthy, happy and contented chooks — and will rarely if ever need to call in the vet.

Polyomavirus (PVD)

Polyomavirus (PVD), sometimes also referred to as Budgerigar Fledgling Disease, is one of the most serious threats to cage birds. A member of the papovavirus family, this highly infectious disease can affect most if not all parrot species as well as other avian species. The virus seems to be most problematic among young birds between the ages 14-56 days, who will often die. Adult birds can develop a certain level of immunity.

Polyoma is believed to have an incubation period of approximately two weeks or less and the pathogen can be spread from one bird to another via feather dust, faeces, aerosols and parental feeding of chicks as well as by direct contact between birds, or contact with infected environments such as incubators and nest boxes.

Birds that are infected but do not have obvious signs of infection are often responsible for spreading the virus to an aviary. Some adult birds can show no symptoms but may be carriers.

Clinical signs:

There are a number of symptoms of the disease and affected birds may show any or all of the visible signs, including…

  • Swollen abdomen
  • Depression
  • Loss of appetite
  • Anorexia
  • Weight loss
  • Delayed crop emptying
  • Regurgitation
  • Diarrhoea
  • Dehydration
  • Feather abnormalities
  • Haemorrhages under the skin
  • Dyspnea (difficulty breathing)
  • Polyuria (excessive urination)
  • Ataxia (loss of muscle control/co-ordination)
  • Tremors
  • Paralysis

Some birds die without any clinical symptoms. Adult birds may die of secondary infection from bacterial, viral, fungal or parasitic pathogen.

How is PVD diagnosed?

Diagnosis is made in the laboratory from whole blood samples and cloacal swabs when possible and is conducted via nested primer PCR testing, sequence analysis of PDV DNA and histopathology. If a sample tests positive, the suspect bird should be placed in quarantine and re-tested in 4-6 weeks. If the bird tests negative the second time, a third test is recommended.

Treatment protocols:

Unfortunately, there is no known treatment for PVD at this time. A vaccine is available, however, its effectiveness in younger birds is open to question. Booster shots are required each year.

Bird fanciers can help prevent this generally fatal disease by isolating all birds shedding the disease and disinfecting all contaminated surfaces with an oxidizer such as chlorine bleach (Polyoma virus is resistant to many disinfectants).

Poultry: Moulting

The moult is a natural process where hens shed and renew their feathers. During the moulting period the reproductive physiology of the bird has a complete rest while the bird builds up its body reserves of nutrients, leading to a seasonal decline in egg production.

The provision of new feathers or a coat (a feature inherent in most animals) is a natural process, designed by nature to maintain a bird’s ability to escape enemies by flight and better protect against cold winter conditions.

Under normal conditions adult hens will moult once a year, in autumn. Some may moult twice in a single year or, more rarely, only once in two years. When a bird begins the moult depends on the time of the year she started laying. In most cases moulting should begin sometime during March or April, and egg production begin again around July.

Young pullets (chicks) will also moult, going through one complete and three partial moults during its growth to point of lay. Generally, the first, complete, moult will occur between 1 to 6 weeks of age. Partial moulting will take place at 7 to 9 weeks, 12 to 16 weeks and 20 to 22 weeks.

The three main factors that trigger moulting in poultry are:

  • Physical exhaustion and fatigue.
  • Completion of the laying cycle (birds can lay eggs only for a specific length of time).
  • Reduction in day length as winter approaches results in reduced feeding time and consequent loss of body weight.

What to watch for:

If a bird stops laying and moulting, it’s a sign that its physical condition is deteriorating, leaving it incapable of either egg production or the continued nourishment needed to produce feathers or maintain body condition. This is usually a sign of inadequate nutrition. During the moult the bird must still get a considerable amount of good quality food to replace feathers and build up condition.

For the backyard poultry keeper the date at which a laying hen stops producing eggs and goes into the moult is a reliable guide as to whether or not an individual hen is a good egg producer. Poor producing hens will moult early and take a long time to complete the process and resume laying, staying out of production for six to seven months. Poor producers seldom cast more than a few feathers at a time and rarely show bare patches.

High-producing hens on the other hand, moult late and for much shorter periods, around 12 weeks, and come back into production very quickly. A good layer will moult quickly, losing not only wing feathers but body feathers generally. A late and rapid moulting hen will often be practically devoid of feathers, with bare patches over its body.

Moulting takes place in a specific sequence. The first plumage is lost from the head and neck, then from the saddle, breast and abdomen (body), then from the wings and finally the tail feathers will drop.

When the first feathers drop from the neck and body, good layers often keep laying. However, when the wing feathers begin to drop, laying usually ceases.

Although it may look random to the inexperienced eye, feather loss occurs in a very specific and observable way. The main wing feathers consist of four tiny ‘finger’ feathers on the extreme tip of the wing, 10 large primary or ‘flight’ feathers, a small axial feather and 14 secondary feathers, which are smaller and softer than the primaries.

When the wing moults, the large primary feathers are shed first, from the axial outwards to the end of the wing. The number one primary feather is the first to drop, followed by a number two and then in order to number 10. While the primaries are shed, the secondaries begin to drop.

The axial feather drops at the same time as the secondary next to it. A new quill starts to grow as soon as the old feather is out, and takes approximately six to seven weeks to grow. The moult is complete when all primary flight feathers on the wing have been replaced. The feathers of bird in which the moult is complete are bigger, softer, cleaner and brighter than the old feathers, which would have been small, hard, dry and somewhat tattered.

The difference between a rapid and a slow moulter (or a good layer versus an indifferent layer) is not the differences in the growth rate of individual feathers, it’s the fact that a quick moulter can renew a large number of feathers at the same time. The backyard poultry keeper can use this to measure the rate of moulting by examining the number of flight feathers on the wing that are simultaneously being replaced. If a hen has grown some of her primaries before starting to lose her secondaries it indicates that she laid well into the moult and is therefore a good layer.

Most often, a layer will moult once egg production ceases, although, if the bird has an inherited tendency for high production, moulting will probably begin before she stops laying eggs.

If a hen ceases egg production for any reason other than mild sickness or broodiness, it loses its feathers. If a hen ceases production during spring or summer it may moult one or two primaries, stop moulting and come into lay again. A bird may also sometimes experience a neck or partial moult without any loss of production. However, if the moulting extends beyond the neck moult stage the hen ceases production.

Some birds may even moult continuously; these individuals are easily identified because of the spotless condition of their new feathers. Birds who moult this way are invariably poor producers and should be culled.

Stress factors and moulting:

Although the moult is a naturally occurring process, moults can also be triggered at any time if birds are subjected to stress. Any change in the flock’s environment or management can present birds with a challenge to which they cannot respond without harmful effects. A mild stress condition in late spring when in full production can trigger a drop in egg production. A hen subjected to the same stress condition in autumn will cease laying and moult.

Common stress factors that can induce moulting include disease, poor nutrition, extremes of temperature, poor housing, the presence of predators and poor flock management.

To ensure healthy birds and regular egg production you should aim to provide adequate housing, adequate nutrition and a balanced diet, access to the outdoors and continually rotating fresh forage.

Scaly Mite

Scaly Leg and Scaly Face are both relatively common conditions in domesticated birds and poultry. An infectious disease that can quickly spread to other birds, both conditions are caused by the Cnemidocoptes mutans mite, commonly known as either the Scaly Face Mite or the Scaly Leg Mite. In canaries the same mite causes a condition called ‘Tassle Foot.’ A smaller cousin of the spider, the tick and the scorpion, the eight-legged Cnemidocoptes burrows under the scales of the legs and into the keratin of the bird’s beak. It can also be found around the vent of birds and on their wing tips.

The mite spends its whole life cycle on the bird and is transmitted between birds by direct contact. Some birds appear to be genetically more susceptible to the mite than others.

Clinical signs:

Signs of Scaly Leg (and  Scaly Face) differ between birds. Infected budgerigars will show a white, chalky and crusty beak with a honeycomb appearance. This crustiness can spread around the bird’s cere (the lump at the base of the beak) and its eyes. The crusty, scaly appearance also appears on the legs and sometimes around the vent. Left untreated the infection in the growing portion of the beak can cause the beak to become permanently deformed.

In canaries and poultry the condition usually shows first as a thickening of the scales on the legs, which develop areas that look something like corns with the scales raised or protruding rather than laying flat.

How is Scaly Mite diagnosed? 

The condition is usually fairly obvious by visual inspection but we may confirm the infection by taking a scraping of the affected areas and examining it under the microscope. If mites are present they are easily visible, looking much like miniature mud crabs.

Treatment protocols:

There are many ‘home remedies’ but the most effective veterinary treatments are Avomectin, Ivermectin or Moxidectin. These are all prescription medications available from the hospital which can be applied at home.

The most commonly used DIY treatment is to apply Vaseline to the the beak, the cere, and the legs of affected birds. It needs to be applied daily for up to 10 days. While Vaseline will not kill the mites it suffocates them over a period and helps soften the crusts and remove them from a bird’s face and legs. While this is a very popular home treatment, we really cannot recommend it. The petroleum jelly (paraffin oil is also popular) is difficult to apply sufficiently well to ensure all the mites are suffocated, and the Vaseline or oil can get onto feathers, where it is very difficult to remove.

The big problem is not how to treat individual birds but the fact that the condition is contagious and unless treated correctly and thoroughly can easily spread to other birds via direct contact. It’s essential to kill off all the mites. The use of swift acting products such as Ivermectin is far more effective than home remedies at killing the wee beasts.

If damage from the mites is causing an affected bird’s beak to grow abnormally, regular trimming may be required. This should only be undertaken by a veterinarian.

The mite can also burrow into tiny cracks and wooden perches in the cage. Perches in an infected cage should be replaced every week. Using branches from native trees helps discourage the mites.

  • A healthy diet for your ferret
  • Adrenal gland tumours in ferrets
  • Desexing (neutering) ferrets
  • Diarrhoea in ferrets
  • Distemper in ferrets
  • Influenza in ferrets

 

A healthy diet for your ferret

Feeding your ferret an appropriate diet is crucial to its health and wellbeing. In fact, it’s critical to your pet’s very survival.

Ferrets are carnivores. More precisely, they are obligate carnivores. Like cats, ferrets cannot survive without meat in their diet. In the wild the ferret is a predator, eating not only the muscle meat of their prey (usually rodents) but also the liver, kidneys, and intestinal tract, and crunching through bones as well. Obligate carnivores have special requirements for several nutrients found only in the ‘side dishes’ the ferret gets when they eat an entire prey animal. A diet limited to meat alone will cause harmful and eventually fatal nutritional imbalances.

More than most companion animals it is vitally important that you start your ferrets on a nutritionally adequate diet when they’re young, and maintain that diet throughout their lives. A good diet for a ferret must be:

  • high in energy
  • high in protein
  • low in fibre

You could of course mimic the diet they would find in the wild by feeding live mice. However, apart from the inconvenience, the mess and the fact that, given the ready availability of correctly formulated foods, it’s unnecessarily cruel to the mice, you also run the risk of your pet ferrets ingesting parasites and viruses.

A much easier way is to feed one of the high quality, premium pelleted ferret foods now available. The best of these are correctly formulated to provide everything the ferret needs in its diet.

If these are not available a good quality kitten or cat food is an acceptable alternative. Because cats are also obligate carnivores a properly formulated premium cat food is nutritionally adequate for ferrets.

That applies only to cat foods. You should not feed your ferrets dog food. Ferrets fed solely on dog food will eventually die of malnutrition.

Ferrets cannot digest the high levels of carbohydrate in dog food. They need to get most of their calories from simple carbohydrates, protein, and fat. The very short intestinal tract of the ferret also means they can’t eat very much at a time, and therefore need a concentrated diet. A well fed ferret should get most of its calories from fat, the most concentrated source of energy.

Why premium foods?

A ‘premium’ pet food, whether for dogs, cats or ferrets, is a commercially prepared concentrated food generally available only from veterinary clinics or specialist pet food suppliers. Whether it is a dry, pelleted food or a canned soft food, premium foods are usually more expensive than those available from supermarkets. At Alpine Animal Doctors we do understand that some pet carers feel that these foods are “no better” than the cheaper generic foods you can buy off the supermarket shelf, or that they are “just a way for the pet food manufacturer to make more money.”

Not surprisingly, as ‘animal dieticians’ most vets, including Dr. Bek, would disagree with that. We know from both experience and from many independent scientific studies that premium foods are not only better nutritionally they are often just as economical as generic foods, because of the smaller amounts needed for a nutritionally balanced meal. But we fully respect the right of a pet carer to make their own choices on what they feed their animals, and we do not normally ‘push’ premium foods on anybody.

Ferrets, however, are a unique case and we stress the importance of feeding them premium foods only because of the ferret’s very specific nutritional requirements.

Modern premium foods for ferrets are precisely formulated to provide everything the ferret needs for a nutritionally balanced diet, and to also be tasty and palatable to an animal that can be very finicky about what they will eat. Ferrets will sometime refuse to even taste an unpalatable food, and will starve if offered no acceptable alternative.

Even with premium foods, some are better than others. Foods containing corn can cause digestion problems and kidney stones in ferrets. Fish protein, sometimes found in dry ferret foods based on mink foods, is not nearly as good for ferrets as is meat protein and can also make the food unpalatable to the ferret, which does not eat fish in the wild. When purchasing a premium ferret food always check the ingredients. We are happy to advise on the most appropriate mixes.

Many people feed their pet ferrets cat foods. In most cases this is fine, although a kitten food is better than those formulated for adult cats. While the premium cat foods sold in veterinary clinics are not formulated specifically for ferrets most of them are palatable and all are nutritionally adequate for pet ferrets. However, generic cat foods — the packeted and canned foods sold in supermarkets — are not good for ferrets. Almost all of them have some important nutritional deficiencies, and all are too low in fat to provide a healthy diet for a ferret.

Although generic cat foods may cost less than premium foods, your ferret will have to eat at least twice as much of these cheaper food to get the calories he needs. That’s not good for your ferret or, in the long run, your pocket.

Mixed Grill

Like cats, ferrets can be very finicky about what they eat. They can become hooked on a particular flavour and refuse to eat anything else. When that happens they can be very hard to wean off their cravings. In extreme cases they may starve rather than eat something they have taken a dislike to. It doesn’t matter to the ferret if their favourite flavour or food is nutritionally inadequate.

To avoid this start your young ferret on a good diet, one that you know is going to be readily available in the future, and stick to it. Feed a mixture of several kinds of ferret and cat foods to get the ferret accustomed to a variety of flavours. If you do have to drastically change the diet of a ferret, stomach upsets and diarrhoea are likely to follow. Always change diets gradually.

Snacks and treats

Your ferret does not need snacks or treats. It’s really best that you don’t feed them at all. It’s not that there is anything inherently wrong with feeding a treat from time to time. The problem is that most ferret parents tend to feed the wrong kinds of treats, which can lead to nutritional issues. Although the ferret is a carnivore that doesn’t stop them loving the taste of other foods, including fruit and sweets.

None of them are good for the ferret. Sugary snacks can cause obesity, tooth decay, and can aggravate hypoglycemia caused by insulinomas. Dairy products, although very palatable to ferrets and nutritionally beneficial, cause diarrhoea.

As bad as the lack of nutritional value in such snacks, ferrets can begin to crave certain tastes and flavours to such a degree that they won’t eat anything else.

A treat is however useful as a training aid. Just be careful not to use anything high in fibre, sugar or with an intense flavour that may hook the ferret. Ideally, use a pellet from their usual diet.

Soft food or pellets?

We believe the best diet is a properly formulated dry food, which is always available when the ferret needs to eat. A good formulation will also have beneficial effects on the ferret’s teeth, limiting the formation of plaque and gingivitis. If you prefer to give your ferrets some soft foods, mix it with a dry food and serve the same mix at every meal to avoid the ferret getting hooked on one flavour. Remove soft foods immediately after meals to avoid it going off and causing stomach upsets.

Ferrets on dry diets need a constant supply of fresh water, about three times the volume of dry pellets eaten. In warm weather they’ll drink much more. Ferrets prefer to drink from a dish rather than a bottle clipped to the cage. A dish of fresh water should preferably be made available to the ferrets when they are let out for exercise to avoid contamination with food and litter. However, if exercise is infrequent or spasmodic water should be left in the cage. Ferrets that have no water for 24 hours will stop eating.

 

Adrenal gland tumours in ferrets

One of the more common health issues ferrets are prone to is developing tumours (lesions) on the adrenal gland. The first sign is generally hair loss.

Although these lesions are themselves not generally life threatening and tumour metastasis is rare, it is important for ferret owners to understand that the alopecia (hair loss) associated with adrenal tumours is not merely cosmetic. The health changes seen in ferrets with adrenal disease are cumulative and progressive. Ferrets with adrenal lesions do not just lose their hair — they also lose muscle mass, have little energy and a poor quality of life

Clinical signs:

Ferrets with adrenal lesions — including areas of hyperplasia as well as both benign and malignant neoplasms of the adrenal cortex — will typically all show the same clinical signs. In the majority of cases these are so characteristic no further diagnostic tests are required.

Adrenal lesions may be seen in animals ranging from one to seven years of age, with the average being around 3.5 years.

Hair loss, or alopecia, is by far the most common clinical sign. It usually begins at the tail, progressing forward over the trunk, flanks, and abdomen until hair is only present on the neck, head, and paws.

In speyed females the vulva will often become swollen and a watery mucus discharge from the vulva may also be seen.

Other clinical signs may include increased scratching, excessive drinking and urination, anemia and weight loss. In advanced cases difficulty in using the hind legs may be observed.

Treatment protocols:

The only treatment for adrenal disease in ferrets is the surgical removal of the affected adrenal gland. In most cases one gland, usually the left, is found to be noticeably larger than the other and is removed. Removal of one adrenal gland is generally successful in treating the condition. In most cases, if disease is confined to that gland, the hair will grow back and the swollen vulva will diminish to its previous size.

In animals with bilateral disease, i.e., where both adrenal glands are affected, removal of one gland and part of another can be done, but carries a more significant risk of post-operative complications. Bilateral adrenal disease is seen in only about 8% of affected ferrets.

Removal of the left adrenal is fairly easy and has a low risk of complication. The right adrenal gland, because it is located near several large blood vessels, is more difficult to remove. Fortunately, the left adrenal gland accounts for about 64% of all lesions.

The surgery is not especially complex and the prognosis is reasonably good for most patients undergoing the surgery.

 

Desexing (neutering) ferrets

Desexing (speying) your female ferrets is not so much an option but more a necessity to ensure their continuing health. Your pet could die if not speyed.

A female ferret is what is known as an ‘induced ovulator,’ meaning they remain in oestrus (heat) for long periods if not mated. This results in too much oestrogen being produced and the hormone eventually poisons the bone marrow, leading to oestrogen toxicosis and aplastic anaemia.

The symptoms of oestrogen toxicosis are weakness, a refusal to eat, pale gums and haemorrhage. By the time a Jill reaches this stage she is extremely sick and has a poor prognosis, often requiring multiple blood transfusions.

Around 90 per cent of un-speyed females will develop aplastic anaemia unless mated every time they come into heat, which can be twice in one season. Unless you are a breeder it’s unlikely that you will want a permanently pregnant pet. Alternatively, your Jill can be given an expensive hormone injection every time she comes into heat.

Neither of these are satisfactory solutions to what can be a serious health issue for your pets. Fortunately, oestrogen toxicosis and aplastic anaemia are easily prevented by the routine desexing of all female ferrets not intended for breeding.

Male ferrets (Hobs) should also be desexed. Neutering a male ferret will reduce his aggressiveness and substantially reduce that ‘musky’ odour the male gives off when in season.

In both males and females desexing will also make your pets more content and help them get along together.

The surgical procedure is routine and safe. For both males and females we recommend the procedure is carried out before the ferret reaches six months of age.

 

Diarrhoea in ferrets

Whilst not a disease in itself, a bout of diarrhoea in your pet ferret is a clear indication that all is not well and that there are underlying issues that need to be addressed.

Because of their small size any episode of diarrhoea can quickly lead to ferrets becoming dehydrated, which can have serious consequences, particularly for very young or very small ferrets and for elderly animals. Both very young and senior ferrets can have trouble regulating their body temperature and coma may result from untreated diarrhoea.

If you see diarrhoea contact the hospital as soon as possible for advice. In the meantime, encourage the affected animal to drink as much as possible.

Clinical signs:
The signs of diarrhoea are fairly obvious: A loose, liquid or semi-liquid stool.

The causes can be less clear and diarrhoea can be triggered by many things. Sudden changes in diet are probably the most common. This includes any abrupt change of food, a sudden increase in one type of food, the use of ‘snacks’ and ‘treats’ particularly dairy products, or even a change in the brand of commercially prepared foods. Ingesting substances such as plastic or contaminated food, over-eating, an intolerance to certain food, stress and hygiene issues are also things that can cause upset stomachs and diarrhoea.

Some chemicals and certain medications can also lead to diarrhoea. Viruses and parasites such as fleas are other causes. Proliferative colitis is a disease of young ferrets caused by a Campylobacter bacterium and results in a mucous-like diarrheoa containing blood. The animal may also show anorexia (loss of appetite), weight loss, lethargy and depression.

Treatment protocols:
Identifying and treating the cause of the diarrhoea is our first concern. We rely heavily on information provided by the owner to isolate possible causes. In most cases we will suggest fluids and electrolytes be given to counteract dehydration. In severe cases this may need to be done in the hospital by means of a drip. Infectious causes of diarrhoea are normally treated by antibiotics. Known ingestion of material likely to cause diarrhoea may be treated with a laxative.

Distemper in ferrets

Ferrets are susceptible to the same virus that causes distemper in dogs. The disease is 100 per cent fatal in ferrets.

Clinical signs:

Symptoms are similar to those seen in dogs and include thick discharges from the eye and nasal cavities, fever and loss of appetite. Often a rash under the chin and groin area will also be seen.

The incubation period is 7 -10 days and the terminal phase of the disease is characterised by central nervous system signs, including muscle tremors, convulsions, coma and death.

Affected ferrets must be isolated from other susceptible animals and supportive treatment may be started until the diagnosis of distemper is certain, after which euthanasia is usually the only option.

NOTE: The clinical signs for distemper are very similar to those of influenza in ferrets.

Treatment protocols:

Fortunately, there is an effective vaccine to protect your ferrets against canine distemper. All ferrets should be vaccinated against distemper at 8, 12 and 16 weeks of age, then annually thereafter.

 

 

 

Influenza in ferrets

Ferrets cannot ‘catch a cold’ but they are susceptible to the human influenza virus (flu). The incubation period is 24 – 48 hours and the course of the disease usually lasts five days.

Clinical signs:

Symptoms are similar to those in people with the flu (and to the early signs of distemper) including nasal discharge, sneezing, fever and loss of appetite.

Treatment protocols:

Treatment consists of decongestants and antibiotics if there is a secondary bacterial infection. Occasionally fluid therapy may be required.

Affected ferrets should be Isolated, especially from young ferrets, who can be more severely affected by the disease.

Never give flu or cold medications intended for humans to your ferrets. These will never help and in most cases are as likely to poison, or even kill, your pet.

COPING WITH PETS IN CRISIS — advice for owners

If your cat is showing any of the symptoms listed here you should seek immediate veterinary advice. You should not attempt to treat a cat showing these potentially serious symptoms yourself — contact a vet.

  • Wounds & Injuries
  • Loss Of Consciousness
  • Breathing Difficulties
  • Discoloured Gums
  • Severe Pain
  • Persistent Vomiting
  • Salivation & Drooling
  • Diarrhoea

Open wounds and trauma

The most obvious symptoms that require visits to the animal hospital are open wounds, serious burns, and broken bones. If your cat has been hit by a car or in a fight with another animal, or been involved in any other kind of accident or trauma, unless the wounds are clearly minor you should seek emergency veterinary treatment as quickly as possible.

For minor wounds you may be able to provide some basic first aid yourself.

When your cat is non-responsive

If your cat does not respond to being touched something is wrong. Non-responsive behaviour is often the first obvious sign of loss of consciousness. It may be because of a foreign object, or a bone, stuck in the airway and impeding breathing, or the result of ingesting something toxic. Other signs of a change in consciousness are stumbling or staggering, confused behaviour and/or listlessness.

Gasping for breath

If your cat is struggling to breathe, gasping for air or his breathing becomes laboured it’s a sign of possible obstructions in the airway or of fluid in the lungs, and can lead to respiratory failure or cardiac arrest. Call the hospital immediately.

If your cat’s gums change colour

If your cat’s gums become white, pale, or have a blue or yellow tinge it’s a sign of serious health issues. Whitish or pale gums may suggest serious iron deficiency. Blue-tinged gums can indicate respiratory or cardiac problems. Yellow gums point to a severe liver disorder, or issues with the red blood cells. Any change from the normal colour of your cat’s gums is a sign she needs emergency treatment.

Screeching or hiding indicates distress

If your cat begins to make a lot of unusual and incessant noise, particularly loud yowling, he is probably in acute pain or severe distress. Loud complaints may also be accompanied by a tendency to hide. This kind of unusual behaviour can indicate a life threatening bladder obstruction, amongst other things. if a cat sounds like he is in pain or distress, he probably is. Treat unusual levels of noise as an emergency.

 

Continual vomiting is not normal

Cats vomit occasionally. It’s normal, for the most part. However, continual vomiting on a daily basis, even when you know the cat’s stomach is empty, indicates illness and can be a sign of serious intestinal obstruction or of ingestion of poison. If your cat is unable to keep even water down something is wrong, and he is at serious risk of rapid dehydration.

May be signs of poisoning

Excessive drooling is not normal and can indicate anything from ingesting things like household cleansers or eating poisonous plants to a burn on the mouth or tongue. It can also be a sign of systemic illness or a mouth tumour, which may not be visible.

Persistent diarrhoea can be serious

Diarrhoea is not necessarily an emergency but diarrhoea that continues for more than a few hours or at most a day or so, especially in kittens, can be a sign of a serious underlying condition, or poison ingestion. Constant diarrhoea can cause rapid physical deterioration and dehydration.

All of the symptoms shown below indicate that your dog could be facing a life threatening situation and may require urgent veterinary treatment

  • Wounds & Injuries
  • Loss Of Consciousness
  • Seizures
  • Breathing Difficulties
  • Discoloured Gums
  • Lethargy
  • Severe Pain
  • Bloating
  • Poisoning
  • Snake Bite

 

Coping with wounds and injuries

The most obvious symptoms that require visits to the hospital are open wounds, serious burns, and broken bones. If your pet has been hit by a car, tangled with some barbed wire, been in a fight with another animal, or been involved in any other kind of accident or trauma, unless the wounds are clearly minor you should seek emergency veterinary treatment as quickly as possible, especially if you are unable to stem any bleeding.

If this is impossible you may wish to attempt some first aid at home. However, this is no substitute for expert veterinary care and should be a last resort. Always seek veterinary advice before attempting anything other than first aid for minor wounds.

Recognising changes in consciousness

Any alteration in consciousness can be a sign of acute illness. Signs can include coma, convulsions, sudden withdrawal, tremors or staggering, sudden onset of blindness or a general lack of responsiveness. Other signs can be biting at imaginary objects or constantly tilting the head.

Any of these symptoms can indicate serious problems requiring prompt treatment. You should not delay in consulting a veterinarian.

Seizures can be serious

A seizure is fairly easy to identify. The animal’s body will shake or tremble for several seconds and you may see the whites of the eyes. The dog will then remain unresponsive for up to a minute.

While a seizure does not always indicate a critical emergency, if you are seeing your dog affected for the first time you should assume it may be a sign of a serious condition. Err on the side of caution and seek immediate veterinary attention or advice.

Changes in breathing

If your dog is gasping for breath or his breathing is laboured, respiration is noisy and/or the tongue has swollen or turned blue it can be a sign of acute illness.

Heavy or laboured breathing can indicate heart problems, overheating or a respiratory problem. It may also just be a sign of localised stress or complications from obesity, in which case a normal appointment at the hospital will be fine.

You need to make a judgement but always err on the side of caution — if in doubt, call the hospital.

When your dog’s gums change colour

If your dog’s gums have become white, blue or otherwise paler than normal this can be one of the first indicators of serious illness. Discoloured gums are signs of:

  • Shock
  • Low blood pressure
  • Poor circulation
  • Anaemia
  • Internal bleeding

Any of the above can be signs of serious issues. Seek veterinary help.

When Rover loses his zest

If your dog becomes unusually lethargic, take its body temperature. Between 37 and 38 degrees celsius is normal for dogs. Anything outside of this range indicates you should seek immediate veterinary attention.

 

When your dog is in pain

Dogs tend to be stoic about pain but if your dog is crying out loudly and continually it is a sign the animal is in severe pain. If there is no obvious external cause such as a wound, dislocation or broken bone the pain may be due to internal injures that require immediate treatment.

Warning: Please do not attempt to treat pain in your pet (dog or cat) with human painkillers such as Paracetomol, Ibuprofen etc, etc. These can prove fatal for animals.

Bloating can be fatal

An abnormally distended abdomen is an acutely serious issue. If your dog appears to have abdominal pain and winces or whimpers when you touch the abdomen but is not vomiting it may indicate potential poisoning or an acute kidney problem.

Worse case is it’s indicative of bloat (Gastric Dilation) a very serious condition which, without prompt treatment, can lead to death within a few hours. If your dog is in pain, pacing and restless and displaying non-productive retching or vomiting, and excessive salivation get to our hospital, or your closest emergency vet, as quickly as possible.

Recognising poisoning symptoms

If you believe that your dog has ingested toxins such as pesticides, cleaning products, chocolate, onions, alcohol or human medicines, call the hospital emergency vet without delay.

Poisoning cases are almost always critical and will definitely require veterinary intervention. For more information check out the article links below…

Dangers of Panadol for Dogs,

Poisons: Recognising Symptoms,

Poisoning: Top 10 Causes in Pets,

Poisons: Health Effects & Treatment,

Poisons: Emergency First Aid.

Snake bites need swift action

Envenomation from the toxins contained in snake venom is a serious and life threatening incident that must be treated swiftly if we are to save the life of a pet that has been bitten.

The first signs of snake envenomation are usually excitement, trembling, salivation and vomiting, gradually developing into weakness, wobbly gait and eventually paralysis. Afflicted dogs often have dilated pupils and slow light reflex, with clotting problems in their blood. Depending on the type of snake and how much venom has been injected, an animal bitten by a snake may show any of the following symptoms.

  • Localised swelling and irritation
  • Vomiting
  • Drooling and trembling
  • Dilated pupils
  • Involuntary bladder or bowel release
  • Red or brown discolouration of urine
  • Rapid breathing and/or panting
  • Bleeding from bite wounds or bloody diarrhoea
  • Collapse. (An animal may collapse immediately but then apparently recover, then develop symptoms over the course of the next hour)
  • Paralysis (starting with the hind legs and progressing towards the head)

Speed is of the essence in successfully treating a snake-bitten pet. Get the animal to a properly equipped vet clinic as quickly as you can.

Despite their size horses can fall prey to a wide range of problems needing prompt veterinary attention.
Use the information below to learn when you need to call an emergency vet to your horse.

  • Assessing The Situation
  • Help Your Vet
  • Critical Emergencies
  • Urgent Emergencies
  • Minor Emergencies

 

Is it serious enough to call the vet?

Deciding what constitutes an emergency and when the vet needs to be called in can be a difficult decision for the horse owner. Despite their size and power horses can be fairly delicate. They can often pull up lame, suffer cuts and bruises and experience mild fever and reduced appetite. In a large percentage of cases these are not true emergencies, but they are always an anxious time for the horse owner.

Observation and a good knowledge of the vital signs of your horse is the best way in which you can learn to accurately judge when urgent veterinary treatment is needed and when you can safely treat the horse yourself, or it’s safe to wait and see if and how a condition progresses.

Use the time you spend grooming and feeding your horse to carefully observe his normal behaviour and vital signs so that you are immediately aware of any changes that might occur later. Learn the points at which you can measure the pulse and respiration of your horse.

Changes in temperature, pulse and respiration are always the first pointer to illness, abnormality or injury. Knowing what’s normal in your horse is critical to your ability to judge the relative seriousness of any condition that may arise and every horse owner should have a couple of simple tools to help them monitor vital signs — an accurate rectal thermometer and a stethoscope.

  • Normal temperature should fall into a range between 37 and 38.3 degrees celsius.
  • Normal pulse/heart rate should be 32-48 beats per minute. This can be affected by a number of factors including age, ambient temperature, humidity, recent exercise and excitement. Spend a little time establishing a record of a baseline for your horse by taking pulse rates at various times, e.g., before and after exercise. The heart/pulse rate can be checked by placing a stethoscope over his ribs, just behind the elbow. Or if you don’t have a stethoscope, take his pulse on the inside of the jaw or on the ankle.
  • Normal respiratory rate in the horse should fall within a range of 12 to 16 breaths per minute, although, again, this can vary due to humidity, exercise and excitement and other external factors. Watch the animal’s nostrils or flanks and carefully count the number of times the horse breathes out.

Equine emergencies are always a difficult and anxious time for the owner. But remember, it’s always harder for the animal, who may be in pain or severe distress. Act promptly to get advice and treatment.

 

Information we need to judge the extent of an injury

If you are in any way uncertain of the cause and severity of the symptoms your horse is showing, always err on the side of caution and call the hospital for advice. An experienced vet will be able to ascertain if it’s an emergency and advise you of the best way to proceed.

When calling for advice you can help the vet by having the following information ready:

  • Temperature, pulse/heart rate, and respiratory rate.
  • Your horse’s behaviour and demeanour; is he agitated, lethargic, depressed.
  • The location and severity of any injury, wound, or lameness.
  • The location of any swelling and if there is any heat present.

To avoid possibly unnecessary home visit costs for the owner, at Alpine Animal Doctors we triage all horse wounds, injuries and illnesses into three categories: Critical, Urgent, and Minor. A critical emergency requires immediate attention. Those classified as urgent still need prompt attention but may be able to be left until the following day on advice from the vet. Minor emergencies can often safely be observed and watched.

 

Issues requiring immediate veterinary treatment

The following details some of the situations we class as Critical Emergencies. If your horse shows any of these symptoms you should contact the hospital immediately, no matter what the time.

  • Profuse bleeding from any injury or wound which cannot be stopped, especially if the blood is bright red, which can indicate arterial bleeding.
    Injury to any vital structure (eyes, genitals, joints, etc) or wounds obviously requiring stitches.
  • Bone fractures or severe lameness on which your horse cannot bear his weight.
  • Abnormal respiration, including rapid, distressed breathing or blocked airways.
  • Signs of severe colic, including loss of appetite and reduced fecal production.
  • Staggering, lack of coordination, or marked behaviour change.
    Any injury or trauma to the eye, particularly where the horse is unwilling to open his eyelids.
  • Abnormal vital signs, including; temperature over 38.9°C; pulse above 80 beats per minute; elevated respiration while at rest.
  • Acute laminitis, seizures, heat stroke, or watery diarrhea.
  • Any mare who takes longer than 30 minutes to deliver her foal.
  • Suspected or known ingestion of a poison or toxin.
  • Puncture wounds.

 

Non-critical issues needing veterinary treatment

For the Urgent category of emergencies you should still contact the hospital the same day or no later than first thing the following morning. These situations do need prompt veterinary attention but can usually be safely managed with first aid measures until the vet can get there during normal hours.

  • Sudden onset of lameness on which your horse can bear his weight.
  • Superficial trauma or injury located away from the eyes, genitals, joints, e.g., relatively minor wounds and scrapes that don’t need stitches.
  • Attacks of chronic laminitis or chronic inflammatory respiratory disease when the horse has already been diagnosed and is being regularly treated/managed for these conditions.
  • Slightly elevated temperature, respiration, or heart rate.

 

Wait and watch

Minor category emergencies such as slight lameness, skin rashes, light discharge from the eye but without pain or vision loss, slightly reduced appetite or slight difficulty chewing when the horse is otherwise healthy, or a minor nasal discharge without any fever or heavy breathing can generally be left until you can schedule a normal appointment. Again, if in doubt, call the hospital.

First Aid

First aid for treating minor wounds in cats at home.
If you cannot stem a bleeding wound, or can see bone or soft tissue or suspect a broken bone or internal injuries, seek immediate veterinary advice.

  • Evaluate The Injury
  • Stop The Bleeding
  • Clean, Disinfect & Cover
  • Beware Of Infection
  • Puncture Wounds
  • Torn Claws

Is it serious enough to see the vet?

Cats can suffer a variety of wounds, the result of accidents, fights with other cats or from dog attacks. If your feline furrball is involved in an incident that leaves her with visible wounds — cuts, puncture wounds, lacerations, torn ears or tail damage — you will need to judge the severity of the wounds and decide if they are serious enough to need treatment by a vet or if they can be initially treated at home.

In all but the most minor of cases it is usually best that your cat sees the vet. With any wound there is always a real risk of infection and many apparently minor wounds may also require a few stitches to aid recovery.

In more serious cases, particularly where you are unable to stem the bleeding, and/or a wound is deep, or where you suspect there is any possibility of a broken bone or internal injuries you should call the hospital immediately for emergency advice.

Superficial wounds and minor injuries — grazes, shallow cuts, minor injuries to the pads of the feet — can generally be safely treated at home, but you might want to bring the cat in the next day for a check.

Warning. Always approach an injured cat with care. When in pain and confused, even normally gentle and docile felines can become aggressive. Crouch or kneel to put yourself on the same level as the cat and approach her slowly and calmly. Do not make direct eye contact and talk to her in a calm, reassuring voice.
You will probably need to restrain the cat to be able to treat the wound. Use a clean towel or blanket to wrap the body so that the paws — and those claws — are covered.

Evaluate the extent of blood loss before treating wounds

If your cat has been scratched bitten, or clawed, or has otherwise torn skin and tissue you first need to stop the bleeding. Apply sterile cold compresses to the wound, several if necessary. Press firmly on the wound and check the rate of bleeding every few minutes. Venous bleeding from minor wounds should stop fairly quickly.

Note: If you cannot stop the bleeding, or the blood is bright red and spurting from the wound it can indicate arterial bleeding. This is always serious. You should call the hospital immediately.

Clean and cover the wound

Once the bleeding has stopped you can clean and disinfect the injured area. If you are dealing with a surface wound, a severe graze for example, that has visible pieces of grit or other foreign material adhering to the skin, flush them away with clean cold water and gently clean the wound with an antibacterial soap to remove blood, dirt and debris. You may also need to cut the hair in and around the wound area. Pat the area dry before covering.

If the wound is deep and you can see foreign objects embedded in the cut these need to be removed at the hospital. Don’t attempt to remove anything deeply embedded in a wound yourself.

Apply some anti-bacterial cream and cover the wound with some sterile gauze (bandage). Wounds will actually heal faster without being covered but initially it is usually better to cover the injury because the cat will want to lick at it and its own saliva can cause infection. Alternatively you can make up a temporary Elizabethan collar from cardboard or plastic, which will prevent the cat from reaching the wound.

Monitor the wound

Even minor wounds can quickly become infected.You will need to see the vet to have the wounds disinfected and to obtain a course of antibiotics. Alternatively you need to change dressing daily and re-apply anti-bacterial cream.

Monitor the wound for any signs of fever, redness or swelling and call the hospital if you notice any of these signs of infection. Those symptoms mean your cat will need an injection or more powerful oral antibiotics to combat the problem.

More serious than they look

Beware of this type of wound, caused by deep bites or the penetration of a sharp foreign object. Puncture wounds can be deceptive, usually closing up and appearing to heal quickly with minimal bleeding. In many cases, however, infection will set in beneath the surface, requiring opening up and draining. Puncture wounds should always be checked at the hospital.

Torn claws should be treated by a vet

This is a common injury for cats and can be very painful, although rarely life-threatening. Limping and blood on a paw that the cat is constantly licking may be the first sign of a torn claw. This kind of injury is best treated at the hospital.

First aid treatment for minor cuts and grazes. If you cannot stem a bleeding wound, or can see bone or soft tissue or suspect a broken bone or internal injuries, seek immediate veterinary advice.

 
  • Evaluate The Injury
  • Stop The Bleeding
  • Clean, Disinfect & Cover
  • Superficial Wounds
  • Puncture Wounds

Is it serious enough to see the vet?

If your pet is involved in a fight or in a minor accident that results in visible wounds — cuts, puncture wounds, lacerations or torn ears etc. — you will need to judge if the wounds are serious enough to need treatment by a vet or if they can be initially treated at home.

In all but the most minor of cases it is usually best that your dog sees the vet, if only for a quick check up the following day. With any wound there is always a real risk of infection and many seemingly minor wounds will also often require a few stitches to ensure a swifter recovery.

In cases where you are unable to stem the bleeding, and/or a wound is deep enough for you to see bone or soft tissue, or where you suspect there is any possibility of a broken bone or internal injuries, you should call the hospital for advice.

Warning. Always approach an injured animal with care. When in pain or shocked, even the most gentle of family pets can be unpredictable. Crouch or kneel to put yourself on the same level as the dog and approach him slowly and calmly. Do not make direct eye contact and talk to the dog in a calm, soothing voice.

Evaluate the extent of blood loss before treating wounds

If your pet has been scratched bitten, or clawed, or has otherwise torn skin and tissue, you first need to stop the bleeding. Apply sterile cold compresses to the wound, several if necessary. Press firmly on the wound and check the rate of bleeding every few minutes. Bleeding from minor wounds should stop fairly quickly.

If bleeding is severe and on the legs, apply a tourniquet (using an elastic band or gauze) between the wound and the body, and apply a bandage and pressure over the wound. Loosen the tourniquet for 20-30 seconds every 15 minutes.

Note: If you cannot stop the bleeding, or the blood is bright red and spurting from the wound it can indicate arterial bleeding. This is always serious. You should call the hospital immediately.

Next, see ‘Clean, Disinfect & Cover’

Clean and cover the wound

Once the bleeding has stopped you can clean and disinfect the injured area. If you are dealing with a surface wound, a severe graze for example, that has visible pieces of grit or other foreign material adhering to the skin, flush them away with clean cold water and gently clean the wound with an antibacterial soap to remove blood, dirt and debris. You may also need to cut the hair in and around the wound area. Pat the area dry before covering.

If the wound is deep and you can see foreign objects embedded in the cut these need to be removed at the hospital. Don’t attempt to remove anything deeply embedded in a wound yourself.

Apply some anti-bacterial cream and cover the wound with some sterile gauze (bandage). Wounds will actually heal faster without being covered but initially it is usually better to cover the injury because the dog will want to lick at it and its own saliva can cause infection. Alternatively you can make up a temporary Elizabethan collar from cardboard or plastic, which will prevent the dog from reaching the wound.

The biggest problem with even minor wounds is the possibility of infection. Most dogs do not have a particularly ‘clean’ lifestyle and the possibility of a wound becoming infected is high. Infections can lead to serious problems. To avoid them you need to change the dressing and re-apply anti-bacterial cream a couple of times a day. While you can use anti-bacterial creams meant for human use as a temporary measure they’re not as effective for dogs as the animal antibiotic medications/drugs we would prescribe.

Minor wounds can be treated at home

Minor injuries — grazes, shallow cuts, minor injuries to the pads of the feet — can generally be safely treated at home but you might want to bring the dog in the next day for a check.

Clean and disinfect the wound, apply some anti-bacterial cream and cover with a soft, sterile bandage. Check the injury regularly for signs of infection and consult the vet if you see signs of redness or swelling.

More serious than they look

Beware of this type of wound, caused by deep bites or the penetration of a sharp foreign object such as a stick, a piece of fence wire or other items. Puncture wounds can be deceptive, usually closing up and appearing to heal quickly with minimal bleeding. In many cases, however, infection will set in beneath the surface, requiring opening up and draining. Puncture wounds should always be checked at the hospital.

All horse owners need to be able to treat minor injuries. If you cannot stem a bleeding wound, or suspect arterial bleeding, seek immediate veterinary advice.

  • Injury Prone
  • Bleeding
  • Assessing The Wound
  • Deep Cut Wounds
  • Full Skin Wounds
  • Cleansing A Wound
  • Puncture Wounds
  • Deeper Injuries
  • Bruising

Horses are more delicate than they look

Whether it’s a knock going over a jump, a nasty cut from tangling with barbed wire fences, or a kick from another horse, open wounds and bruising are something most horse owners will face at one time or another.

Unlike a dog or cat, you can’t bring your injured horse to the hospital. And if you are located a long way from our hospital it may be some time before we can get a vet to your property. These factors mean that horse owners need to be able to recognise the degree of severity of an injury and be able to apply common sense first aid to minimise damage and prevent infection.

For minor injuries first aid may be all you need. In most cases, however, it should be a temporary measure until the vet arrives. For any injury to a horse we suggest that you always call the hospital for advice on how to proceed.

Warning: When facing an injured horse, do not approach the animal unless you can do so safely. The horse will be in pain and confused, which can make him dangerous. It doesn’t help the horse, or you, if you end up in hospital with a fractured skull from a flying hoof. Stay calm. If you are panicked your excitement will be picked up by the horse and may make things worse. Step back and assess the situation carefully. Ask yourself: what do I need to do make it safe for me to help my horse?
Try to move the injured animal to a familiar, quiet place. Being somewhere he knows will help calm him down.

Is it arterial or venous?

Profuse bleeding can be very distressing but it can often look worse than it is. The average 500 kilogram horse has more than 26 litres of blood pulsing through his system so he can afford to lose a couple of litres without serious effects. Your job is to first assess the severity of the bleeding, and then to slow it down.

Arterial bleeding — where the blood is bright red and spurting from the wound — is much more serious, and much harder to control, than venous bleeding, where blood tends to seep and run. If you suspect arterial bleeding you need to seek immediate veterinary assistance.

To slow the rate of venous bleeding you can apply a clean cloth, folded several times to make a thick pad, over the source of the blood. You must apply firm, continuous pressure until the bleeding slows or stops. With a deep laceration or cut this can take some time so you are usually better to use a bandage or tape to hold the pad firmly in place. If the wound is located in a part of the body where you can’t tape a pad in place you’ll need to hold the bandage firmly in place until the bleeding stops.

Too much padding or too loose a pad will not allow you to apply adequate pressure. A thick towel wrapped around a cut on the leg will not help; you need to improvise a pad that can be held firmly in place. But not so tightly that it may impair circulation where the injury is on a leg. If you think the bandage or tape holding the pad is very tight around the leg, loosen it off and change it every 10 minutes to allow circulation to the rest of the leg.

What kind of cut are you dealing with?

You must first decide if what you are dealing with is a ‘deep cut‘ or a ‘full skin thickness cut.’ Each needs different treatment.

A skin laceration/cut may be quite deep without fully penetrating the skin to the tissue below. Deeper layers of the skin can be white to pink, and look very similar to tissue, so it can be difficult to decide if you are facing a full skin thickness cut or not. One simple way is to remember that, with a cut that does not penetrate the skin all the way to the tissue beneath you will not be able to pull the edges of the wound apart.

Depending on the site of the injury, a ‘deep cut’ wound might be as shallow as 3 mm or as deep as 7 mm where the skin is thickest, but even the deepest of these wounds do not normally require stitches and antibiotics, a good spray with Centrigen to deter flies will usually be sufficient. A full skin thickness laceration does require stitches.

Stitches not needed

For a laceration which does not fully penetrate the skin, clean gently and thoroughly with soap and water and apply Centrigen — which deters flies and has anti-bacterial agents — twice a day. Non-prescription anti-bacterial products do not last long. The hospital can prescribe a more long lasting medication.

If the wound is on a lower leg or any area which will get dirty or muddy apply a clean bandage.

‘Deep cut’ wounds do not require sutures but they should be carefully monitored to be sure the wound is healing properly without infection.

When soft tissue is exposed

Wounds which do fully penetrate the skin, exposing underlying soft tissue, need more involved treatment and usually require a vet. The wound may need sutures, although this will depend on a number of factors. As long as it is being monitored and cared for properly, a contaminated or badly traumatised wound may heal more quickly if left open. A sutured wound is unable to drain, which may lead to ascending infection.

Always clean and bandage a wound

Wounds should be flushed out with clean water to remove any dirt. A garden hose with a spray nozzle is ideal, as long as it’s drawn from a clean supply, i.e., not from the creek. The wound should then be bandaged.

If the wound is likely to need stitches, do not apply any medications or creams until a vet can examine the injury. Some products can interfere with the medications we may need to use to treat your horse, and prevent us using the most appropriate treatment. If veterinary help is more than a couple of hours away, consult the hospital for advice on what might be best to dress the wound with.

High risk of infection

Beware of puncture wounds, usually caused by penetrating sharp objects such as wire. They will often look like a minor injury while, under the surface, infection is raging. They can quickly become infected, with the first signs of problems — usually pain and swelling — only showing up 24 to 72 hours after an accident. A puncture wound seals rapidly, leaving the infection nowhere to go but to spread to surrounding tissues. Always have a deep puncture wound examined by the vet, who will open the wound, check for foreign bodies and clean and drain the wound to avoid infection.

Injury to underlying structures

Any wound or laceration has the potential to have also affected deeper underlying structures, particularly full skin thickness lacerations. For example, a lower limb injury that has penetrated into a joint capsule or tendon sheath can mean what looks like a routine cut may actually be more serious, potentially affecting the performance of the horse for life.

Any deep wound to areas around joints and tendons should always be examined by a veterinarian.

 

Reduce the swelling

If your horse suffers a kick or a hard blow that does not break the skin the area around the injury will be swollen and hot. Hose the area and apply an ice compress for a minimum of 30 minutes. Ask the hospital for advice on medications to limit swelling and pain.

One area where bruising is particularly serious is to the back of the thigh. The large muscle masses found there are prone to forming scars from deep bruises, which can affect the gait of the horse for life. Special veterinary care to quickly reduce the inflammation will be needed in these cases, together with controlled exercise during the following 60 to 90 days.

How you can help your pet in a crisis, and safely transport a sick or injured animal to the vet.

  • Fractures
  • Seizures
  • Internal bleeding
  • Burns
  • Shock
  • Choking
  • Resuscitation

 

Transporting pets with broken bones

If you know you are dealing with a fracture/broken bone, gently lay your pet on a flat surface such as a board for support and to use as a stretcher during transport (or use a throw rug or blanket as a sling).

Making sure you don’t put pressure on either the injured area or the animal’s chest, try to secure your pet to the stretcher — wrapping a blanket around the animal and the stretcher will usually do the trick.

In cases where you are unable to stem the bleeding, and/or a wound is deep enough for you to see bone or soft tissue, or where you suspect there is any possibility of a broken bone or internal injuries, you should first call the hospital for advice.

There is little you can do at home in the way of first aid for a fracture but if it’s impossible for you to get your pet to a veterinarian promptly you could attempt to set the fracture with a homemade splint but this should always be a last resort unless you have previous experience. A badly-placed splint may cause more harm than good.

 

Coping with ‘fits’

Don’t attempt to restrain an animal suffering a seizure but do keep it away from anything that might hurt it — open fires, hot surfaces, pools, furniture etc.

Time the seizure — they can last from seconds to minutes, usually 2-3 minutes. Once it has stopped, keep your pet warm and quiet and contact the hospital. Do not assume a seizure is a ‘one-off’ event; these incidents almost always indicate wider problems that require investigation.

 

Internal bleeding requires veterinary attention!

If you see your pet is bleeding from the nose, mouth or rectum, there is blood in their urine, or they are coughing up blood, have pale gums and are weak with a rapid pulse, you should suspect internal bleeding.

In these cases there is nothing you can do at home. Keep the animal as warm and quiet as possible and get it to the hospital or the nearest veterinarian as quickly as you can.

 

First aid for chemical and other burns

For a chemical burn caused by caustic solutions, flush the burn immediately with copious quantities of water.

For burns caused by direct heat (flame, hot surfaces etc) immediately apply an ice water compress to the burned area. You can also use packets of frozen vegetables like peas.

Any burn can cause infection and should be promptly checked by a veterinarian.

 

When your pet goes into shock

Many conditions can cause an animal to go into shock but it usually follows a severe injury or extreme fright. Shock symptoms include a weak pulse, shallow breathing, nervousness and dazed eyes.

Keep the animal restrained, warm and quiet. If your pet is unconscious, keep the head level with the rest of the body, gently place the animal on a flat stretcher and get it to the hospital.

 

Helping a pet that’s choking

A choking animal will show symptoms including difficulty breathing, constant pawing at the mouth and choking sounds when breathing or coughing. The lips and/or tongue may also have a blue tinge. The most common cause of choking is food or a foreign object stuck in the windpipe.

Approach a choking pet carefully – panic can make it bite.

If the animal can still breathe, keep it calm and get it to the hospital.

Look into the pet’s mouth. If you can clearly see a foreign object, try to gently remove it with tweezers, but be very, very careful you do not push the object further down the throat. The potential for you to make matters worse by poking around in your pet’s mouth and throat is so great that we cannot recommend you attempt it. But, in situations where you are unable to quickly get the animal to the vet it may be the only thing you can do.

One alternative is to try holding the dog upright with its nose pointing to the ground and shaking the animal. It sounds a little drastic but it can work, and it’s less likely to cause problems than using tweezers.

Even if you can clearly see what’s causing the blockage, if the animal is still breathing don’t spend too much time trying to get it out it if it’s not easy to reach — get your pet to the hospital, where we have specialised equipment for these cases.

 

‘Mouth to mouth’ for pets

If your pet is unconscious and/or has stopped breathing you can attempt to resuscitate them. Try to have someone else call the hospital while you attempt this so we can talk you through the process. If there are enough friends or family available it can be helpful to carry out the procedures below while on your way to the hospital.

Always stay calm and act in a methodical way.

First, check to see if your pet actually is unconscious. Open your pet’s airway by gently grasping the tongue and pulling it forward out of the mouth until it is flat. If the tongue does not stay flat when pulled out of the mouth, i.e., it retracts back into the mouth, it indicates your pet is still conscious. You should always pull out the tongue (gently!) to secure an airway before transporting an unconscious pet to the vet.

Check the animal’s throat to see if there are any foreign objects blocking the airway (see the previous section on Choking).

Perform resuscitation breathing by holding your pet’s mouth closed with your hand and breathing with your mouth directly into its nose until you see the animal’s chest expand (your pet breathes through its nose, not its mouth). Once the chest expands, continue resuscitation breathing once every 4 or 5 seconds.

Unfortunately, if your pet is already unconscious and/or has no pulse, the chances of a successful first aid resuscitation are not high. In an emergency however it may give your pet its only chance. Whatever else you might try to help your pet, your first action should be to call the hospital for advice.

Poison & Toxins

Pet poisoning is all too common, and all too often fatal. If you think your pet may have ingested a toxic substance, seek immediate veterinary advice.

  • Human Medications
  • Insecticides
  • Rat and Mice Baits
  • Human Food Items
  • Veterinary Medications
  • Chocolate
  • Household Products
  • Plant Material
  • Herbicides
  • Outdoor Toxins

When medicines can kill

Almost 25 per cent of calls were related to human medications accidentally ingested by pets. The most common were over-the-counter medicines such as painkillers (Panadol, Ibuprofen etc.), and prescription antidepressants and ADHD medications. Keep all medications well out of the reach of curious pets. So-called ‘recreational’ drugs are particularly dangerous to pets.

Killer fly sprays

About 20 per cent of calls concerned insecticide poisonings, mostly from incorrect use of the products commonly used on pets for flea control, or careless use of household insecticides used to control crawling and flying insects. The most serious poisonings occurred when products not labelled for use in cats were applied to them. Always follow label directions.

Dangerous baits

Number three in the top ten were baits used to kill mice and rats. These are mostly grain-based, and flavoured to attract rodents, which also makes them attractive to dogs and cats.

Several different types of rodenticides can cause seizures, internal bleeding or kidney failure. Make sure these products are kept out of reach of curious pets, and baits are carefully placed only in areas that pets cannot access.

Their last meal?

Xylitol, grapes, raisins, onions and garlic are the most commonly toxic foods ingested by pets in the USA. Grapes and raisins can cause kidney failure in dogs, while onions and garlic can cause anaemia if sufficient is ingested. Xylitol, a sugar alcohol used to sweeten sugar-free gums and mints, can cause low blood sugar and liver failure in dogs. For more information see this fact sheet.

Like lollies to your pet

Although they’re not as dangerous to animals as medicines designed for people, overdosing can be a serious problem. Many medications made for pets are flavoured to make it easier for you to get your pet to take their medicine. Some are tasty enough that animals may ingest the entire bottle. Common chewable medications include arthritis and incontinence medications. You should contact the hospital if your pet ingests more than the correct dose of medication.

Sweet treats that can kill

Although it comes in at number six in the ASPCA findings, our own experience is that toxic reactions to chocolate in local pets are much more common.

Chocolate contains methylxanthines, which act as stimulants to our pets. Methylxanthines can cause agitation, vomiting, diarrhoea, high heart rate, muscle tremors, seizures and death. The darker the chocolate, the more methylxanthines it contains, with unsweetened cooking chocolate the worse. You can learn more about how chocolate, and other common food items, affects your pet, and how Alpine Animal Doctors treats poison cases in this article.

Pets will eat anything

Cleaning supplies, such as bleach, acids, alkalis and other detergents were surprisingly high on the list of common poison causes. These can cause corrosive injury to the mouth and stomach. Other household items such as batteries and liquid potpourri can cause similar problems. Always keep these toxins behind securely locked doors.

Dangers in the garden

Both house plants and outdoor plants can be ingested by pets, and many contain toxins in their leaves, stems or flowers. Lilies can cause life-threatening kidney failure in cats, while sago palms can cause liver failure in dogs and cats. Tomato stems and leaves are also toxic. Choose your house and garden plants carefully to avoid risk to pets.

Be careful when you spray

Many herbicides have a salty taste, and pets will commonly ingest them. Always follow label directions and keep pets away from treated plants/areas until they are dry (the product label should tell you how long this should be). Even eating grass recently treated with herbicides can cause toxic reactions in your pet.

Lock the shed!

Antifreeze, fertilisers and solvents are a few of many common substances that animals can find outdoors, and will often be curious enough to ingest. Keep these items in securely locked sheds/cupboards or on high shelves where pets cannot get to them.

Common foods, household products and human medications are some of the biggest killers of domestic pets. Emergency veterinary treatment is critical.

  • Swift Action Is Vital
  • Death By Poison
  • Symptoms In Dogs
  • Symptoms In Cats
  • What To Tell The Vet

Know the symptoms of poisoning in your pet!

If your pet ingests or is otherwise affected by any toxic substance you should treat it as an emergency requiring urgent veterinary treatment. Call the hospital immediately for advice.

5756 2444

Please note that treatment at home is not a substitute for veterinary care in cases of poisoning.

 
 
 
 
 

Poisoning is one of the biggest killers of domestic pets

There are literally hundreds of products and substances harmful to domestic animals — dogs, cat and horses. They range from plant materials, indoor and outdoor, to seemingly harmless food items like onions and chocolate, to common household products such as snail baits, rat poisons and household cleansers. Ingestion of human medication, prescription and over the counter, is the single biggest cause of pet poisoning cases. Even products designed to help your pet — flea and tick powders and your pet’s own medication for example — can be toxic in some circumstances.

In all cases of poisoning there is potential for serious ongoing health issues. With many toxic substances the consequences are immediately severe, life threatening and, often, fatal. Even if we can pull a pet through the immediate danger there is a chance of permanent damage to various organs.

Successfully treating cases of poisoning requires prompt, appropriate hospital care. Time is of the essence and every pet carer needs to be familiar with the various symptoms that may indicate poisoning.

Dogs: Symptoms of poisoning 

Dogs are the pets most often involved in incidences of poisoning. Dogs that have swallowed something toxic may display any of the following symptoms. Symptoms may arise within minutes of ingestion, or take an hour or two to appear.

  • Drowsiness.
  • Diarrhoea and vomiting.
  • Changes in the colour of the gums and mucous membranes
  • Excessive salivation.
  • Laboured breathing and/or changes in normal respiration patterns.
  • Hyper-sensitivity to light or noise, apparent hallucinations and disorientation.
  • Ataxia, or staggering and wobbly gait.
  • Muscle tremors or convulsions.
  • Unusual odour on the breath or from the skin.
  • Burns inside the mouth or on the tongue or lips, generally an indicator of eating caustic chemicals.

You may also see stings, bite marks, or small punctures. These, coupled with any of the symptoms above, may indicate a bite from a venomous snake or spider. These cases require different treatment (see Snake bite: Symptoms and Treatment).

Cats: Symptoms of poisoning

Most of the symptoms of poisoning in cats are similar to those in dogs. Other, cat specific, symptoms include:

  • Lack of balance and co-ordination.
  • Excessive drooling.
  • Vomiting and diarrhoea.
  • Agitation. Some cats may become agitated after ingesting a toxin, walking aimlessly and excessively licking the fur or chewing at the paws. A poisoned cat may also become lethargic and present general weakness.
  • Rapid and superficial respiration that comes on suddenly.
  • Seizures. A cat experiencing a seizure may indicate that a toxic substance has already entered the cat’s bloodstream, an often fatal development. A seizing cat needs to see the vet immediately.
  • Coma. If a cat enters a coma after ingesting a poisonous substance it’s usually the last stage of toxic shock. The body is shutting down and even veterinary treatment is unlikely to save the animal. If the cat does come out of the coma there is a high probability of brain damage.

Is it really poison?

While there can be causes other than poisoning for your pet showing the kind of symptoms listed here, if you see any of them you should always first assume ingestion of a toxic substance and treat them as an emergency.

Unfortunately, pet owners are not always around to see exactly what their pets may have swallowed but if you suspect poisoning please try to have the following information when you call the hospital:

  • Species, breed, age, sex and weight.
  • Symptoms
  • Name/description of the substance involved, if known, and the amount the animal was exposed to.
  • How long it has been since your pet ate the material or was exposed to it.

Try to collect any material your pet may have vomited or chewed. Put it in a plastic sealable bag and bring it with you when you bring the animal in to the hospital. If possible, also bring the product container.

Poison cases are distressing, for your pet, for you and for the treating vet. Prevention is always preferable to cure. In addition to understanding the symptoms, pet owners should also familiarise themselves with the many substances that can be toxic, and try to keep them away from your pet.

Envenomation from the toxins contained in snake venom is always a serious and life threatening incident that must be treated swiftly to save the life of a pet that has been bitten

  • Signs And Symptoms
  • Which Snake
  • Emergency First Aid
  • Treatment At The Hospital
  • Prognosis

How to recognise a snake bite

If your pet is unfortunate enough to have an encounter with a snake you can help by being aware of the signs and symptoms of snake bite, and getting the affected animal to the hospital as quickly as possible. Dogs, cats and horses are all at risk, although curious dogs are the most frequently affected.

The first signs of snake envenomation are usually excitement, trembling, salivation and vomiting, gradually developing into weakness, wobbly gait and eventually paralysis. Afflicted dogs often have dilated pupils and slow light reflex, with clotting problems in their blood. Depending on the type of snake and how much venom has been injected, an animal bitten by a snake may show any of the following symptoms:-

  • Localised swelling and irritation
  • Vomiting
  • Drooling and trembling
  • Dilated pupils
  • Involuntary bladder or bowel release
  • Red or brown discolouration of urine
  • Rapid breathing and/or panting
  • Bleeding from bite wounds or bloody diarrhoea
  • Collapse. An animal may collapse immediately but then apparently recover, then develop symptoms over the course of the next hour
  • Paralysis (starting with the hind legs and progressing towards the head)

Identifying the offender

Venomous snakes are prevalent all over Australia. Pets in rural and country areas are at greater risk of being bitten. In the Alpine region we see a number of different types of venomous snakes, including Red-bellied Blacks, Tiger snakes, Death Adders and Brown snakes.

There are a few signs that can indicate what type of snake has envenomated your pet. When bitten by brown snakes some pets may experience a sudden collapse followed by a temporary recovery, while black snake bites will often create an area of localised swelling and irritation.

If you see the snake biting your pet and can identify the type you should tell us, but do not waste time trying to find it; we have facilities at the hospital to test your pet’s blood and quickly determine the type of venom involved.

Most snake bites in humans occur when people try to capture or kill snakes, often after seeing them bite a pet. It’s almost always best to leave the snake alone — it’s only doing what snakes do after all — and concentrate on helping your pet. There can also be legal ramifications if you kill a native reptile.

How to help a pet bitten by a snake

If your pet is still breathing keep him calm and as still as possible to minimise venom absorption. An elevated heart rate will allow the venom to move more quickly through the bloodstream.

Most snake bite wounds are not visible but if a limb has been bitten, bandage the entire leg firmly to limit venom movement through the lymphatics. Do not use a tourniquet to cut off circulation.

If your pet has stopped breathing, attempt mouth to nose resuscitation, and, if possible, continue this during transport of your pet to the hospital.

What we do to save your pet’s life

Once the affected animal reaches the hospital, treatment for snake envenomation may use some or all of the following techniques/procedures.

  • Blood or urine tests to identify the type of snake.
  • Oxygen therapy, and intravenous fluid therapy, to maintain blood pressure and protect the kidneys.
  • Give intravenous medications and antivenin if required. Some animals will need multiple doses.
  • Put your pet on a ventilator, a machine that breathes for your pet until it has recovered sufficiently to breathe on its own.
  • Use special feeding tubes to provide nutrition if the food pipe muscles are affected.

Treatment usually involves hospitalisation for at least 24 hours. In some cases your pet may be in the hospital for several days. After being discharged we recommend they be confined and rested for up to two weeks.

It is possible for your pet to be bitten by a snake and not become envenomated. In cases where clinical and laboratory evidence shows that the animal has not been envenomated we may only need to monitor your pet for any delayed symptoms for 12 – 24 hours.

Road to recovery

These days the treatment of snake bite has a good success rate and, with appropriate and timely medical therapy, many patients recover completely. But treating snake bite is always challenging — for the affected pet, for anxious pet parents and for the treating vet and nurses.

An animal that has suffered a snake bite will require constant monitoring, day and night, every one or two hours for the first 24 hours then every three hours for the next two days, with high rate intravenous infusions, blood clotting products and regular blood tests. Consequently, it can be expensive, although it may be covered by pet health insurance if you have it. It’s always better to keep your pets out of likely snake habitat and avoid the risk.

Any known or suspected case of poisoning needs immediate veterinary treatment. Your first priority should be to call the hospital for emergency advice and to get your pet to us — or your nearest vet — as quickly as you can. If this is not possible, here are some basic first aid treatments you can use at home.

  • Safety First
  • Skin & Eyes
  • Toxic Foods & Substances
  • Inducing Vomiting

Know your poisons before attempting first aid!

While there are some first aid treatments you can carry out to assist a pet that has been in contact with toxic materials, you should only do so if you:
a) Know the type and source of the poison/toxic substance.
b) Know when it was ingested by your pet, or when they came in contact with it.

Different types of toxic substances require different treatments and it’s critically important that you give your pet the correct first aid. Treatment may also vary depending on the length of time between a substance being swallowed and the onset of visible symptoms. For symptoms of poisoning in dogs and cats, see Poisons: Recognising Symptoms. If your pet is showing any of the symptoms listed the effects of the toxin are already underway.

If in doubt, call the hospital and speak to Dr Bek, who will advise you how to proceed. Remember that there is a very narrow window of opportunity for a veterinarian to successfully treat poisoning cases. Contact us promptly!

First aid for products affecting skin and eyes

If your pet has been in contact with a common household product that can be toxic to the skin or eyes, which includes many caustic cleansers, solvents, bleach, fertilisers, garden products etc., check the product label and treat your pet accordingly.

If the label advises you to wash with soap and water or flush your skin or eyes after contact with the product treat your pet the same way. Wash the animal’s skin with a mild soap such as dishwashing liquid and plenty of water (avoiding the eyes, mouth and nose) or flush the skin and eyes with plenty of fresh, clean water.

First aid for ingestion of toxic foods or substances

If your dog or cat has swallowed any of the following items you should induce vomiting immediately afterwards, before it has time to digest. That means within 5 to 15 minutes. After half an hour digestion has begun and the toxin will have already entered the bloodstream.

  • Prescription and over the counter drugs and medication (human and animal).
  • Insecticides, including flea and tick products for pets.
  • Herbicides, weed killers etc.
  • Rat, mice and slug poisons/baits.
  • Alcohol (including products that may contain alcohol, such as cough medicines, mouthwashes, after shave etc.).
  • Chocolate, especially unsweetened baking chocolate
  • Sugar free chewing gum, lollies and other sweets containing xylitol
  • Onions, including leeks, garlic, shallots
  • Macadamia nuts
  • Avocado
  • Grapes and raisins
  • Pear pips and stone fruit kernels (peaches, apricots, plums and cherries etc.)
  • Broccoli
  • Potato peelings
  • Tomato leaves and stems
  • Yeast dough
  • Mouldy foods
  • Tobacco products (do not attempt to induce vomiting if your pet has ingested nicotine)
  • Caffeine (more toxic to cats)
  • Plant materials from the lily, ivy, Aloe Vera and Chrysanthemum families.

Some of these materials may be less toxic to cats than to dogs. Some, like caffeine, are more dangerous to cats, but none should ever be fed to your pets, either raw or cooked. If your dog or cat is known or suspected to have ingested quantities of any of these foods/materials you can assume some degree of poisoning has occurred even if your pet is not showing symptoms. Consult the hospital for advice.

 
How to safely induce vomiting

Safely inducing vomiting in an animal is not as simple as in humans unfortunately. If you can get your pet to the hospital promptly — within an hour or so after they have swallowed something toxic — the prognosis is reasonably good even without you carrying out any first aid.

However, sometimes you may have to take action before coming to the hospital. If the animal has not already vomited, by inducing vomiting you may eliminate at least part of the toxin from the stomach. This must be done before the swallowed material has time to begin to digest. The most common recommendation — using a 3% solution of hydrogen peroxide — is not really appropriate in Australia, where most homes will not have this in the cupboard. Hydrogen peroxide is also not effective on cats, who need stronger veterinary drugs to force vomiting.

  • Alternative emetics which are reasonably safe to use are:
  • Table salt. A maximum of 2 teaspoons in a cup of water for an average sized dog, less for small breeds.
  • Copper sulphate crystals — give two crystals, about the size of a pea.
  • Washing soda crystals (sodium carbonate). Give 3 to 5 crystals orally. Do not use laundry detergents or washing powders.

Make your dog walk around after administering these emetics. He should vomit within about 10 to 15 minutes.

These solutions are not suitable for cats. Get a poisoned cat to the hospital where we can use the appropriate treatments.

You should also not try to induce vomiting for any acidic compound or petroleum based or corrosive substance. You should not attempt to induce vomiting except on veterinary advice.

Many common foods can make your pet ill. Some, ingested in sufficient quantity, can kill. If your pet eats large quantities of any of the foods listed here it’s critical that you call us and get your pet to the hospital quickly.

  • Chocoloate
  • Onions & Garlic
  • Macadamia Nuts
  • Grapes & Raisins
  • Sugar Free
  • Yeast & Dough
  • Mouldy Foods
  • Other

 

The ‘treat’ that’s toxic to dogs

At Alpine Animal Doctors we put chocolate at the top of the toxic foods list because it’s one food that dogs particularly crave, and will eat as much as they can get their greedy teeth into. You should never feed your dog chocolate. While a tiny piece is not likely to trigger toxicity it’s better for your pet if they never experience the taste at all.

Chocolate contains both theobromine and caffeine. These are methylxanthines and dogs are very sensitive to them. Depending on the amount ingested the effects of methylxanthines can include vomiting, diarrhoea, hyperactivity, increased heart rate, low blood pressure, seizure and tremors. We may also see increased thirst, more frequent urination, and lethargy.

These symptoms can lead to heart failure, coma and death within 12 to 36 hours of a dog overdosing on chocolate.

The amount of methylxanthines in chocolate varies. White chocolate has the lowest levels, followed by milk chocolate. Dark chocolate is the most dangerous. Gram for gram, unsweetened baking chocolate contains almost seven times the amount of theobromine as milk chocolate. The general rule is the more bitter the chocolate, the more toxic it will be.

Early treatment is vital. Once your pet reaches the hospital we will initiate decontamination procedures by making your dog vomit and tubing activated charcoal into the stomach. We may also use IV fluids to aid in eliminating toxins from the animal’s system. We will also use heart monitoring and supportive care. Treated early enough, most pets will recover from chocolate toxicity with no lasting damage.

 

Fried or sliced, onions are bad for pets

Onions come a close second to chocolate. All members of the onion family — garlic, leeks, chives, shallots et al — are toxic to pets, including onion powders and cooked onion.

Onions and garlic contain the toxin thiosulphate, which dogs and cats cannot break down. Thiosulphate can burst the red blood cells of dogs, causing haemolytic anaemia. Poisoning is usually seen a few days after the animal has eaten onions. Symptoms include vomiting and diarrhoea, very pale gums, blood in the urine, lethargy and anorexia. Their urine turns red and they become breathless due to the lack of red blood cells available to carry oxygen.

Effective treatment relies on early diagnosis and palliative care — treating symptoms as they arise. We will induce vomiting if the onion has been eaten in the previous two hours, we will administer activated charcoal to bind the offending material in the gut. If they are suffering from severe anaemia, a whole-blood transfusion will be necessary.

 

Not a health food for dogs 

Macadamia nut poisoning is often not as drastic as other food toxicities but swift treatment is just as important. Dogs can develop symptoms from eating as few as six to eight kernels, raw or roasted, or even macadamia butter.

Affected dogs will usually develop clinical signs within 12 hours and present with muscle tremors with weakness or paralysis of the hindquarters. Other symptoms can include vomiting, inability to walk straight, and a high temperature.Their limbs can become swollen and some owners report pain on movement of the legs.

Treatment involves administering activated charcoal, enemas and fluid therapy. With prompt treatment most dogs survive macadamia poisoning with no permanent damage. Macadamia poisoning has not been seen in cats.

 

Don’t peel them a grape!

Grapes and raisins are a recent addition to the list of toxicities seen in pets. The toxin involved has yet to be isolated but is suspected to be a mycotoxin that causes acute kidney failure. Initial symptoms are vomiting and diarrhoea within a few hours of ingestion, followed by weakness, anorexia, increased thirst and stomach pain, then acute renal failure within 48 hours of eating the offending material.

Treatment is by control of absorption if possible — inducing vomiting if eaten within two hours — or via activated charcoal to bind toxins within the gut. Intravenous fluid therapy is also used to manage renal failure.

Unfortunately, the damage that can be done in a short period means recovery is always uncertain, and if there is weakness, no urinating or high calcium levels in the blood the prognosis is invariably negative.

Sugar substitutes are dangerous to dogs

Xylitol toxicity is another relatively recently identified toxin. A naturally occurring sugar substitute it is used extensively in sugar-free lollies and chewing gum. It takes only small amounts of xylitol — as little as half a gram per kilo — to cause toxicity. Unlike in humans, in dogs xylitol is absorbed very quickly into the blood where it stimulates insulin release, causing profound low blood sugar leading to liver failure, bleeding, and death.

Vomiting is the first sign, with symptoms of hypoglycaemia occurring rapidly, followed by diarrhoea, collapse and seizures. In some cases dogs have developed acute liver failure with no signs of hypoglycaemia.

There is no antidote and treatment can only be palliative. The prognosis for uncomplicated low blood glucose is good, however, those animals that develop liver failure or fall into a coma have a very poor prognosis.

No dough for dogs

Ingestion of yeast/bread dough can be fatal to dogs because the dog’s body heat will cause the dough to rise in the stomach, often to several times its original size.

Symptoms seen with bread dough ingestion are caused by the ethanol produced during the rising process, and by foreign body obstruction. Symptoms may include severe abdominal pain, bloating, vomiting, lack of coordination, and depression.

Treatment, in cases of recent ingestion, involves inducing the animal to vomit. We may also administer cool water via a stomach tube to try to halt the rising process. Pain relief is also usually needed. In severe cases we may need to remove the dough surgically. Ethanol can cause an acidosis so we will also monitor blood parameters through ongoing blood tests.

 

If it’s off, keep it away from pets

Spoiled foods that are growing mould, whether in the bin or outdoors, can also be poisonous. All moulds contain mycotoxins. Some can contain tremorgenic mycotoxins, which can induce muscle tremors, ataxia, and convulsions that can last for several days. Effects have been reported in many species although dogs that roam are more at risk. The degree of severity of symptoms can vary from mild to severe, depending on the particular mycotoxin ingested.

Treatment for tremorgenic mycotoxin poisoning includes halting or minimising absorption by making your dog vomit, lavaging the stomach, and tubing with activated charcoal. The hospital will also attempt to control tremors and seizures using drugs delivered intravenously, and providing supportive care with IV fluid therapy. With early, appropriate treatment, prognosis is good for most pets.

 

But wait, there’s more…

Although the foods listed above are the most dangerous of foods toxic to your furry friends there are others, including potato peelings, the pips from pears and the kernels from stone fruit such as peaches, which can cause cyanide poisoning. All parts of the avocado are toxic to dogs, as are the leaves and stems of the tomato plant. Broccoli, eaten in large quantities, can be poisonous too.

While these may not be as life threatening as the major toxic foods, they can definitely make your cat or dog very ill. Animals are going to continue to poison themselves because they have no idea what’s good for them, or what’s bad for them. We can never hope to eliminate those bad habits but pet parents do need to be vigilant — especially about that enticing chocolate cake left out on the kitchen bench.

Contact Us

Contact Us Form

Saving one animal won’t change the world, but it will change the world for that one animal. – Dr Bek

Useful Links

FAQ
Privacy
Terms And Conditions
Vertical Floating Emergency Button with Breathing Animation

Subscribe Now

Don’t miss our future updates! Get Subscribed Today!

©2025. Alpine Animal Doctors | Alpine Hospital. All Rights Reserved.

Made With Love By OWS

Call Now Button